Role of activation of protein kinase C in the stimulation of colonic epithelial proliferation and reactive oxygen formation by bile acids
about
Resistance to ursodeoxycholic acid-induced growth arrest can also result in resistance to deoxycholic acid-induced apoptosis and increased tumorgenicityBile acid stimulation of early growth response gene and mitogen-activated protein kinase is protein kinase C-dependentEffects of bile acids on proliferation and ultrastructural alteration of pancreatic cancer cell lines.The Yin and Yang of bile acid action on tight junctions in a model colonic epithelium.Chemical carcinogenesis: too many rodent carcinogensTaurodeoxycholate activates potassium and chloride conductances via an IP3-mediated release of calcium from intracellular stores in a colonic cell line (T84)1,25-Dihydroxyvitamin D3 and 12-O-tetradecanoyl phorbol 13-acetate cause differential activation of Ca(2+)-dependent and Ca(2+)-independent isoforms of protein kinase C in rat colonocytes.1,25(OH)2 vitamin D3 stimulates membrane phosphoinositide turnover, activates protein kinase C, and increases cytosolic calcium in rat colonic epithelium.Measurement in vivo of proliferation rates of slow turnover cells by 2H2O labeling of the deoxyribose moiety of DNA.Protein kinase C is activated in glomeruli from streptozotocin diabetic rats. Possible mediation by glucose.DNA lesions, inducible DNA repair, and cell division: three key factors in mutagenesis and carcinogenesis.Bile acids influence the growth, oestrogen receptor and oestrogen-regulated proteins of MCF-7 human breast cancer cells.The role of peroxisome proliferator-activated receptors in colorectal cancer.Overexpression of protein kinase C in HT29 colon cancer cells causes growth inhibition and tumor suppression.Types and amount of dietary fat and colon cancer risk: Prevention by omega-3 fatty acid-rich dietsBile acids as regulatory molecules.Bile acids inhibit NAD+-dependent 15-hydroxyprostaglandin dehydrogenase transcription in colonocytes.Farnesoid X receptor-Acting through bile acids to treat metabolic disorders.Bile acid accelerates erbB2-induced pro-tumorigenic activities in biliary tract cancer.Effects of bile acids on cyclooxygenase-2 expression in a rat model of duodenoesophageal anastomosis.Nutritional factors and colon cancer.COX-2 induction by unconjugated bile acids involves reactive oxygen species-mediated signalling pathways in Barrett's oesophagus and oesophageal adenocarcinoma.Protein kinase C: a family of isoenzymes with distinct roles in pathogenesis.Sulphation of lithocholic acid in the colon-carcinoma cell line CaCo-2.Secondary bile acid induced DNA damage in HT29 cells: are free radicals involved?Bile acids in the assessment of hepatocellular function.Alteration of membrane fatty acid composition and inositol phosphate metabolism in HT-29 human colon cancer cells.Characterization of phosphoinositide-specific phospholipase C in rat colonocyte membranes.Clostridium scindens baiCD and baiH genes encode stereo-specific 7alpha/7beta-hydroxy-3-oxo-delta4-cholenoic acid oxidoreductases.Influences of dietary deoxycholic acid on progression of hepatocellular neoplasms and expression of glutathione S-transferases in rats.Influence of dietary fat and feeding period on phosphoinositide metabolism in rat colonocytes.Modulation of antioxidant enzymes, reactive oxygen species, and glutathione levels in manganese superoxide dismutase-overexpressing NIH/3T3 fibroblasts during the cell cycle.On the in vitro vasoactivity of bile acids.Bile acids increase intracellular Ca(2+) concentration and nitric oxide production in vascular endothelial cells.Acid- and bile-induced PGE(2) release and hyperproliferation in Barrett's esophagus are COX-2 and PKC-epsilon dependent.Protein kinase CbetaII regulates its own expression in rat intestinal epithelial cells and the colonic epithelium in vivo.Effects of deoxycholate on human colon cancer cells: apoptosis or proliferation.Multiple calcium-mediated effector mechanisms regulate chloride secretory responses in T84-cells.Tauroursodeoxycholic acid, a bile acid, promotes blood vessel repair by recruiting vasculogenic progenitor cells.Acid and bile salt up-regulate BMP4 expression in human esophageal epithelium cells.
P2860
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P2860
Role of activation of protein kinase C in the stimulation of colonic epithelial proliferation and reactive oxygen formation by bile acids
description
1987 nî lūn-bûn
@nan
1987 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
Role of activation of protein ...... oxygen formation by bile acids
@ast
Role of activation of protein ...... oxygen formation by bile acids
@en
Role of activation of protein ...... oxygen formation by bile acids
@nl
type
label
Role of activation of protein ...... oxygen formation by bile acids
@ast
Role of activation of protein ...... oxygen formation by bile acids
@en
Role of activation of protein ...... oxygen formation by bile acids
@nl
prefLabel
Role of activation of protein ...... oxygen formation by bile acids
@ast
Role of activation of protein ...... oxygen formation by bile acids
@en
Role of activation of protein ...... oxygen formation by bile acids
@nl
P2093
P2860
P356
P1476
Role of activation of protein ...... oxygen formation by bile acids
@en
P2093
F R DeRubertis
J Pfanstiel
P A Craven
P2860
P304
P356
10.1172/JCI112844
P407
P577
1987-02-01T00:00:00Z