Tregs utilize beta-galactoside-binding protein to transiently inhibit PI3K/p21ras activity of human CD8+ T cells to block their TCR-mediated ERK activity and proliferation - Wikidata - wikimedia - TriplyDB

Tregs utilize beta-galactoside-binding protein to transiently inhibit PI3K/p21ras activity of human CD8+ T cells to block their TCR-mediated ERK activity and proliferation

Tregs utilize beta-galactoside-binding protein to transiently inhibit PI3K/p21ras activity of human CD8+ T cells to block their TCR-mediated ERK activity and proliferation

http://www.wikidata.org/entity/Q34608828

about

Neoplastic "Black Ops": cancer's subversive tactics in overcoming host defenses Inhibition of lung metastasis by chemokine CCL17-mediated in vivo silencing of genes in CCR4+ Tregs.Molecular mechanisms of treg-mediated T cell suppression Re-wiring regulatory cell networks in immunity by galectin-glycan interactions.Thiodigalactoside shows antitumour activity by beta-galactoside-binding protein and regulatory T cells inhibition in oral squamous cell carcinoma.PI3K is involved in β1 integrin clustering by PSGL-1 and promotes β1 integrin-mediated Jurkat cell adhesion to fibronectin.

P2860

Q27001081-70B0D9D5-A718-428A-B519-53274E06EA34 Q37000445-4FD52052-A617-4E7A-B285-50ACEAC6CB80 Q38008361-46986F00-2B7E-49D5-92B0-E0A008479AAD Q38583449-ECC645EA-B038-4116-8FC1-528E4F8EE79A Q52669705-9E229D4E-7455-49D5-A772-80AA46D80395 Q54403261-55379F91-CAC7-4499-A07A-143CAA0C220F

P2860

Tregs utilize beta-galactoside-binding protein to transiently inhibit PI3K/p21ras activity of human CD8+ T cells to block their TCR-mediated ERK activity and proliferation

http://www.wikidata.org/entity/Q34608828

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@ast

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@en

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@nl

type

label

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@ast

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@en

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@nl

prefLabel

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@ast

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@en

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@nl

P1433

Q34608828-B3A4BD1F-7B30-4D50-8244-1080FEA11613

P1476

Q34608828-07254C76-1845-4584-8C79-FB8AA874FF35

P2093

Q34608828-502C60D6-C945-4113-A7A8-4114A2F08665

Q34608828-793F183E-88C8-433F-BB8F-EBE52E8E11DB

Q34608828-8C03C3DC-40B1-42E0-91B4-8F554809950A

Q34608828-ADE2669A-50B1-4AF0-B315-F5E47453B0E7

Q34608828-B5EF7F24-5047-40B9-B509-8D41980DC84F

Q34608828-C9469D10-AF5A-447F-A58D-FFD55A610CD0

P2860

Q34608828-06E3CE7C-18C2-4146-86E0-8882F32EDCAA

Q34608828-18134A55-8FEE-4F5B-B324-4A8D830A8407

Q34608828-1B791146-F0D3-44CC-81FD-752FF873D17D

Q34608828-1EC3101B-958F-4DFF-BCE8-41B94B314056

Q34608828-1F46BB4D-61D7-4E87-9A1D-0A9CB3FD422A

Q34608828-2F0F51CB-7601-4537-B957-CFB474D57FD2

Q34608828-36BC815B-E70A-4ED9-9FA5-2FEAE4FA4298

Q34608828-3B799B7E-D737-44DA-BE15-3C506A01AB92

Q34608828-46D4C94D-0D9B-4437-BBBC-D5990F9D1AE0

Q34608828-480134AC-E9BA-40FF-9AE1-1CC1327AD22C

P304

Q34608828-AD0B398B-5450-4AAC-9E6D-3CB45AAF409C

P31

Q34608828-E2F078AF-1255-4A3F-97C5-6044445CFD32

P356

Q34608828-D4A7415A-F0B3-4914-8A57-B4FD4678E4CB

P433

Q34608828-E93C4B40-FC38-47A5-B7D8-FC363337A634

P478

Q34608828-1685C87E-CC3E-4570-9D2E-E9CF31CC5D67

P577

Q34608828-AB3E4B7D-9F52-4BA2-8FF7-0930F14B74FB

P5875

Q34608828-00BCA2E6-CEEC-469E-BF3D-DBC966E99F8A

P698

Q34608828-AE4EC241-55B5-421F-B68C-ECE7CBE00C2C

P932

Q34608828-8573F44C-F2FE-436F-9119-29D2208663C9

P356

J.BBI.2009.06.003

P1433

Brain, Behavior, and Immunity

P1476

Tregs utilize beta-galactoside ...... ERK activity and proliferation

@en

P2093

Arya Biragyn

http://www.w3.org/2001/XMLSchema#string

Dolgor Baatar

http://www.w3.org/2001/XMLSchema#string

Fred E Indig

http://www.w3.org/2001/XMLSchema#string

Livio Mallucci

http://www.w3.org/2001/XMLSchema#string

Purevdorj B Olkhanud

http://www.w3.org/2001/XMLSchema#string

Valerie Wells

http://www.w3.org/2001/XMLSchema#string

P2860

Immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor alpha-chains (CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases

Human CD4+ T lymphocytes with remarkable regulatory functions on dendritic cells and nickel-specific Th1 immune responses

Specific recruitment of regulatory T cells in ovarian carcinoma fosters immune privilege and predicts reduced survival

Galectin-1 supports survival of naive T cells without promoting cell proliferation.

Identification of an autocrine negative growth factor: mouse beta-galactoside-binding protein is a cytostatic factor and cell growth regulator.

Galectins and gliomas.

Phosphorylation of Tyr319 in ZAP-70 is required for T-cell antigen receptor-dependent phospholipase C-gamma1 and Ras activation.

Regulatory T cells: key controllers of immunologic self-tolerance.

Zap-70-independent Ca(2+) mobilization and Erk activation in Jurkat T cells in response to T-cell antigen receptor ligation.

Loss of functional suppression by CD4+CD25+ regulatory T cells in patients with multiple sclerosis.

P304

1028-1037

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.BBI.2009.06.003

http://www.w3.org/2001/XMLSchema#string

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

23

http://www.w3.org/2001/XMLSchema#string

P577

2009-06-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

26286243

http://www.w3.org/2001/XMLSchema#string

P698

19520156

http://www.w3.org/2001/XMLSchema#string

P932

2752359

http://www.w3.org/2001/XMLSchema#string