An essential role for Notch in neural crest during cardiovascular development and smooth muscle differentiation. - Wikidata - wikimedia - TriplyDB

An essential role for Notch in neural crest during cardiovascular development and smooth muscle differentiation.

An essential role for Notch in neural crest during cardiovascular development and smooth muscle differentiation.

http://www.wikidata.org/entity/Q34608845

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Developmental coronary maturation is disturbed by aberrant cardiac vascular endothelial growth factor expression and Notch signalling Hairy-related transcription factors inhibit Notch-induced smooth muscle alpha-actin expression by interfering with Notch intracellular domain/CBF-1 complex interaction with the CBF-1-binding site Characterization and in vivo pharmacological rescue of a Wnt2-Gata6 pathway required for cardiac inflow tract development NOTCH3 expression is induced in mural cells through an autoregulatory loop that requires endothelial-expressed JAGGED1 Coordinating tissue interactions: Notch signaling in cardiac development and disease Programming and reprogramming a human heart cell Induced pluripotent stem cell-derived vascular smooth muscle cells: methods and application Role of Notch signaling in the mammalian heart Connecting teratogen-induced congenital heart defects to neural crest cells and their effect on cardiac function The neural crest in cardiac congenital anomalies Origin and differentiation of vascular smooth muscle cells Endothelial expression of the Notch ligand Jagged1 is required for vascular smooth muscle development Biochemical characterization and cellular effects of CADASIL mutants of NOTCH3 Murine Jagged1/Notch signaling in the second heart field orchestrates Fgf8 expression and tissue-tissue interactions during outflow tract development Hes1 is expressed in the second heart field and is required for outflow tract development Endoglin is required for myogenic differentiation potential of neural crest stem cells Hes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndrome Mind bomb 1 is required for pancreatic β-cell formation Tbx1 controls cardiac neural crest cell migration during arch artery development by regulating Gbx2 expression in the pharyngeal ectoderm Notch signaling regulates murine atrioventricular conduction and the formation of accessory pathways.Ankrd17, an ubiquitously expressed ankyrin factor, is essential for the vascular integrity during embryogenesis Requirement for integrin-linked kinase in neural crest migration and differentiation and outflow tract morphogenesis Notch signaling in vascular smooth muscle cells is required to pattern the cerebral vasculature Cardiac neural crest orchestrates remodeling and functional maturation of mouse semilunar valves.Postembryonic neuronal addition in zebrafish dorsal root ganglia is regulated by Notch signaling.Cardiovascular malformations caused by NOTCH1 mutations do not keep left: data on 428 probands with left-sided CHD and their families.Deletion of ETS-1, a gene in the Jacobsen syndrome critical region, causes ventricular septal defects and abnormal ventricular morphology in mice.Embryonic aortic arch hemodynamics are a functional biomarker for ethanol-induced congenital heart defects [Invited]Smad2 and myocardin-related transcription factor B cooperatively regulate vascular smooth muscle differentiation from neural crest cells.Prediction of miRNA-mRNA associations in Alzheimer's disease mice using network topology.Endoglin plays distinct roles in vascular smooth muscle cell recruitment and regulation of arteriovenous identity during angiogenesis.Epithelial Notch signaling regulates interstitial fibrosis development in the kidneys of mice and humans Radial construction of an arterial wall Vascular smooth muscle phenotypic diversity and function Fibronectins in vascular morphogenesis Spatio-temporal distribution of Smads and role of Smads/TGF-β/BMP-4 in the regulation of mouse bladder organogenesis Mastermind-like transcriptional co-activators: emerging roles in regulating cross talk among multiple signaling pathways.Repression of Sox9 by Jag1 is continuously required to suppress the default chondrogenic fate of vascular smooth muscle cells.Temporal and embryonic lineage-dependent regulation of human vascular SMC development by NOTCH3 MicroRNA-processing enzyme Dicer is required in epicardium for coronary vasculature development.

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An essential role for Notch in neural crest during cardiovascular development and smooth muscle differentiation.

http://www.wikidata.org/entity/Q34608845

description

2007 nî lūn-bûn

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2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2007 թվականի փետրվարին հրատարակված գիտական հոդված

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2007年の論文

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An essential role for Notch in ...... smooth muscle differentiation.

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An essential role for Notch in ...... smooth muscle differentiation.

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An essential role for Notch in ...... smooth muscle differentiation.

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An essential role for Notch in ...... smooth muscle differentiation

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Aaron Proweller

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Frances A High

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Jonathan A Epstein

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Lili Tu

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Maozhen Zhang

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Warren S Pear

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P2860

Dll4, a novel Notch ligand expressed in arterial endothelium

The Notch coactivator, MAML1, functions as a novel coactivator for MEF2C-mediated transcription and is required for normal myogenesis

Structural basis for cooperativity in recruitment of MAML coactivators to Notch transcription complexes

Mutations in NOTCH1 cause aortic valve disease

Tetralogy of fallot and other congenital heart defects in Hey2 mutant mice

Mutations in the human Jagged1 gene are responsible for Alagille syndrome

Alagille syndrome is caused by mutations in human Jagged1, which encodes a ligand for Notch1

HRC is a direct transcriptional target of MEF2 during cardiac, skeletal, and arterial smooth muscle development in vivo

Notch signaling: cell fate control and signal integration in development

Fate of the mammalian cardiac neural crest

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117

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Michael S Parmacek

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17273555

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Aparato circulatorio

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1783803

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