Modeling a genetic risk for schizophrenia in iPSCs and mice reveals neural stem cell deficits associated with adherens junctions and polarity. - Wikidata - wikimedia - TriplyDB

Modeling a genetic risk for schizophrenia in iPSCs and mice reveals neural stem cell deficits associated with adherens junctions and polarity.

Modeling a genetic risk for schizophrenia in iPSCs and mice reveals neural stem cell deficits associated with adherens junctions and polarity.

http://www.wikidata.org/entity/Q34540416

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Brain-Region-Specific Organoids Using Mini-bioreactors for Modeling ZIKV Exposure Dishing out mini-brains: Current progress and future prospects in brain organoid research Molecular signatures associated with ZIKV exposure in human cortical neural progenitors A Dishful of a Troubled Mind: Induced Pluripotent Stem Cells in Psychiatric Research Decoding the non-coding genome: elucidating genetic risk outside the coding genome Using Patient-Derived Induced Pluripotent Stem Cells to Model and Treat Epilepsies From "directed differentiation" to "neuronal induction": modeling neuropsychiatric disease Utilizing induced pluripotent stem cells (iPSCs) to understand the actions of estrogens in human neurons Cyfip1 Regulates Presynaptic Activity during Development 15q11.2 CNV affects cognitive, structural and functional correlates of dyslexia and dyscalculia.Genetically-Informed Patient Selection for iPSC Studies of Complex Diseases May Aid in Reducing Cellular Heterogeneity.Induced Pluripotent Stem Cells: Global Research Trends.Altered expression of CDC42 signaling pathway components in cortical layer 3 pyramidal cells in schizophrenia.Probing disorders of the nervous system using reprogramming approaches.Reprogramming patient-derived cells to study the epilepsies A Common CYFIP1 Variant at the 15q11.2 Disease Locus Is Associated with Structural Variation at the Language-Related Left Supramarginal Gyrus.Altered Expression of ARP2/3 Complex Signaling Pathway Genes in Prefrontal Layer 3 Pyramidal Cells in Schizophrenia.Reduced CYFIP1 in Human Neural Progenitors Results in Dysregulation of Schizophrenia and Epilepsy Gene Networks.Modeling developmental neuropsychiatric disorders with iPSC technology: challenges and opportunities The TLX-miR-219 cascade regulates neural stem cell proliferation in neurodevelopment and schizophrenia iPSC model.Concise Review: Progress and Challenges in Using Human Stem Cells for Biological and Therapeutics Discovery: Neuropsychiatric Disorders.Disease signatures for schizophrenia and bipolar disorder using patient-derived induced pluripotent stem cells.Copy Number Variations in DISC1 and DISC1-Interacting Partners in Major Mental Illness.Increased abundance of translation machinery in stem cell-derived neural progenitor cells from four schizophrenia patients.Reduced protein synthesis in schizophrenia patient-derived olfactory cells.Modeling psychiatric disorders: from genomic findings to cellular phenotypes The hominoid-specific gene TBC1D3 promotes generation of basal neural progenitors and induces cortical folding in mice.The Proteome of BLOC-1 Genetic Defects Identifies the Arp2/3 Actin Polymerization Complex to Function Downstream of the Schizophrenia Susceptibility Factor Dysbindin at the Synapse Analysis of induced pluripotent stem cells carrying 22q11.2 deletion.The translational potential of human induced pluripotent stem cells for clinical neurology : The translational potential of hiPSCs in neurology.Induced pluripotent stem cells from patients with focal cortical dysplasia and refractory epilepsy.Zika-Virus-Encoded NS2A Disrupts Mammalian Cortical Neurogenesis by Degrading Adherens Junction Proteins.Genetics of schizophrenia.Human pluripotent stem cells as tools for neurodegenerative and neurodevelopmental disease modeling and drug discovery.Using hiPSCs to model neuropsychiatric copy number variations (CNVs) has potential to reveal underlying disease mechanisms.Modeling psychiatric disorders with patient-derived iPSCs.Altering the course of schizophrenia: progress and perspectives.Self-Organization of Spatial Patterning in Human Embryonic Stem Cells.The promises and challenges of human brain organoids as models of neuropsychiatric disease.Are reprogrammed cells a useful tool for studying dopamine dysfunction in psychotic disorders? A review of the current evidence.

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P2860

Modeling a genetic risk for schizophrenia in iPSCs and mice reveals neural stem cell deficits associated with adherens junctions and polarity.

http://www.wikidata.org/entity/Q34540416

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Modeling a genetic risk for sc ...... herens junctions and polarity.

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Ce Zhang

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Fengyu Zhang

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Georgia Makri

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Gregory Krauss

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Ha Nam Nguyen

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Joel E Kleinman

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P2860

Advances in autism

Rare chromosomal deletions and duplications increase risk of schizophrenia

A highly conserved protein family interacting with the fragile X mental retardation protein (FMRP) and displaying selective interactions with FMRP-related proteins FXR1P and FXR2P

Genome-wide expression profiling of lymphoblastoid cell lines distinguishes different forms of autism and reveals shared pathways

The fragile X syndrome protein represses activity-dependent translation through CYFIP1, a new 4E-BP

The glial nature of embryonic and adult neural stem cells

Large recurrent microdeletions associated with schizophrenia

CNVs: harbingers of a rare variant revolution in psychiatric genetics

Haploinsufficiency of Cyfip1 produces fragile X-like phenotypes in mice

beta1 integrin maintains integrity of the embryonic neocortical stem cell niche

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