Gamma interferon is spontaneously released by alveolar macrophages and lung T lymphocytes in patients with pulmonary sarcoidosis.
about
PSMB2 and RPL32 are suitable denominators to normalize gene expression profiles in bronchoalveolar cellsAssociation between sarcoidosis and lymphoma revisitedHost microenvironment in breast cancer development: inflammatory and immune cells in tumour angiogenesis and arteriogenesisGranuloma formation in pulmonary sarcoidosisCarbon Nanotubes and Chronic Granulomatous DiseaseIFN-γ-Producing T-Helper 17.1 Cells Are Increased in Sarcoidosis and Are More Prevalent than T-Helper Type 1 CellsMycobacterial catalase-peroxidase is a tissue antigen and target of the adaptive immune response in systemic sarcoidosis.Mycobacterial ESAT-6 and katG are recognized by sarcoidosis CD4+ T cells when presented by the American sarcoidosis susceptibility allele, DRB1*1101.Feasibility and diagnostic utility of antigen-specific interferon-gamma responses for rapid immunodiagnosis of tuberculosis using induced sputum.Expression of class II major histocompatibility complex antigens on alveolar epithelium in interstitial lung disease: relevance to pathogenesis of idiopathic pulmonary fibrosis.Pulmonary alveolar macrophages from patients with active sarcoidosis express type IV collagenolytic proteinase. An enzymatic mechanism for influx of mononuclear phagocytes at sites of disease activity.Lung T-helper cells expressing T-cell receptor AV2S3 associate with clinical features of pulmonary sarcoidosis.Immune mechanisms in interstitial lung diseases.Cytokines involved in interferon-gamma production by human macrophagesIncreased numbers of T lymphocytes with gamma delta-positive antigen receptors in a subgroup of individuals with pulmonary sarcoidosis.The role of interferon-γ in the pathogenesis of acute intra-abdominal sepsis.STAT4 is a critical mediator of early innate immune responses against pulmonary Klebsiella infection.Chemokines and their receptors guiding T lymphocyte recruitment in lung inflammation.Modulation of alveolar macrophage-driven fibroblast proliferation by alternative macrophage mediators.Mathematical model of sarcoidosisSecretory products of macrophages.Spontaneous expression of the interleukin 2 receptor gene and presence of functional interleukin 2 receptors on T lymphocytes in the blood of individuals with active pulmonary sarcoidosisImmune interferon inhibits proliferation and induces 2'-5'-oligoadenylate synthetase gene expression in human vascular smooth muscle cellsRecombinant interferon-gamma primes alveolar macrophages cultured in vitro for the release of leukotriene B4 in response to IgG stimulationCurrent and emerging strategies for the management of sarcoidosis.M2 polarized macrophages and giant cells contribute to myofibrosis in neuromuscular sarcoidosisInterferon-induced sarcoidosisType 1 and type 2 cytokine dysregulation in human infectious, neoplastic, and inflammatory diseases.Clinical impact of bone and calcium metabolism changes in sarcoidosisIntracellular cytokine repertoire in different T cell subsets from patients with sarcoidosisLung restricted T cell receptor AV2S3+ CD4+ T cell expansions in sarcoidosis patients with a shared HLA-DRbeta chain conformation.Increased interleukin-13 expression in patients with sarcoidosisInvolvement of dendritic cells in sarcoidosis.Hydrogen peroxide release by alveolar macrophages from sarcoid patients and by alveolar macrophages from normals after exposure to recombinant interferons alpha A, beta, and gamma and 1,25-dihydroxyvitamin D3.Cytokine regulation of human lung fibroblast hyaluronan (hyaluronic acid) production. Evidence for cytokine-regulated hyaluronan (hyaluronic acid) degradation and human lung fibroblast-derived hyaluronidase.Sarcoidosis blood transcriptome reflects lung inflammation and overlaps with tuberculosis.Cellular recognition of Mycobacterium tuberculosis ESAT-6 and KatG peptides in systemic sarcoidosis.Regulation of human lung fibroblast glycosaminoglycan production by recombinant interferons, tumor necrosis factor, and lymphotoxin.Granuloma formation by artificial microparticles in vitro. Macrophages and monokines play a critical role in granuloma formation.Immunohistochemical analysis of sarcoid granulomas. Evaluation of Ki67+ and interleukin-1+ cells.
P2860
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P2860
Gamma interferon is spontaneously released by alveolar macrophages and lung T lymphocytes in patients with pulmonary sarcoidosis.
description
1985 nî lūn-bûn
@nan
1985 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1985 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1985年の論文
@ja
1985年論文
@yue
1985年論文
@zh-hant
1985年論文
@zh-hk
1985年論文
@zh-mo
1985年論文
@zh-tw
1985年论文
@wuu
name
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@ast
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@en
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@nl
type
label
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@ast
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@en
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@nl
prefLabel
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@ast
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@en
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@nl
P2093
P2860
P356
P1476
Gamma interferon is spontaneou ...... ts with pulmonary sarcoidosis.
@en
P2093
Crystal RG
McLemore TL
Robinson BW
P2860
P304
P356
10.1172/JCI111852
P407
P577
1985-05-01T00:00:00Z