Epithelial-mesenchymal transition (EMT) induced by TNF-α requires AKT/GSK-3β-mediated stabilization of snail in colorectal cancer - Wikidata - wikimedia - TriplyDB

Epithelial-mesenchymal transition (EMT) induced by TNF-α requires AKT/GSK-3β-mediated stabilization of snail in colorectal cancer

Epithelial-mesenchymal transition (EMT) induced by TNF-α requires AKT/GSK-3β-mediated stabilization of snail in colorectal cancer

http://www.wikidata.org/entity/Q34595133

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GSK-3 as potential target for therapeutic intervention in cancer Roles of GSK-3 and microRNAs on epithelial mesenchymal transition and cancer stem cells Environmental immune disruptors, inflammation and cancer risk RBP2 induces epithelial-mesenchymal transition in non-small cell lung cancer Organoid-based epithelial to mesenchymal transition (OEMT) model: from an intestinal fibrosis perspective.Snail contributes to the maintenance of stem cell-like phenotype cells in human pancreatic cancer.Hsa-miR-19a is associated with lymph metastasis and mediates the TNF-α induced epithelial-to-mesenchymal transition in colorectal cancer ShaoYao decoction ameliorates colitis-associated colorectal cancer by downregulating proinflammatory cytokines and promoting epithelial-mesenchymal transition.ROS-induced epithelial-mesenchymal transition in mammary epithelial cells is mediated by NF-kB-dependent activation of Snail.A Combined ULBP2 and SEMA5A Expression Signature as a Prognostic and Predictive Biomarker for Colon Cancer Epithelial-mesenchymal transition in prostate cancer: an overview.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Loss of 4E-BP1 function induces EMT and promotes cancer cell migration and invasion via cap-dependent translational activation of snail.Chemical compounds from anthropogenic environment and immune evasion mechanisms: potential interactions Crosstalk of Oncogenic Signaling Pathways during Epithelial-Mesenchymal Transition.The host microenvironment influences prostate cancer invasion, systemic spread, bone colonization, and osteoblastic metastasis.Sub-sets of cancer stem cells differ intrinsically in their patterns of oxygen metabolism.Increased expression of tumor necrosis factor-α is associated with advanced colorectal cancer stages Inositol pyrophosphates promote tumor growth and metastasis by antagonizing liver kinase B1 Bisphenol A modulates colorectal cancer protein profile and promotes the metastasis via induction of epithelial to mesenchymal transitions.Short hairpin RNA- mediated gene knockdown of FOXM1 inhibits the proliferation and metastasis of human colon cancer cells through reversal of epithelial-to-mesenchymal transformation.Shenling Baizhu San supresses colitis associated colorectal cancer through inhibition of epithelial-mesenchymal transition and myeloid-derived suppressor infiltration Endogenous Nodal promotes melanoma undergoing epithelial-mesenchymal transition via Snail and Slug in vitro and in vivo Enolase-1 is a therapeutic target in endometrial carcinoma.Inflammatory mediators, tumor necrosis factor-α and interferon-γ, induce EMT in human PTC cell lines Activin and TGFβ use diverging mitogenic signaling in advanced colon cancer IWR-1 inhibits epithelial-mesenchymal transition of colorectal cancer cells through suppressing Wnt/β-catenin signaling as well as survivin expression CCL21 Facilitates Chemoresistance and Cancer Stem Cell-Like Properties of Colorectal Cancer Cells through AKT/GSK-3β/Snail Signals.A link between lipid metabolism and epithelial-mesenchymal transition provides a target for colon cancer therapy.Histone deacetylase inhibitor valproic acid (VPA) promotes the epithelial mesenchymal transition of colorectal cancer cells via up regulation of Snail Akt Activation Correlates with Snail Expression and Potentially Determines the Recurrence of Prostate Cancer in Patients at Stage T2 after a Radical Prostatectomy.Visfatin is involved in promotion of colorectal carcinoma malignancy through an inducing EMT mechanism.TGF-β induces M2-like macrophage polarization via SNAIL-mediated suppression of a pro-inflammatory phenotype Enhanced myeloid differentiation factor 88 promotes tumor metastasis via induction of epithelial-mesenchymal transition in human hepatocellular carcinoma.STC2 promotes the epithelial-mesenchymal transition of colorectal cancer cells through AKT-ERK signaling pathways.Cytosolic THUMPD1 promotes breast cancer cells invasion and metastasis via the AKT-GSK3-Snail pathway SMAD7: a timer of tumor progression targeting TGF-β signaling.A clinical perspective on the role of chronic inflammation in gastrointestinal cancer.Inflammation increases NOTCH1 activity via MMP9 and is counteracted by Eicosapentaenoic Acid-free fatty acid in colon cancer cells.Emerging cytokine networks in colorectal cancer.

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Epithelial-mesenchymal transition (EMT) induced by TNF-α requires AKT/GSK-3β-mediated stabilization of snail in colorectal cancer

http://www.wikidata.org/entity/Q34595133

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2013 nî lūn-bûn

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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Epithelial-mesenchymal transit ...... of snail in colorectal cancer

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Epithelial-mesenchymal transit ...... of snail in colorectal cancer

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Epithelial-mesenchymal transit ...... of snail in colorectal cancer

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Epithelial-mesenchymal transit ...... of snail in colorectal cancer

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Epithelial-mesenchymal transit ...... of snail in colorectal cancer

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Bin-Hua Zhou

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Cui-Lin Li

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Fan Zhang

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Ge Zhang

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Hao Wang

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Hong-Sheng Wang

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Jun Du

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Shao-Hui Cai

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Xian-Feng Wang

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Xian-Zhang Bu

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P2860

Dual regulation of Snail by GSK-3beta-mediated phosphorylation in control of epithelial-mesenchymal transition

Stabilization of snail by NF-kappaB is required for inflammation-induced cell migration and invasion

Inflammation and cancer

The basics of epithelial-mesenchymal transition

Phosphorylation regulates the subcellular location and activity of the snail transcriptional repressor

Epithelial-mesenchymal transitions in development and disease

Control of beta-catenin phosphorylation/degradation by a dual-kinase mechanism

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

Correlation of Snail expression with histological grade and lymph node status in breast carcinomas

The snail superfamily of zinc-finger transcription factors

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colorectal cancer

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3576347

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