Statistical modeling of interlocus interactions in a complex disease: rejection of the multiplicative model of epistasis in type 1 diabetes

Statistical modeling of interlocus interactions in a complex disease: rejection of the multiplicative model of epistasis in type 1 diabetes

Item
http://www.wikidata.org/entity/Q34612353
Cellular mechanisms of restored β-cell tolerance mediated by protective alleles of Idd3 and Idd5Integrative analysis for finding genes and networks involved in diabetes and other complex diseasesSearch for haplotype interactions that influence susceptibility to type 1 diabetes, through use of unphased genotype data.Retrospective analysis of main and interaction effects in genetic association studies of human complex traitsAn omnibus permutation test on ensembles of two-locus analyses can detect pure epistasis and genetic heterogeneity in genome-wide association studies.Nonobese diabetic congenic strain analysis of autoimmune diabetes reveals genetic complexity of the Idd18 locus and identifies Vav3 as a candidate gene.Identification of two cerebral malaria resistance loci using an inbred wild-derived mouse strainGenome-wide interaction analysis reveals replicated epistatic effects on brain structure.Evidence for association of OCTN genes and IBD5 with ulcerative colitisVoluntary ethanol consumption by mice: genome-wide analysis of quantitative trait loci and their interactions in a C57BL/6ByJ x 129P3/J F2 intercross.A cooperative interaction between LPHN3 and 11q doubles the risk for ADHD.Ptpn22 and Cd2 Variations Are Associated with Altered Protein Expression and Susceptibility to Type 1 Diabetes in Nonobese Diabetic Mice.An ensemble learning approach jointly modeling main and interaction effects in genetic association studies.Genetic interactions among Idd3, Idd5.1, Idd5.2, and Idd5.3 protective loci in the nonobese diabetic mouse model of type 1 diabetes.Epistasis and its implications for personal genetics.Genome-wide microarray expression analysis of CD4+ T Cells from nonobese diabetic congenic mice identifies Cd55 (Daf1) and Acadl as candidate genes for type 1 diabetes.Neuroimaging genomics in psychiatry-a translational approach.
P2860

Statistical modeling of interlocus interactions in a complex disease: rejection of the multiplicative model of epistasis in type 1 diabetes

Item
http://www.wikidata.org/entity/Q34612353
description
2001 nî lūn-bûn
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2001 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2001 թվականի մայիսին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年论文
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name
Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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P1433
P1476
P2093
P2860
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Statistical modeling of interl ...... f epistasis in type 1 diabetes
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P2093
P2860
P304
P31
P407
P433
P478
P50
P577
P698
P921
P932