Limited sufficiency of antigen presentation by dendritic cells in models of central nervous system autoimmunity.
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The role of dendritic cells in autoimmunityApolipoprotein E mediation of neuro-inflammation in a murine model of multiple sclerosis.Antibody blockade of CLEC12A delays EAE onset and attenuates disease severity by impairing myeloid cell CNS infiltration and restoring positive immunity.Secondary B cell receptor diversification is necessary for T cell mediated neuro-inflammation during experimental autoimmune encephalomyelitis.Role and therapeutic value of dendritic cells in central nervous system autoimmunity.Flt3L controls the development of radiosensitive dendritic cells in the meninges and choroid plexus of the steady-state mouse brain.The critical role of antigen-presentation-induced cytokine crosstalk in the central nervous system in multiple sclerosis and experimental autoimmune encephalomyelitis.B cell antigen presentation is sufficient to drive neuroinflammation in an animal model of multiple sclerosis.Environmental pathways to autoimmune diseases: the cases of primary biliary cirrhosis and multiple sclerosis.Mechanisms of dendritic cell trafficking across the blood-brain barrier.OGR1/GPR68 Modulates the Severity of Experimental Autoimmune Encephalomyelitis and Regulates Nitric Oxide Production by Macrophages.Differential targeting of IL-2 and T cell receptor signaling pathways selectively expands regulatory T cells while inhibiting conventional T cellsSynergistic effect of methionine encephalin (MENK) combined with pidotimod(PTD) on the maturation of murine dendritic cells (DCs)B cells promote induction of experimental autoimmune encephalomyelitis by facilitating reactivation of T cells in the central nervous system.Autoimmunity in 2011.Differential roles of resident microglia and infiltrating monocytes in murine CNS autoimmunity.Functional connectivity alterations in a murine model of optic neuritis.Cutting edge: Conditional MHC class II expression reveals a limited role for B cell antigen presentation in primary and secondary CD4 T cell responses.Optic Neuritis: A Model for the Immuno-pathogenesis of Central Nervous System Inflammatory Demyelinating Diseases.B cells are capable of independently eliciting rapid reactivation of encephalitogenic CD4 T cells in a murine model of multiple sclerosis.
P2860
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P2860
Limited sufficiency of antigen presentation by dendritic cells in models of central nervous system autoimmunity.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@ast
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@en
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@nl
type
label
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@ast
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@en
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@nl
prefLabel
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@ast
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@en
Limited sufficiency of antigen ...... l nervous system autoimmunity.
@nl
P2093
P2860
P1476
Limited sufficiency of antigen ...... al nervous system autoimmunity
@en
P2093
Gregory F Wu
Hannah L Thomas
Kenneth S Shindler
Robert J Mikesell
Terri M Laufer
Tom L Stephen
P2860
P356
10.1016/J.JAUT.2010.10.006
P577
2010-11-20T00:00:00Z