SIN3A, generally regarded as a transcriptional repressor, is required for induction of gene transcription by the aryl hydrocarbon receptor.
about
TET1 modulates H4K16 acetylation by controlling auto-acetylation of hMOF to affect gene regulation and DNA repair function.Targeted interference of SIN3A-TGIF1 function by SID decoy treatment inhibits Wnt signaling and invasion in triple negative breast cancer cells.The SIN3A histone deacetylase complex is required for a complete transcriptional response to hypoxia.Hypoxia and Chromatin: A Focus on Transcriptional Repression Mechanisms.
P2860
SIN3A, generally regarded as a transcriptional repressor, is required for induction of gene transcription by the aryl hydrocarbon receptor.
description
2014 nî lūn-bûn
@nan
2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@ast
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@en
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@nl
type
label
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@ast
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@en
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@nl
prefLabel
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@ast
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@en
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@nl
P2093
P2860
P356
P1476
SIN3A, generally regarded as a ...... the aryl hydrocarbon receptor.
@en
P2093
Oliver Hankinson
Parrisa Solaimani
P2860
P304
33655-33662
P356
10.1074/JBC.M114.611236
P407
P577
2014-10-10T00:00:00Z