Lung matrix metalloproteinase-9 correlates with cigarette smoking and obstruction of airflow.
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Serum metalloproteinase-9 is related to COPD severity and symptoms - cross-sectional data from a population based cohort-study.Mechanistic Evaluation of the Impact of Smoking and Chronic Obstructive Pulmonary Disease on the Nasal EpitheliumFEV1 inversely correlates with metalloproteinases 1, 7, 9 and CRP in COPD by biomass smoke exposure.HIV Reprograms Human Airway Basal Stem/Progenitor Cells to Acquire a Tissue-Destructive Phenotype.Tiotropium bromide inhibits TGF-β-induced MMP production from lung fibroblasts by interfering with Smad and MAPK pathways in vitroAcute exacerbations of COPD are associated with significant activation of matrix metalloproteinase 9 irrespectively of airway obstruction, emphysema and infection.Role of matrix metalloproteinases in the development of airway inflammation and remodeling.CD8+ T cells contribute to macrophage accumulation and airspace enlargement following repeated irritant exposure.Proteinases and oxidants as targets in the treatment of chronic obstructive pulmonary disease.Electronic cigarette exposure triggers neutrophil inflammatory responses.Modeling the influence of vitamin D deficiency on cigarette smoke-induced emphysema.Roles for proteinases in the pathogenesis of chronic obstructive pulmonary disease.Suppressive activity of tiotropium bromide on matrix metalloproteinase production from lung fibroblasts in vitro.Matrix metalloproteinases -8, -9 and -12 in smokers and patients with stage 0 COPD.Discovery of Emphysema Relevant Molecular Networks from an A/J Mouse Inhalation Study Using Reverse Engineering and Forward Simulation (REFS™).Impact of tobacco smoking on cytokine signaling via interleukin-17A in the peripheral airways.Identification of MMP-9 as a biomarker for detecting progression of chronic obstructive pulmonary disease.N-Acetyl-L-cysteine and pyrrolidine dithiocarbamate inhibited nuclear factor-kappaB activation in alveolar macrophages by different mechanisms.Peroxisome proliferator-activated receptor-γ in induced sputum is correlated with MMP-9/TIMP-1 imbalance and formation of emphysema in COPD patients.From COPD epidemiology to studies of pathophysiological disease mechanisms: challenges with regard to study design and recruitment process: Respiratory and Cardiovascular Effects in COPD (KOLIN).Activity of matrix metalloproteinase in airway epithelial cells of COPD patients.Cigarette smoke-induced hypercapnic emphysema in C3H mice is associated with increases of macrophage metalloelastase and substance P in the lungs.
P2860
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P2860
Lung matrix metalloproteinase-9 correlates with cigarette smoking and obstruction of airflow.
description
2003 nî lūn-bûn
@nan
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@ast
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@en
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@nl
type
label
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@ast
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@en
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@nl
prefLabel
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@ast
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@en
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@nl
P2093
P1476
Lung matrix metalloproteinase- ...... ng and obstruction of airflow.
@en
P2093
Dong Gyu Kim
In Gyu Hyun
Jae Cheol Lee
Ki-Suck Jung
Min Jong Kang
Myung Goo Lee
Myung Jae Park
Sung Koo Han
Yeon-Mok Oh
Young-Soo Shim
P304
P356
10.3346/JKMS.2003.18.6.821
P577
2003-12-01T00:00:00Z