Cell biology of pathologic renal calcification: contribution of crystal transcytosis, cell-mediated calcification, and nanoparticles. - Wikidata - wikimedia - TriplyDB

Cell biology of pathologic renal calcification: contribution of crystal transcytosis, cell-mediated calcification, and nanoparticles.

Cell biology of pathologic renal calcification: contribution of crystal transcytosis, cell-mediated calcification, and nanoparticles.

http://www.wikidata.org/entity/Q34591050

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Characterization of granulations of calcium and apatite in serum as pleomorphic mineralo-protein complexes and as precursors of putative nanobacteria Putative nanobacteria represent physiological remnants and culture by-products of normal calcium homeostasis Fetuin-A/albumin-mineral complexes resembling serum calcium granules and putative nanobacteria: demonstration of a dual inhibition-seeding concept Filterability of staphylococcal species through membrane filters following application of stressors Critical evaluation of gamma-irradiated serum used as feeder in the culture and demonstration of putative nanobacteria and calcifying nanoparticles.Iron nanoparticles increase 7-ketocholesterol-induced cell death, inflammation, and oxidation on murine cardiac HL1-NB cells.Contribution of biologically derived nanoparticles to disease Calcifying nanoparticles promote mineralization in vascular smooth muscle cells: implications for atherosclerosis.Proteomic evaluation of biological nanoparticles isolated from human kidney stones and calcified arteries.Reactive oxygen species, inflammation and calcium oxalate nephrolithiasis.Characterization of biofilm formed by human-derived nanoparticles Key role of alkaline phosphatase in the development of human-derived nanoparticles in vitro.Macropinocytosis is the major mechanism for endocytosis of calcium oxalate crystals into renal tubular cells.Bions: a family of biomimetic mineralo-organic complexes derived from biological fluids Clinical implications of calcifying nanoparticles in dental diseases: a critical review.Unified theory on the pathogenesis of Randall's plaques and plugs Spontaneous calcification process in primary renal cells from a medullary sponge kidney patient harbouring a GDNF mutation.Chronic hypokalaemia and nephrocalcinosis.The role of calcifying nanoparticles in biology and medicine.Vascular Calcification and Stone Disease: A New Look towards the Mechanism.Osteogenic changes in kidneys of hyperoxaluric rats.Idiopathic Recurrent Calcium Urolithiasis (IRCU): pathophysiology evaluated in light of oxidative metabolism, without and with variation of several biomarkers in fasting urine and plasma--a comparison of stone-free and -bearing male patients, emphas Surface heat shock protein 90 serves as a potential receptor for calcium oxalate crystal on apical membrane of renal tubular epithelial cells.Ultrastructural investigation of crystal deposits in Npt2a knockout mice: are they similar to human Randall's plaques?Association between Randall's plaque and calcifying nanoparticles.Do blood-borne calcifying nanoparticles self-propagate?Systemic injection of planktonic forms of mammalian-derived nanoparticles alters arterial response to injury in rabbits.Biologic nanoparticles and platelet reactivity Pathological calcification and replicating calcifying-nanoparticles: general approach and correlation.Sphingomonas sp. is a novel cell culture contaminant.Histological aspects of the "fixed-particle" model of stone formation: animal studies.Crystal nephropathies: mechanisms of crystal-induced kidney injury.Of nanobacteria, nanoparticles, biofilms and their role in health and disease: facts, fancy and future.NF-kappaB activation in renal tubular epithelial cells by oxalate stimulation.Pathogenesis of nephrolithiasis: recent insight from cell biology and renal pathology.Human-derived nanoparticles and vascular response to injury in rabbit carotid arteries: proof of principle.Anatomically-specific intratubular and interstitial biominerals in the human renal medullo-papillary complex.How do stones form? Is unification of theories on stone formation possible?Role of Osteogenesis in the Formation of Randall's Plaques.

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P2860

Cell biology of pathologic renal calcification: contribution of crystal transcytosis, cell-mediated calcification, and nanoparticles.

http://www.wikidata.org/entity/Q34591050

description

2006 nî lūn-bûn

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2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2006年の論文

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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type

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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6650.2006.06021

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Journal of Investigative Medicine

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Cell biology of pathologic ren ...... cification, and nanoparticles.

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P2093

Ewa Rzewuska

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Gerard Farell

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John C Lieske

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Lorraine Fitzpatrick

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Sean Harrington

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Shihui Yu

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Virginia M Miller

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Vivek Kumar

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P2860

An alternative interpretation of nanobacteria-induced biomineralization

Nanobacteria: an alternative mechanism for pathogenic intra- and extracellular calcification and stone formation

Tamm-Horsfall protein is a critical renal defense factor protecting against calcium oxalate crystal formation

A study of crystal matrix extract and urinary prothrombin fragment 1 from a stone-prone and stone-free population.

Organization of the inter-alpha-inhibitor heavy chains on the chondroitin sulfate originating from Ser(10) of bikunin: posttranslational modification of IalphaI-derived bikunin.

Inhibition of nanobacteria by antimicrobial drugs as measured by a modified microdilution method.

High expression of genes for calcification-regulating proteins in human atherosclerotic plaques.

Diffuse calcification in human coronary arteries. Association of osteopontin with atherosclerosis

Evidence of nanobacterial-like structures in calcified human arteries and cardiac valves.

Calcification of vascular smooth muscle cell cultures: inhibition by osteopontin.

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