Consequences of AhR activation in steady-state dendritic cells. - Wikidata - wikimedia - TriplyDB

Consequences of AhR activation in steady-state dendritic cells.

Consequences of AhR activation in steady-state dendritic cells.

http://www.wikidata.org/entity/Q34503487

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IDO2 in Immunomodulation and Autoimmune Disease Autoimmunity and infection in Sjögren's syndrome Toward understanding the role of aryl hydrocarbon receptor in the immune system: current progress and future trends New insights into the role of the aryl hydrocarbon receptor in the function of CD11c⁺ cells during respiratory viral infection Role and therapeutic value of dendritic cells in central nervous system autoimmunity.Linking the aryl hydrocarbon receptor with altered DNA methylation patterns and developmentally induced aberrant antiviral CD8+ T cell responses.Dietary ligands of the aryl hydrocarbon receptor induce anti-inflammatory and immunoregulatory effects on murine dendritic cells.The Aryl Hydrocarbon Receptor Influences Transplant Outcomes in Response to Environmental Signals.3,3'-Diindolylmethane Inhibits Flt3L/GM-CSF-induced-bone Marrow-derived CD103(+) Dendritic Cell Differentiation Regulating Phosphorylation of STAT3 and STAT5.Dendritic cells provide a potential link between smoking and inflammation in rheumatoid arthritis.Indoleamine 2,3-dioxygenase and dendritic cell tolerogenicity.Regulation of central nervous system autoimmunity by the aryl hydrocarbon receptor.Cytokines as molecular targets for aryl hydrocarbon receptor ligands: implications for toxicity and xenobiotic detoxification.The aryl hydrocarbon receptor and food allergy.Mode of action and dose-response framework analysis for receptor-mediated toxicity: The aryl hydrocarbon receptor as a case study.Mechanisms of chemical cooperative carcinogenesis by epidermal Langerhans cells.Ablating the aryl hydrocarbon receptor (AhR) in CD11c+ cells perturbs intestinal epithelium development and intestinal immunity.Interrelated Roles for the Aryl Hydrocarbon Receptor and Hypoxia Inducible Factor-1α in the Immune Response to Infection.Discovery of IDO1 Inhibitors: From Bench to Bedside.Indoleamine 2,3-Dioxygenase and Its Therapeutic Inhibition in Cancer.Aryl Hydrocarbon Receptor Ligands Indoxyl 3-sulfate and Indole-3-carbinol Inhibit FMS-like Tyrosine Kinase 3 Ligand-induced Bone Marrow-derived plasmacytoid Dendritic Cell Differentiation

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Consequences of AhR activation in steady-state dendritic cells.

http://www.wikidata.org/entity/Q34503487

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Consequences of AhR activation in steady-state dendritic cells.

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Consequences of AhR activation in steady-state dendritic cells.

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Consequences of AhR activation in steady-state dendritic cells.

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type

label

Consequences of AhR activation in steady-state dendritic cells.

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Consequences of AhR activation in steady-state dendritic cells.

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Consequences of AhR activation in steady-state dendritic cells.

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prefLabel

Consequences of AhR activation in steady-state dendritic cells.

@ast

Consequences of AhR activation in steady-state dendritic cells.

@en

Consequences of AhR activation in steady-state dendritic cells.

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Toxicological Sciences

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Consequences of AhR activation in steady-state dendritic cells.

@en

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David M Shepherd

http://www.w3.org/2001/XMLSchema#string

Tom Simones

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P2860

2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses tumor necrosis factor-alpha and anti-CD40-induced activation of NF-kappaB/Rel in dendritic cells: p50 homodimer activation is not affected

RelB, a new partner of aryl hydrocarbon receptor-mediated transcription

Control of T(reg) and T(H)17 cell differentiation by the aryl hydrocarbon receptor

Characterization of a murine Ahr null allele: involvement of the Ah receptor in hepatic growth and development

Immunobiology of dendritic cells

IDO expression by dendritic cells: tolerance and tryptophan catabolism

Development of monocytes, macrophages, and dendritic cells

2,3,7,8-Tetrachlorodibenzo-p-dioxin affects the number and function of murine splenic dendritic cells and their expression of accessory molecules.

Effects of TCDD on the fate of naive dendritic cells.

Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection.

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293-307

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scholarly article

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10.1093/TOXSCI/KFQ354

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2

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119

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2010-11-19T00:00:00Z

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49628931

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21097750

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3023567

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