Basophil-mediated protection against gastrointestinal helminths requires IgE-induced cytokine secretion. - Wikidata - wikimedia - TriplyDB

Basophil-mediated protection against gastrointestinal helminths requires IgE-induced cytokine secretion.

Basophil-mediated protection against gastrointestinal helminths requires IgE-induced cytokine secretion.

http://www.wikidata.org/entity/Q34661411

about

Immune antibodies and helminth products drive CXCR2-dependent macrophage-myofibroblast crosstalk to promote intestinal repair Concerted activity of IgG1 antibodies and IL-4/IL-25-dependent effector cells trap helminth larvae in the tissues following vaccination with defined secreted antigens, providing sterile immunity to challenge infection Antibody-mediated trapping of helminth larvae requires CD11b and Fcγ receptor I.Looking beyond the induction of Th2 responses to explain immunomodulation by helminths.Innate immunological function of TH2 cells in vivo.The Mast Cell-IgE Paradox: From Homeostasis to Anaphylaxis Role of basophils in protective immunity to parasitic infections.New insights into basophil heterogeneity.IgE and mast cells in host defense against parasites and venoms.Regulation of IL-4 Expression in Immunity and Diseases.MicroRNA-mediated regulation of immune responses to intestinal helminth infections.Mucosal mast cells are indispensable for the timely termination of Strongyloides ratti infection.Recent advances in understanding basophil functions in vivo.ILC2s regulate adaptive Th2 cell functions via PD-L1 checkpoint control.The role of rare innate immune cells in Type 2 immune activation against parasitic helminths.T Follicular Helper Cell-Derived IL-4 Is Required for IgE Production during Intestinal Helminth Infection.T cells are the critical source of IL-4/IL-13 in a mouse model of allergic asthma.Mast Cells and IgE can Enhance Survival During Innate and Acquired Host Responses to Venoms.The Evolution of Human Basophil Biology from Neglect towards Understanding of Their Immune Functions The Study of Host Immune Responses Elicited by the Model Murine Hookworms Nippostrongylus brasiliensis and Heligmosomoides polygyrus.Host-Parasite Interactions Promote Disease Tolerance to Intestinal Helminth Infection Egg-Released IPSE/alpha-1 Dampens Inflammatory Cytokine Responses Basophil Interleukin (IL)-4 and IL-13 Schistosoma "Eggs-Iting" the Host: Granuloma Formation and Egg Excretion

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Basophil-mediated protection against gastrointestinal helminths requires IgE-induced cytokine secretion.

http://www.wikidata.org/entity/Q34661411

description

2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Basophil-mediated protection a ...... gE-induced cytokine secretion.

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Basophil-mediated protection a ...... gE-induced cytokine secretion.

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Basophil-mediated protection a ...... gE-induced cytokine secretion.

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Basophil-mediated protection a ...... gE-induced cytokine secretion.

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Basophil-mediated protection a ...... gE-induced cytokine secretion.

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PNAS.1412663111

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Basophil-mediated protection a ...... gE-induced cytokine secretion.

@en

P2093

Adriana Turqueti-Neves

http://www.w3.org/2001/XMLSchema#string

Christian Schwartz

http://www.w3.org/2001/XMLSchema#string

David Voehringer

http://www.w3.org/2001/XMLSchema#string

Philipp Yu

http://www.w3.org/2001/XMLSchema#string

Susanne Hartmann

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P2860

Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity

Soil-transmitted helminth infections: ascariasis, trichuriasis, and hookworm

Innate and adaptive type 2 immune cell responses in genetically controlled resistance to intestinal helminth infection

Active anaphylaxis in IgE-deficient mice

Intestinal epithelial cell secretion of RELM-beta protects against gastrointestinal worm infection.

Trapping and immobilization of Nippostrongylus brasiliensis larvae at the site of inoculation in primary infections of interleukin-5 transgenic mice.

The lung is an important site for priming CD4 T-cell-mediated protective immunity against gastrointestinal helminth parasites

A nonredundant role for IL-21 receptor signaling in plasma cell differentiation and protective type 2 immunity against gastrointestinal helminth infection.

To B or not to B: B cells and the Th2-type immune response to helminths.

The T helper type 2 response to cysteine proteases requires dendritic cell-basophil cooperation via ROS-mediated signaling.

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E5169-77

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scholarly article

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10.1073/PNAS.1412663111

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48

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111

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Falk Nimmerjahn

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2014-11-17T00:00:00Z

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270653726

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25404305

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4260590

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