Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children. - Wikidata - wikimedia - TriplyDB

Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children.

Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children.

http://www.wikidata.org/entity/Q34616166

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Lipidomics identifies cardiolipin oxidation as a mitochondrial target for redox therapy of brain injury Mass-spectrometry based oxidative lipidomics and lipid imaging: applications in traumatic brain injury A high-throughput screening assay of ascorbate in brain samples Emerging therapies in traumatic brain injury Sex differences in mitochondrial (dys)function: Implications for neuroprotection Antioxidant-chemoprevention diet ameliorates late effects of total-body irradiation and supplements radioprotection by MnSOD-plasmid liposome administration Clinical characteristics and pathophysiological mechanisms of focal and diffuse traumatic brain injury Therapies targeting lipid peroxidation in traumatic brain injury The effect of mild traumatic brain injury on peripheral nervous system pathology in wild-type mice and the G93A mutant mouse model of motor neuron disease The potential for bio-mediators and biomarkers in pediatric traumatic brain injury and neurocritical care S-glutathionylation: from molecular mechanisms to health outcomes Dietary Docosahexaenoic Acid Improves Cognitive Function, Tissue Sparing, and Magnetic Resonance Imaging Indices of Edema and White Matter Injury in the Immature Rat after Traumatic Brain Injury The 1H NMR profile of healthy dog cerebrospinal fluid.Chronic cerebrovascular dysfunction after traumatic brain injury Chronic vitamin C deficiency promotes redox imbalance in the brain but does not alter sodium-dependent vitamin C transporter 2 expression.Effect of short periods of normobaric hyperoxia on local brain tissue oxygenation and cerebrospinal fluid oxidative stress markers in severe traumatic brain injury.L-ascorbate attenuates the endotoxin-induced production of inflammatory mediators by inhibiting MAPK activation and NF-κB translocation in cortical neurons/glia Cocultures Peroxiredoxin VI oxidation in cerebrospinal fluid correlates with traumatic brain injury outcome Cyclopentenone eicosanoids as mediators of neurodegeneration: a pathogenic mechanism of oxidative stress-mediated and cyclooxygenase-mediated neurotoxicity.Evaluation of autophagy using mouse models of brain injury.Factors associated with hemispheric hypodensity after subdural hematoma following abusive head trauma in children.Are isofurans and neuroprostanes increased after subarachnoid hemorrhage and traumatic brain injury?Increase in oxidative stress as measured by cerebrospinal fluid lipid peroxidation during treatment for childhood acute lymphoblastic leukemia.Vascular endothelial growth factor is increased in cerebrospinal fluid after traumatic brain injury in infants and children.Glutathione peroxidase overexpression does not rescue impaired neurogenesis in the injured immature brain Heme regulation in traumatic brain injury: relevance to the adult and developing brain.Social interaction attenuates the extent of secondary neuronal damage following closed head injury in mice Antioxidative and steroid systems in neurological and psychiatric disorders.A blood-based biomarker panel to risk-stratify mild traumatic brain injury.Current status of fluid biomarkers in mild traumatic brain injury Traumatic injury to the immature brain: inflammation, oxidative injury, and iron-mediated damage as potential therapeutic targets.Expression of ATP-Binding Cassette Transporters B1 and C1 after Severe Traumatic Brain Injury in Humans.Ascorbic acid: chemistry, biology and the treatment of cancer.Traumatic Brain Injury Alters Methionine Metabolism: Implications for Pathophysiology.Electrophilic cyclopentenone isoprostanes in neurodegeneration.Therapeutic hypothermia preserves antioxidant defenses after severe traumatic brain injury in infants and children.Biofuel cell based on microscale nanostructured electrodes with inductive coupling to rat brain neurons.Drug targets for traumatic brain injury from poly(ADP-ribose)polymerase pathway modulation.New concepts in treatment of pediatric traumatic brain injury.Mechanical stretch exacerbates the cell death in SH-SY5Y cells exposed to paraquat: mitochondrial dysfunction and oxidative stress

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P2860

Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children.

http://www.wikidata.org/entity/Q34616166

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2002 nî lūn-bûn

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Vladimir Tyurin

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Yulia Y Tyurina

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P2860

Oxidants, antioxidants, and the degenerative diseases of aging

v-Crk modulation of growth factor-induced PC12 cell differentiation involves the Src homology 2 domain of v-Crk and sustained activation of the Ras/mitogen-activated protein kinase pathway

Role of free radicals and catalytic metal ions in human disease: an overview

Reactive oxygen species, cell signaling, and cell injury.

Ascorbate is an outstanding antioxidant in human blood plasma.

Reactive oxygen species and the central nervous system.

On the antioxidant effects of ascorbic acid and glutathione.

21-Aminosteroid reduces ion shifts and edema in the rat middle cerebral artery occlusion model of regional ischemia.

Mild intraischemic hypothermia suppresses consumption of endogenous antioxidants after temporary focal ischemia in rats.

Non-cyclooxygenase-derived prostanoids (F2-isoprostanes) are formed in situ on phospholipids.

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Traumatic stress

traumatic brain injury

cerebrospinal fluid