Toll-like receptor-induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens.
about
Mesenchymal Stem Cells and Myeloid Derived Suppressor Cells: Common Traits in Immune RegulationTargeting innate immunity for neurodegenerative disorders of the central nervous systemArginine Metabolism in Bacterial Pathogenesis and Cancer TherapyThe Regulation of CD4(+) T Cell Responses during Protozoan InfectionsMacrophage proliferation, provenance, and plasticity in macroparasite infectionImmunometabolism in TuberculosisIL-4Rα-dependent alternative activation of macrophages is not decisive for Mycobacterium tuberculosis pathology and bacterial burden in miceAntibodies trap tissue migrating helminth larvae and prevent tissue damage by driving IL-4Rα-independent alternative differentiation of macrophagesMAP kinase phosphatase-2 plays a key role in the control of infection with Toxoplasma gondii by modulating iNOS and arginase-1 activities in miceChitinase dependent control of protozoan cyst burden in the brainTH1/TH2 paradigm extended: macrophage polarization as an unappreciated pathogen-driven escape mechanism?Metabolic reprogramming in macrophages and dendritic cells in innate immunityContribution of metabolic reprogramming to macrophage plasticity and functionSelective endothelial overexpression of arginase II induces endothelial dysfunction and hypertension and enhances atherosclerosis in micePhosphodiesterase-4 inhibition combined with isoniazid treatment of rabbits with pulmonary tuberculosis reduces macrophage activation and lung pathologyInducible arginase 1 deficiency in mice leads to hyperargininemia and altered amino acid metabolismGrowth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasisStrategies to rescue the consequences of inducible arginase-1 deficiency in miceThe E3 ubiquitin ligase neuregulin receptor degradation protein 1 (Nrdp1) promotes M2 macrophage polarization by ubiquitinating and activating transcription factor CCAAT/enhancer-binding Protein β (C/EBPβ)Macrophage polarization drives granuloma outcome during Mycobacterium tuberculosis infectionProtective and pathogenic functions of macrophage subsetsMacrophage activation and polarization: nomenclature and experimental guidelinesFatal outcome of pandemic H1N1 2009 influenza virus infection is associated with immunopathology and impaired lung repair, not enhanced viral burden, in pregnant miceMicrobial manipulation of receptor crosstalk in innate immunityProfiling early lung immune responses in the mouse model of tuberculosis.Arginase-1-expressing macrophages suppress Th2 cytokine-driven inflammation and fibrosisBone marrow cell derived arginase I is the major source of allergen-induced lung arginase but is not required for airway hyperresponsiveness, remodeling and lung inflammatory responses in mice.Proteophosophoglycans regurgitated by Leishmania-infected sand flies target the L-arginine metabolism of host macrophages to promote parasite survivalMacrophage plasticity in experimental atherosclerosis.Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines.Metformin-treated cancer cells modulate macrophage polarization through AMPK-NF-κB signaling.Modulation of the arginase pathway in the context of microbial pathogenesis: a metabolic enzyme moonlighting as an immune modulatorAlternatively activated macrophages derived from monocytes and tissue macrophages are phenotypically and functionally distinct.Similarity and diversity in macrophage activation by nematodes, trematodes, and cestodes.Comparative studies of macrophage-biased responses in mice to infection with Toxoplasma gondii ToxoDB #9 strains of different virulence isolated from China.Differential activation and antagonistic function of HIF-{alpha} isoforms in macrophages are essential for NO homeostasis.Co-existence of classical and alternative activation programs in macrophages responding to Toxoplasma gondiiRegulation of macrophage arginase expression and tumor growth by the Ron receptor tyrosine kinaseArginase II restricts host defense to Helicobacter pylori by attenuating inducible nitric oxide synthase translation in macrophagesMAP kinase phosphatase-2 plays a critical role in response to infection by Leishmania mexicana.
P2860
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P2860
Toll-like receptor-induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens.
description
2008 nî lūn-bûn
@nan
2008 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@ast
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@en
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@nl
type
label
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@ast
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@en
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@nl
prefLabel
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@ast
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@en
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@nl
P2093
P2860
P50
P356
P1433
P1476
Toll-like receptor-induced arg ...... ainst intracellular pathogens.
@en
P2093
Amber M Smith
Ian M Orme
John T Pesce
Karim C El Kasmi
Katherine A Fitzgerald
Marcela Henao-Tamayo
Mi-Sun Koo
Peter J Murray
Randall J Basaraba
Robert W Thompson
P2860
P304
P356
10.1038/NI.1671
P407
P577
2008-11-02T00:00:00Z