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Molecular mechanisms of excitotoxicity and their relevance to pathogenesis of neurodegenerative diseases

Molecular mechanisms of excitotoxicity and their relevance to pathogenesis of neurodegenerative diseases

http://www.wikidata.org/entity/Q34606186

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Alzheimer's disease--input of vitamin D with mEmantine assay (AD-IDEA trial): study protocol for a randomized controlled trial Excitatory amino acid transporter 2 associates with phosphorylated tau and is localized in neurofibrillary tangles of tauopathic brains The excitatory neurotransmitter glutamate stimulates DNA repair to increase neuronal resiliency Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy Behavioral Abnormality Induced by Enhanced Hypothalamo-Pituitary-Adrenocortical Axis Activity under Dietary Zinc Deficiency and Its Usefulness as a Model Risk of Parkinson's Disease in the Users of Antihypertensive Agents: An Evidence from the Meta-Analysis of Observational Studies Nuclear Tau and Its Potential Role in Alzheimer's Disease Migraine Triggers and Oxidative Stress: A Narrative Review and Synthesis Neural and mesenchymal stem cells in animal models of Huntington's disease: past experiences and future challenges Pharmacological Alternatives for the Treatment of Neurodegenerative Disorders: Wasp and Bee Venoms and Their Components as New Neuroactive Tools Cathepsin L plays a role in quinolinic acid-induced NF-Κb activation and excitotoxicity in rat striatal neurons Defects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's disease Astrocyte Differentiation of Human Pluripotent Stem Cells: New Tools for Neurological Disorder Research Isothiocyanates Are Promising Compounds against Oxidative Stress, Neuroinflammation and Cell Death that May Benefit Neurodegeneration in Parkinson's Disease Autophagy in Neurodegenerative Diseases and Metal Neurotoxicity Chlorpyrifos Toxicity in Mouse Cultured Cerebellar Granule Neurons at Different Stages of Development: Additive Effect on Glutamate-Induced Excitotoxicity Conditioned medium of human adipose-derived mesenchymal stem cells mediates protection in neurons following glutamate excitotoxicity by regulating energy metabolism and GAP-43 expression Plants and phytochemicals for Huntington's disease A mitochondrial division inhibitor, Mdivi-1, inhibits mitochondrial fragmentation and attenuates kainic acid-induced hippocampal cell death Genetic variation influences glutamate concentrations in brains of patients with multiple sclerosis Prolactin-induced neuroprotection against glutamate excitotoxicity is mediated by the reduction of [Ca2+]i overload and NF-κB activation.Phosphorylation of tau at Y18, but not tau-fyn binding, is required for tau to modulate NMDA receptor-dependent excitotoxicity in primary neuronal culture.Insulin blocks glutamate-induced neurotoxicity in differentiated SH-SY5Y neuronal cells Selective neuronal vulnerability to oxidative stress in the brain.A 24-residue peptide (p5), derived from p35, the Cdk5 neuronal activator, specifically inhibits Cdk5-p25 hyperactivity and tau hyperphosphorylation.Water extract from the leaves of Withania somnifera protect RA differentiated C6 and IMR-32 cells against glutamate-induced excitotoxicity Brain glutamate levels measured by magnetic resonance spectroscopy in patients with bipolar disorder: a meta-analysis.Transcriptional expression patterns triggered by chemically distinct neuroprotective molecules.Fragile X mental retardation protein expression in Alzheimer's disease Korean Red Ginseng inhibits apoptosis in neuroblastoma cells via estrogen receptor β-mediated phosphatidylinositol-3 kinase/Akt signaling.Glutamatergic targets for enhancing extinction learning in drug addiction Increased glutamate receptor and transporter expression in the cerebral cortex and striatum of gcdh-/- mice: possible implications for the neuropathology of glutaric acidemia type I.Protection of pyruvate against glutamate excitotoxicity is mediated by regulating DAPK1 protein complex Neuronal damage and protection in the pathophysiology and treatment of psychiatric illness: stress and depression.Focal cortical lesions induce bidirectional changes in the excitability of fast spiking and non fast spiking cortical interneurons Glutamate excitotoxicity is involved in the induction of paralysis in mice after infection by a human coronavirus with a single point mutation in its spike protein.Analysis of synaptic gene expression in the neocortex of primates reveals evolutionary changes in glutamatergic neurotransmission Ketamine Protects Gamma Oscillations by Inhibiting Hippocampal LTD Transcriptional Regulation of the Astrocytic Excitatory Amino Acid Transporter 1 (EAAT1) via NF-κB and Yin Yang 1 (YY1).Transient mitochondrial permeability transition mediates excitotoxicity in glutamate-sensitive NSC34D motor neuron-like cells

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Molecular mechanisms of excitotoxicity and their relevance to pathogenesis of neurodegenerative diseases

http://www.wikidata.org/entity/Q34606186

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2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年论文

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Acta Pharmacologica Sinica

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Molecular mechanisms of excito ...... of neurodegenerative diseases

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Xiao-xia Dong

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Yan Wang

http://www.w3.org/2001/XMLSchema#string

Zheng-hong Qin

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P2860

Selective degradation of mitochondria by mitophagy

Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD

Apoptosis of T cells mediated by Ca2+-induced release of the transcription factor MEF2

An autophagic mechanism is involved in apoptotic death of rat striatal neurons induced by the non-N-methyl-D-aspartate receptor agonist kainic acid

Pruning and loss of excitatory synapses by the parkin ubiquitin ligase

Abeta oligomers induce neuronal oxidative stress through an N-methyl-D-aspartate receptor-dependent mechanism that is blocked by the Alzheimer drug memantine.

Parkinson's disease: mechanisms and models

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

Parkinson's disease. First of two parts

Polyglutamine-expanded huntingtin promotes sensitization of N-methyl-D-aspartate receptors via post-synaptic density 95.

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379-387

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10.1038/APS.2009.24

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4

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30

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neurodegeneration

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4002277

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