Role of cardiac- and myeloid-MyD88 signaling in endotoxin shock: a study with tissue-specific deletion models.
about
Pathophysiology of sepsis-induced myocardial dysfunctionTissue inflammation and nitric oxide-mediated alterations in cardiovascular function are major determinants of endotoxin-induced insulin resistanceOverexpression of myeloid differentiation protein 88 in mice induces mild cardiac dysfunction, but no deficit in heart morphologyMyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control
P2860
Role of cardiac- and myeloid-MyD88 signaling in endotoxin shock: a study with tissue-specific deletion models.
description
2014 nî lūn-bûn
@nan
2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@en
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@nl
type
label
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@en
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@nl
prefLabel
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@en
Role of cardiac- and myeloid-M ...... ssue-specific deletion models.
@nl
P2093
P2860
P1433
P1476
Role of cardiac- and myeloid-M ...... issue-specific deletion models
@en
P2093
P2860
P304
P356
10.1097/ALN.0000000000000398
P407
P50
P577
2014-12-01T00:00:00Z