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Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

http://www.wikidata.org/entity/Q34626763

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A comprehensive glossary of autophagy-related molecules and processes (2nd edition)Autophagy, mitochondria and oxidative stress: cross-talk and redox signalling Lacritin rescues stressed epithelia via rapid forkhead box O3 (FOXO3)-associated autophagy that restores metabolism Fundamentals of cancer metabolism Autophagy, cell death, and cancer KRAS Mutant Pancreatic Cancer: No Lone Path to an Effective Treatment Genetics and biology of pancreatic ductal adenocarcinoma Autophagy in hepatocellular carcinomas: from pathophysiology to therapeutic response Oncogene Overdose: Too Much of a Bad Thing for Oncogene-Addicted Cancer Cells Autophagy in colorectal cancer: An important switch from physiology to pathology Pancreatic Cancer Metabolism: Breaking It Down to Build It Back Up Targeting autophagy in cancer management - strategies and developments Oncogenes strike a balance between cellular growth and homeostasis Targeting LKB1 in cancer - exposing and exploiting vulnerabilities Chloroquine and hydroxychloroquine for cancer therapy Self-consumption: the interplay of autophagy and apoptosis Autophagy paradox and ceramide Mitophagy in hematopoietic stem cells: the case for exploration Autophagy and disease: always two sides to a problem Autophagy in cellular metabolism and cancer Autophagy: a targetable linchpin of cancer cell metabolism Autophagy modulation as a potential therapeutic target for diverse diseases Autophagy-mediated tumor promotion Targeting mitochondrial metabolism by inhibiting autophagy in BRAF-driven cancers Autophagy and cancer cell metabolism Targeting mitochondria metabolism for cancer therapy Autophagy suppresses RIP kinase-dependent necrosis enabling survival to mTOR inhibition The multifaceted roles of autophagy in tumors-implications for breast cancer.Nucleotide degradation and ribose salvage in yeast.Changing perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agents Autophagy Induced during Pancreatitis Promotes KRAS-Dependent Transformation in the Pancreas Therapeutic Targeting of Autophagy Autophagy-Dependent Secretion: Contribution to Tumor Progression Interplay between ROS and autophagy in cancer cells, from tumor initiation to cancer therapy Metabolic pathways promoting cancer cell survival and growth The potential for chemical mixtures from the environment to enable the cancer hallmark of sustained proliferative signalling RAS oncogenes: weaving a tumorigenic web Ral GTPases in tumorigenesis: emerging from the shadows Ceramide induced mitophagy and tumor suppression Sustained proliferation in cancer: Mechanisms and novel therapeutic targets

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Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

http://www.wikidata.org/entity/Q34626763

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@ast

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@en

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@nl

type

label

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@ast

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@en

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@nl

prefLabel

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@ast

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@en

Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis.

@nl

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Genes & Development

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Activated Ras requires autophagy to maintain oxidative metabolism and tumorigenesis

@en

P2093

Anne M Strohecker

http://www.w3.org/2001/XMLSchema#string

Celine Gelinas

http://www.w3.org/2001/XMLSchema#string

Gizem Karsli-Uzunbas

http://www.w3.org/2001/XMLSchema#string

Guanghua Chen

http://www.w3.org/2001/XMLSchema#string

Hilary A Coller

http://www.w3.org/2001/XMLSchema#string

Hsin-Yi Chen

http://www.w3.org/2001/XMLSchema#string

Jessie Yanxiang Guo

http://www.w3.org/2001/XMLSchema#string

Johanna M S Lemons

http://www.w3.org/2001/XMLSchema#string

Joshua D Rabinowitz

http://www.w3.org/2001/XMLSchema#string

Robert S Dipaola

http://www.w3.org/2001/XMLSchema#string

P2860

Hypoxia and defective apoptosis drive genomic instability and tumorigenesis

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

Understanding the Warburg effect: the metabolic requirements of cell proliferation

Autophagy suppresses tumorigenesis through elimination of p62

Autophagy in the Pathogenesis of Disease

Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

Loss of autophagy in the central nervous system causes neurodegeneration in mice

P304

460-470

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scholarly article

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10.1101/GAD.2016311

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5

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25

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Jurre J Kamphorst

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2011-02-11T00:00:00Z

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49832375

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21317241

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3049287

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