Normal pregnancy is characterized by systemic activation of the complement system. - Wikidata - wikimedia - TriplyDB

Normal pregnancy is characterized by systemic activation of the complement system.

Normal pregnancy is characterized by systemic activation of the complement system.

http://www.wikidata.org/entity/Q34499492

about

Emerging and Novel Functions of Complement Protein C1q The complement system and adverse pregnancy outcomes An AGM model for changes in complement during pregnancy: neutralization of influenza virus by serum is diminished in late third trimester An integrated approach based on multiplexed protein array and iTRAQ labeling for in-depth identification of pathways associated to IVF outcome The Microbiome and Complement Activation: A Mechanistic Model for Preterm Birth.Post-bone marrow transplant thrombotic microangiopathy.Dysregulated complement activation as a common pathway of injury in preeclampsia and other pregnancy complications.Changes in amniotic fluid and umbilical cord serum proteomic profiles of foetuses with intrauterine growth retardation.Fragment Bb: evidence for activation of the alternative pathway of the complement system in pregnant women with acute pyelonephritis.Pentraxin 3 in amniotic fluid: a novel association with intra-amniotic infection and inflammation.Increased concentration of the complement split product C5a in acute pyelonephritis during pregnancy.Anaphylatoxins in preterm and term labor Characterization of the myometrial transcriptome and biological pathways of spontaneous human labor at term.Pyelonephritis during pregnancy: a cause for an acquired deficiency of protein Z.Increased eculizumab requirements during pregnancy in a patient with paroxysmal nocturnal hemoglobinuria: case report and review of the literature.Changes in the biochemical and immunological components of serum and colostrum of overweight and obese mothers Progesterone and autoimmune disease.Complement Split Products in Amniotic Fluid in Pregnancies Subsequently Developing Early-Onset Preeclampsia Pentraxin 3 in maternal circulation: an association with preterm labor and preterm PROM, but not with intra-amniotic infection/inflammation Preeclampsia and pregnancies with small-for-gestational age neonates have different profiles of complement split products.Complement Activation in Placental Malaria.Serum Adipsin Levels throughout Normal Pregnancy and Preeclampsia.Maternal serum soluble CD30 is increased in pregnancies complicated with acute pyelonephritis Alternative complement pathway activation fragment Bb in early pregnancy as a predictor of preeclampsia.Activation of the alternative pathway of complement is a feature of pre-term parturition but not of spontaneous labor at term Fragment Bb in amniotic fluid: evidence for complement activation by the alternative pathway in women with intra-amniotic infection/inflammation Over-expression of the thrombin receptor (PAR-1) in the placenta in preeclampsia: a mechanism for the intersection of coagulation and inflammation.A role for mannose-binding lectin, a component of the innate immune system in pre-eclampsia Embryonic resorption and polycyclic aromatic hydrocarbons: putative immune-mediated mechanisms.Immune cells and molecules in pregnancy: friends or foes to the fetus?Complement in pregnancy: a delicate balance.Proteomic analysis of human follicular fluid associated with successful in vitro fertilization.The Role of C5a Receptor Signaling in Endotoxin-Induced Miscarriage and Preterm Birth.Complement component C4A and apolipoprotein A-I in plasmas as biomarkers of the severe, early-onset preeclampsia.Longitudinal characterization of renal proximal tubular markers in normotensive and preeclamptic pregnancies.Fetal membranes as an interface between inflammation and metabolism: increased aquaporin 9 expression in the presence of spontaneous labor at term and chorioamnionitis.Characterization of an anti-fetal AChR monoclonal antibody isolated from a myasthenia gravis patient.Spontaneous abortion is associated with elevated systemic C5a and reduced mRNA of complement inhibitory proteins in placenta.Chorionic gonadotropin up-regulates long pentraxin 3 expression in myeloid cells.Two Distinct Myeloid Subsets at the Term Human Fetal-Maternal Interface.

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P2860

Normal pregnancy is characterized by systemic activation of the complement system.

http://www.wikidata.org/entity/Q34499492

description

2005 nî lūn-bûn

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2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2005 թվականի ապրիլին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Normal pregnancy is characterized by systemic activation of the complement system.

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Normal pregnancy is characterized by systemic activation of the complement system.

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Normal pregnancy is characterized by systemic activation of the complement system.

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type

label

Normal pregnancy is characterized by systemic activation of the complement system.

@ast

Normal pregnancy is characterized by systemic activation of the complement system.

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Normal pregnancy is characterized by systemic activation of the complement system.

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prefLabel

Normal pregnancy is characterized by systemic activation of the complement system.

@ast

Normal pregnancy is characterized by systemic activation of the complement system.

@en

Normal pregnancy is characterized by systemic activation of the complement system.

@nl

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Journal of Maternal-Fetal and Neonatal Medicine

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Normal pregnancy is characterized by systemic activation of the complement system.

@en

P2093

Eleazar Soto

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Jimmy Espinoza

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Joon-Seok Hong

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Jyh Kae Nien

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Karina Richani

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Moshe Mazor

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Roberto Romero

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Samuel Edwin

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Yeon Mee Kim

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P2860

Complement regulatory proteins at the feto-maternal interface during human placental development: distribution of CD59 by comparison with membrane cofactor protein (CD46) and decay accelerating factor (CD55)

Chemotactic and anaphylatoxic fragment cleaved from the fifth component of guinea pig complement

The dark side of C5a in sepsis

A critical role for murine complement regulator crry in fetomaternal tolerance

Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome.

C3 and C4 complement components and acute phase proteins in late pregnancy and parturition.

Anti-C5 monoclonal antibody therapy prevents collagen-induced arthritis and ameliorates established disease.

C4a: the third anaphylatoxin of the human complement system.

Complement and immunoglobulins stimulate superoxide production by human leukocytes independently of phagocytosis.

The role of complement in pregnancy and fetal loss.

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239-245

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10.1080/14767050500072722

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4

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17

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Tinnakorn Chaiworapongsa

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2005-04-01T00:00:00Z

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7613828

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16147832

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1421513

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