Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H(2)O(2) and impaired cytokine pattern in cultured keratinocytes and blood of vitiligo patients.

Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H(2)O(2) and impaired cytokine pattern in cultured keratinocytes and blood of vitiligo patients.

Item
http://www.wikidata.org/entity/Q34615683
In vitro research on vitiligo: strategies, principles, methodological options and common pitfallsDysfunction of Autophagy: A Possible Mechanism Involved in the Pathogenesis of Vitiligo by Breaking the Redox Balance of MelanocytesProtective actions of 17β-estradiol and progesterone on oxidative neuronal injury induced by organometallic compounds.Vitiligo: a possible model of degenerative diseases.Oxidative stress and mitochondrial dysfunction across broad-ranging pathologies: toward mitochondria-targeted clinical strategies.Koebner's phenomenon in vitiligo: European position paper.Integrated Haematological Profiles of Redox Status, Lipid, and Inflammatory Protein Biomarkers in Benign Obesity and Unhealthy Obesity with Metabolic Syndrome.Stress chaperone mortalin regulates human melanogenesis.Topical apigenin improves epidermal permeability barrier homoeostasis in normal murine skin by divergent mechanisms.Vitiligo: an update on current pharmacotherapy and future directions.Advances in the treatment options for vitiligo: activated low-dose cytokines-based therapy.Metabolic and redox barriers in the skin exposed to drugs and xenobiotics.Glutathione S-transferase M1 and T1 genetic polymorphism in Egyptian patients with nonsegmental vitiligo.Previous physical exercise alters the hepatic profile of oxidative-inflammatory status and limits the secondary brain damage induced by severe traumatic brain injury in rats.Vitiligo
P2860

Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H(2)O(2) and impaired cytokine pattern in cultured keratinocytes and blood of vitiligo patients.

Item
http://www.wikidata.org/entity/Q34615683
description
2010 nî lūn-bûn
@nan
2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H
@nl
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@ast
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@en
type
label
Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H
@nl
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@ast
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@en
prefLabel
Dysfunction of glutathione S-transferase leads to excess 4-hydroxy-2-nonenal and H
@nl
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@ast
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@en
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P356
P1433
P1476
Dysfunction of glutathione S-t ...... nd blood of vitiligo patients.
@en
P2093
P304
P31
P356
P433
P478
P577
P698