miR-199a-5p Is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1 - Wikidata - wikimedia - TriplyDB

miR-199a-5p Is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1

miR-199a-5p Is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1

http://www.wikidata.org/entity/Q34607960

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Epigenetic modulation as a therapeutic approach for pulmonary arterial hypertension Beyond the genome: epigenetic mechanisms in lung remodeling MicroRNAs in fibrosis: opportunities and challenges MicroRNAs in idiopathic pulmonary fibrosis: involvement in pathogenesis and potential use in diagnosis and therapeutics Regulatory Mechanisms of the Molecular Pathways in Fibrosis Induced by MicroRNAs Validated prediction of pro-invasive growth factors using a transcriptome-wide invasion signature derived from a complex 3D invasion assay The code of non-coding RNAs in lung fibrosis Regulation of Collagen V Expression and Epithelial-Mesenchymal Transition by miR-185 and miR-186 during Idiopathic Pulmonary Fibrosis.The roles of microRNA families in hepatic fibrosis.Pathogenesis of idiopathic pulmonary fibrosis: review of recent findings.Caveolin-1 is down-regulated in alveolar rhabdomyosarcomas and negatively regulates tumor growth.Comprehensive investigation of the caveolin 2 gene: resequencing and association for kidney transplant outcomes MicroRNA-gene expression network in murine liver during Schistosoma japonicum infection.MiRNA-30a inhibits AECs-II apoptosis by blocking mitochondrial fission dependent on Drp-1.The role of miRNAs in the regulation of inflammatory processes during hepatofibrogenesis Pharmacological modulation of the AKT/microRNA-199a-5p/CAV1 pathway ameliorates cystic fibrosis lung hyper-inflammation Molecular mechanisms of regulation and action of microRNA-199a in testicular germ cell tumor and glioblastomas.MicroRNA MiR-199a-5p regulates smooth muscle cell proliferation and morphology by targeting WNT2 signaling pathway.Effect of vitrification on the microRNA transcriptome in mouse blastocysts.A computational approach to identifying gene-microRNA modules in cancer.SMAD-independent down-regulation of caveolin-1 by TGF-β: effects on proliferation and survival of myofibroblasts Donor caveolin 1 (CAV1) genetic polymorphism influences graft function after renal transplantation.Epigenetic modifications of caveolae associated proteins in health and disease.MiR-199a inhibits the angiogenic potential of endometrial stromal cells under hypoxia by targeting HIF-1α/VEGF pathway.miR-130b-3p Modulates Epithelial-Mesenchymal Crosstalk in Lung Fibrosis by Targeting IGF-1 The Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in Pulmonary Fibrosis The miR-199-dynamin regulatory axis controls receptor-mediated endocytosis.Interaction of long noncoding RNAs and microRNAs in the pathogenesis of idiopathic pulmonary fibrosis Alternatively Activated (M2) Macrophage Phenotype Is Inducible by Endothelin-1 in Cultured Human Macrophages.MicroRNAs as potential targets for progressive pulmonary fibrosis Long Non-coding RNA Growth Arrest-specific Transcript 5 (GAS5) Inhibits Liver Fibrogenesis through a Mechanism of Competing Endogenous RNA.A YAP/TAZ-miR-130/301 molecular circuit exerts systems-level control of fibrosis in a network of human diseases and physiologic conditions A Patient-Derived, Pan-Cancer EMT Signature Identifies Global Molecular Alterations and Immune Target Enrichment Following Epithelial-to-Mesenchymal Transition.MicroRNA-27a-3p Is a Negative Regulator of Lung Fibrosis by Targeting Myofibroblast Differentiation.MicroRNA-199a is induced in dystrophic muscle and affects WNT signaling, cell proliferation, and myogenic differentiation.miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.U6 is unsuitable for normalization of serum miRNA levels in patients with sepsis or liver fibrosis.Serum extracellular vesicular miR-21-5p is a predictor of the prognosis in idiopathic pulmonary fibrosis.The role of miR-497-5p in myofibroblast differentiation of LR-MSCs and pulmonary fibrogenesis.

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miR-199a-5p Is upregulated during fibrogenic response to tissue injury and mediates TGFbeta-induced lung fibroblast activation by targeting caveolin-1

http://www.wikidata.org/entity/Q34607960

description

2013 nî lūn-bûn

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2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@ast

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@en

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@nl

type

label

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@ast

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@en

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@nl

prefLabel

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@ast

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@en

miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@nl

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JOURNAL.PGEN.1003291

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miR-199a-5p Is upregulated dur ...... vation by targeting caveolin-1

@en

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Benoit Wallaert

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Brice Marcet

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Christian Lacks Lino Cardenas

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Christoph Roderburg

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Edmone Dewaeles

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Elisabeth Courcot

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François Glowacki

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Imène Sarah Henaoui

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Jean-Marc Lo-Guidice

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John Tedrow

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e1003291

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10.1371/JOURNAL.PGEN.1003291

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2

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9

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Naftali Kaminski

Jadranka Milosevic

Marie-Christine Copin

Imène Sarah Henaoui

Nicolas Pottier

Christelle Cauffiez

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2013-02-14T00:00:00Z

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235786746

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23459460

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3573122

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