Mucus clearance as a primary innate defense mechanism for mammalian airways. - Wikidata - wikimedia - TriplyDB

Mucus clearance as a primary innate defense mechanism for mammalian airways.

Mucus clearance as a primary innate defense mechanism for mammalian airways.

http://www.wikidata.org/entity/Q34551378

about

17beta-Estradiol inhibits Ca2+-dependent homeostasis of airway surface liquid volume in human cystic fibrosis airway epithelia.TRPV4 channel is involved in the coupling of fluid viscosity changes to epithelial ciliary activity Identification of the SPLUNC1 ENaC-inhibitory domain yields novel strategies to treat sodium hyperabsorption in cystic fibrosis airway epithelial cultures Effects of reduced mucus oxygen concentration in airway Pseudomonas infections of cystic fibrosis patients Antimicrobial polypeptides in host defense of the respiratory tract Airway surface liquid volume regulation determines different airway phenotypes in liddle compared with betaENaC-overexpressing mice Micro- and macrorheology of mucus SPLUNC1 regulates airway surface liquid volume by protecting ENaC from proteolytic cleavage The antioxidant role of thiocyanate in the pathogenesis of cystic fibrosis and other inflammation-related diseases Risk of high blood pressure in salt workers working near salt milling plants: a cross-sectional and interventional study Mucociliary and long-term particle clearance in airways of patients with immotile cilia Epithelial Electrolyte Transport Physiology and the Gasotransmitter Hydrogen Sulfide Nanoparticle based insulin delivery system: the next generation efficient therapy for Type 1 diabetes The Evolution of Cystic Fibrosis Care Cystic fibrosis chronic rhinosinusitis: a comprehensive review Mucociliary clearance: pathophysiological aspects Control of airway tube diameter and integrity by secreted chitin-binding proteins in Drosophila Basal foot MTOC organizes pillar MTs required for coordination of beating cilia.Mucociliary clearance defects in a murine in vitro model of pneumococcal airway infection LRRC6 mutation causes primary ciliary dyskinesia with dynein arm defects Serotonin increases cilia-driven particle transport via an acetylcholine-independent pathway in the mouse trachea Altering mucus rheology to"solidify" human mucus at the nanoscale Molecular basis for pH-dependent mucosal dehydration in cystic fibrosis airways Airway Gland Structure and Function Airway hydration and COPD Increased susceptibility to pulmonary Pseudomonas infection in Splunc1 knockout mice Particle transport and deposition: basic physics of particle kinetics Aerosol Delivery of siRNA to the Lungs. Part 1: Rationale for Gene Delivery Systems Diminished self-chaperoning activity of the DeltaF508 mutant of CFTR results in protein misfolding 1,8-Cineol Reduces Mucus-Production in a Novel Human Ex Vivo Model of Late Rhinosinusitis Mutation of murine adenylate kinase 7 underlies a primary ciliary dyskinesia phenotype.nPKCepsilon, a P2Y2-R downstream effector in regulated mucin secretion from airway goblet cells Effect of Shear Stress on Pseudomonas aeruginosaIsolated from the Cystic Fibrosis Lung Inhaled innate immune ligands to prevent pneumonia.The expansion of autologous adipose-derived stem cells in vitro for the functional reconstruction of nasal mucosal tissue.Vocal fold epithelial barrier in health and injury: a research review.Myb promotes centriole amplification and later steps of the multiciliogenesis program.Innate immunity to Aspergillus species.Quantifying the effects of altering ambient humidity on ionic composition of vocal fold surface fluid.Substance P stimulates human airway submucosal gland secretion mainly via a CFTR-dependent process.

P2860

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P2860

Mucus clearance as a primary innate defense mechanism for mammalian airways.

http://www.wikidata.org/entity/Q34551378

description

2002 nî lūn-bûn

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2002 թուականի Մարտին հրատարակուած գիտական յօդուած

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2002 թվականի մարտին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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type

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Mucus clearance as a primary innate defense mechanism for mammalian airways.

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Journal of Clinical Investigation

P1476

Mucus clearance as a primary innate defense mechanism for mammalian airways.

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P2093

Michael R Knowles

http://www.w3.org/2001/XMLSchema#string

Richard C Boucher

http://www.w3.org/2001/XMLSchema#string

P2860

Evidence for periciliary liquid layer depletion, not abnormal ion composition, in the pathogenesis of cystic fibrosis airways disease

Loss-of-function mutations in a human gene related to Chlamydomonas reinhardtii dynein IC78 result in primary ciliary dyskinesia.

Effects of reduced mucus oxygen concentration in airway Pseudomonas infections of cystic fibrosis patients

Antimicrobial polypeptides in host defense of the respiratory tract

Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1

Human beta-defensin-1 is a salt-sensitive antibiotic in lung that is inactivated in cystic fibrosis

Cystic fibrosis and the salt controversy.

Molecular insights into the physiology of the 'thin film' of airway surface liquid.

Innate antimicrobial activity of nasal secretions.

Purification and functional reconstitution of the cystic fibrosis transmembrane conductance regulator (CFTR).

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571-577

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10.1172/JCI0215217

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5

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109

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11877463

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150901

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