Does IGFR1 inhibition result in increased muscle mass loss in patients undergoing treatment for pancreatic cancer? - Wikidata - wikimedia - TriplyDB

Does IGFR1 inhibition result in increased muscle mass loss in patients undergoing treatment for pancreatic cancer?

Does IGFR1 inhibition result in increased muscle mass loss in patients undergoing treatment for pancreatic cancer?

http://www.wikidata.org/entity/Q34590992

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Cardiac cachexia: hic et nunc Dysfunction of respiratory muscles in critically ill patients on the intensive care unit Muscle wasting: an overview of recent developments in basic research.Cancer cachexia, recent advances, and future directions.Evidence for partial pharmaceutical reversal of the cancer anorexia-cachexia syndrome: the case of anamorelin.Prediagnosis Circulating Insulin-Like Growth Factors and Pancreatic Cancer Survival.Characterization of Anthropometric Changes that Occur During Neoadjuvant Therapy for Potentially Resectable Pancreatic Cancer.Computed tomography diagnosed cachexia and sarcopenia in 725 oncology patients: is nutritional screening capturing hidden malnutrition?Anthropometric Changes in Patients with Pancreatic Cancer Undergoing Preoperative Therapy and Pancreatoduodenectomy.Loss of skeletal muscle during systemic chemotherapy is prognostic of poor survival in patients with foregut cancer.

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Does IGFR1 inhibition result in increased muscle mass loss in patients undergoing treatment for pancreatic cancer?

http://www.wikidata.org/entity/Q34590992

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2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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S13539-014-0145-Y

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Journal of Cachexia, Sarcopenia and Muscle

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Carla M Prado

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Christopher Garrett

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David R Fogelman

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Gauri R Varadhachary

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Holly Holmes

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Jessica Lieffers

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Khalil Mohammed

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Matthew H G Katz

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Michael J Overman

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Milind Javle

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P2860

Insulin receptor substrate is a mediator of phosphoinositide 3-kinase activation in quiescent pancreatic cancer cells

Strength conditioning in older men: skeletal muscle hypertrophy and improved function

FOLFIRINOX versus gemcitabine for metastatic pancreatic cancer

Localized Igf-1 transgene expression sustains hypertrophy and regeneration in senescent skeletal muscle.

IGF-1, inflammation and stem cells: interactions during muscle regeneration.

Sarcopenia and hypertrophy: a role for insulin-like growth factor-1 in aged muscle?

The emerging role of computerized tomography in assessing cancer cachexia.

Central tenet of cancer cachexia therapy: do patients with advanced cancer have exploitable anabolic potential?

Signaling pathways mediating the effects of insulin-like growth factor-I in bovine muscle satellite cells.

An anti-insulin-like growth factor I receptor antibody that is a potent inhibitor of cancer cell proliferation.

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s13539-014-0145-y

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