Cancer genes: rare recombinants instead of activated oncogenes (a review).
about
Human immunodeficiency virus and acquired immunodeficiency syndrome: correlation but not causationA stochastic model of oncogene expression and the relevance of this model to cancer therapy.Illegitimate recombination induced by benzo[a]pyrene diol epoxide in Escherichia coli.Evidence that retroviral transduction is mediated by DNA not by RNA.Retroviral transduction of oncogenic sequences involves viral DNA instead of RNA.Generation of fibrosarcomas in vivo by a retrovirus that expresses the normal B chain of platelet-derived growth factor and mimics the alternative splice pattern of the v-sis oncogeneA retroviral promoter is sufficient to convert proto-src to a transforming gene that is distinct from the src gene of Rous sarcoma virus.Avian proto-myc genes promoted by defective or nondefective retroviruses are single-hit transforming genes in primary cells.Avian erythroblastosis virus E26: only one (myb) of two cell-derived coding regions is necessary for oncogenicityCellular aging is a critical determinant of primary cell resistance to v-src transformation.Recombinant BALB and Harvey sarcoma viruses with normal proto-ras-coding regions transform embryo cells in culture and cause tumors in mice.Harvey murine sarcoma virus: influences of coding and noncoding sequences on cell transformation in vitro and oncogenicity in vivo.Transformation of human fibroblasts by ionizing radiation, a chemical carcinogen, or simian virus 40 correlates with an increase in susceptibility to the autonomous parvoviruses H-1 virus and minute virus of mice.Unmutated proto-src coding region is tumorigenic if expressed from the promoter of Rous sarcoma virus: implications for the gene-mutation hypothesis of cancer.Transforming function of proto-ras genes depends on heterologous promoters and is enhanced by specific point mutations.An overview of tumor biology.Human cancer and the food chain: an alternative etiologic perspective.Cancer genes by illegitimate recombination.Two autonomous myc oncogenes in avian carcinoma virus OK10.Genome reorganization through cell division. Implications for aging of the organism and cancer development.Multiple pathways leading to genomic instability and tumorigenesis.Nonhomologous recombination in human cells.Modelling haemopoietic stem cell division by analysis of mutant red cells.
P2860
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P2860
Cancer genes: rare recombinants instead of activated oncogenes (a review).
description
1987 nî lūn-bûn
@nan
1987 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1987年の論文
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1987年学术文章
@wuu
1987年学术文章
@zh-cn
1987年学术文章
@zh-hans
1987年学术文章
@zh-my
1987年学术文章
@zh-sg
1987年學術文章
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name
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@ast
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@en
Cancer genes: rare recombinants instead of activated oncogenes
@nl
type
label
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@ast
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@en
Cancer genes: rare recombinants instead of activated oncogenes
@nl
prefLabel
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@ast
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@en
Cancer genes: rare recombinants instead of activated oncogenes
@nl
P2860
P356
P1476
Cancer genes: rare recombinants instead of activated oncogenes (a review).
@en
P2860
P304
P356
10.1073/PNAS.84.8.2117
P407
P577
1987-04-01T00:00:00Z