Sequestration of cellular interacting partners by protein aggregates: implication in a loss-of-function pathology.
about
Sequestration of Ribosome during Protein Aggregate Formation: Contribution of ribosomal RNAHSP90 recognizes the N-terminus of huntingtin involved in regulation of huntingtin aggregation by USP19.Intrinsic Disorder in Proteins with Pathogenic Repeat Expansions.The proteome of neurofilament-containing protein aggregates in blood.
P2860
Sequestration of cellular interacting partners by protein aggregates: implication in a loss-of-function pathology.
description
2016 nî lūn-bûn
@nan
2016 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի մարտին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Sequestration of cellular inte ...... a loss-of-function pathology.
@ast
Sequestration of cellular inte ...... a loss-of-function pathology.
@en
Sequestration of cellular inte ...... a loss-of-function pathology.
@nl
type
label
Sequestration of cellular inte ...... a loss-of-function pathology.
@ast
Sequestration of cellular inte ...... a loss-of-function pathology.
@en
Sequestration of cellular inte ...... a loss-of-function pathology.
@nl
prefLabel
Sequestration of cellular inte ...... a loss-of-function pathology.
@ast
Sequestration of cellular inte ...... a loss-of-function pathology.
@en
Sequestration of cellular inte ...... a loss-of-function pathology.
@nl
P2860
P356
P1433
P1476
Sequestration of cellular inte ...... a loss-of-function pathology.
@en
P2093
P2860
P304
P356
10.1111/FEBS.13722
P407
P577
2016-03-26T00:00:00Z