about
The effect of Rho kinase inhibition on long-term keratinocyte proliferation is rapid and conditional.AP1S3 mutations are associated with pustular psoriasis and impaired Toll-like receptor 3 traffickingAutoinflammatory pustular neutrophilic diseasesRecent advances in understanding ichthyosis pathogenesisAutoimmune diseases - connecting risk alleles with molecular traits of the immune system.The paracaspase MALT1: biological function and potential for therapeutic inhibitionLighting the fires within: the cell biology of autoinflammatory diseasesImmunology of psoriasisDiscovery of single-gene inborn errors of immunity by next generation sequencingBeyond the NLRP3 inflammasome: autoinflammatory diseases reach adolescenceNovel factors in the pathogenesis of psoriasis and potential drug candidates are found with systems biology approachThe paracaspase MALT1 mediates CARD14-induced signaling in keratinocytesInteractions of the Immune System with Skin and Bone Tissue in Psoriatic Arthritis: A Comprehensive Review.Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity.The expanding spectrum of rare monogenic autoinflammatory diseases.RIG-I antiviral signaling drives interleukin-23 production and psoriasis-like skin disease.CARD14 alterations in Tunisian patients with psoriasis and further characterization in European cohorts.Rare and common variants in CARD14, encoding an epidermal regulator of NF-kappaB, in psoriasis.When less is more: primary immunodeficiency with an autoinflammatory kickPsoriasis mutations disrupt CARD14 autoinhibition promoting BCL10-MALT1-dependent NF-κB activation.Cathepsin S is the major activator of the psoriasis-associated proinflammatory cytokine IL-36γ.Differentiation between osteoarthritis and psoriatic arthritis: implications for pathogenesis and treatment in the biologic therapy eraGenetic Epidemiology of PsoriasisIntegration of sequence data from a Consanguineous family with genetic data from an outbred population identifies PLB1 as a candidate rheumatoid arthritis risk geneImmunological loss-of-function due to genetic gain-of-function in humans: autosomal dominance of the third kindPsoriasis is not an autoimmune disease?CARD14 expression in dermal endothelial cells in psoriasis.Analysis of CARD14 Polymorphisms in Pityriasis Rubra Pilaris: Activation of NF-κBCARMA3 Is Critical for the Initiation of Allergic Airway Inflammation.New monogenic autoinflammatory diseases--a clinical overviewMolecular mechanisms in genetically defined autoinflammatory diseases: disorders of amplified danger signaling.Familial pityriasis rubra pilaris is caused by mutations in CARD14.Novel CARD11 Mutations in Human Cutaneous Squamous Cell Carcinoma Lead to Aberrant NF-κB Regulation.The immunogenetics of Psoriasis: A comprehensive review.Update on psoriasis immunopathogenesis and targeted immunotherapy.Vitamin D receptor gene polymorphisms and haplotypes (Apa I, Bsm I, Fok I, Taq I) in Turkish psoriasis patientsTREM-1 as a potential therapeutic target in psoriasisThe c-Rel subunit of NF-κB regulates epidermal homeostasis and promotes skin fibrosis in mice.The IL-23/T17 pathogenic axis in psoriasis is amplified by keratinocyte responses.Monogenic autoinflammatory diseases: concept and clinical manifestations.
P2860
Q21999272-54F7F7B8-4C9A-4C6F-81DA-3E9971156F5BQ24296788-9DE332F8-B4F8-4065-8600-B84CF355424EQ24603182-34394CB7-ED52-4E88-AB51-54D955DF04F2Q26740640-90389781-EB14-4386-936F-175137BB5E15Q26767040-BDAFE815-D4E4-4616-B292-EB3859A75E42Q26778357-88529209-F6AE-487A-BC98-B47BEC197C50Q26827163-A7851FEC-8844-429F-90A5-AB29AC450E1BQ26995800-80CDC797-94DE-4115-8648-F39C3EF333A3Q27003187-18830EF4-244A-44E6-9CF2-E83CF28F1A5EQ27011650-A1657FE3-8123-4988-8E43-859E41C10C27Q28535430-7374115F-70E0-45F8-9076-2B15E66428B3Q28975763-2FBD20FE-7AEE-4033-A39A-EA44080B1865Q30251957-8FA1E45E-F6EE-4040-A862-9BD553BFF5FCQ30419116-7BE04179-E713-4E37-8557-C51E771444AAQ33592763-C7D41113-CD6F-449A-AB6D-2D427FB92F7CQ33625887-D2B9160B-CC2A-456B-ABA4-E6FDBB5F1CB1Q33726533-AA92A90C-B20D-4168-90AA-8FFAFDE11026Q34269865-A58B982B-E8C7-4F9C-8167-2513125B43C4Q34420031-5EAAFE4A-EF72-439A-AAC8-26EE996CF086Q34521887-7D2C1980-80E8-4066-9C28-0A26E508BF21Q34553191-B87B3F2F-C995-45BD-A067-43F59D78973FQ34727681-4F131D24-F2BE-4656-9298-A402CD841C88Q34845927-9DD06CD6-BB41-4C85-9FF8-948928FB79B2Q35092644-88621BAC-7B6E-4146-A964-7D9AE2292D46Q35190461-26232655-2102-46AE-83DE-9AC9990E5D6FQ35369427-5DAE8148-8D80-4CBC-B9FF-4B69A5AC3412Q35392242-78D24598-231D-438E-BE92-07F014C5D265Q35738571-BF3607F5-EE03-4CB4-A657-7B2900B2164DQ35809995-CB79BCBE-5C6A-4D72-9E6C-3D8A7742709EQ36016335-289AE8FF-CA0A-4C62-B65A-007B3846C206Q36041225-07C511DC-350B-422A-8750-472177C1A0FBQ36096111-0589E8C7-EF4F-4E42-A13A-75CC0C17A3EAQ36134453-AA7A1C3E-594E-4F03-BCE9-E7596818F85EQ36236780-3CE6C97E-6B00-45E1-A35C-0907102CBAAAQ36445919-B4274793-2BB1-4568-AEEA-AE2E5A2A2843Q36576962-9D6535DE-EEAC-4A42-BAD9-01B8D45BEA21Q36972038-0C021BF0-C02F-4808-B7C2-1760C7E099E9Q36989123-43043DE3-7090-490C-B522-2CD93A3B69F0Q37040371-F2F55AA1-7EF2-4B9F-A186-51CDE530DFEAQ37162157-F28F0A6D-77A6-4D0A-953A-5157A9B53257
P2860
description
2012 nî lūn-bûn
@nan
2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
PSORS2 is due to mutations in CARD14.
@ast
PSORS2 is due to mutations in CARD14.
@en
PSORS2 is due to mutations in CARD14.
@nl
type
label
PSORS2 is due to mutations in CARD14.
@ast
PSORS2 is due to mutations in CARD14.
@en
PSORS2 is due to mutations in CARD14.
@nl
prefLabel
PSORS2 is due to mutations in CARD14.
@ast
PSORS2 is due to mutations in CARD14.
@en
PSORS2 is due to mutations in CARD14.
@nl
P2093
P2860
P50
P1476
PSORS2 is due to mutations in CARD14
@en
P2093
Alan Menter
Cailin E Joyce
Catherine T Jordan
Chi-Fan Yang
Cynthia A Helms
Jer-Yuarn Wu
Katherine C Pierson
Raphaela Goldbach-Mansky
Shenghui Duan
P2860
P304
P356
10.1016/J.AJHG.2012.03.012
P407
P577
2012-04-19T00:00:00Z