Making the case for skeletal myopathy as the major limitation of exercise capacity in heart failure
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Aerobic Exercise and Pharmacological Therapies for Skeletal Myopathy in Heart Failure: Similarities and DifferencesFrailty in Advanced Heart Failure: A Consequence of Aging or a Separate Entity?Exercise intolerance in heart failure with preserved ejection fraction: more than a heart problemReference values for vastus lateralis fiber size and type in healthy subjects over 40 years old: a systematic review and metaanalysisEffects of exercise interventions on peripheral vascular endothelial vasoreactivity in patients with heart failure with reduced ejection fractionThe clinical epidemiology of fatigue in newly diagnosed heart failure.Skeletal muscle abnormalities and exercise intolerance in older patients with heart failure and preserved ejection fraction.An official American Thoracic Society/European Respiratory Society statement: update on limb muscle dysfunction in chronic obstructive pulmonary disease.Preoperative muscle weakness as defined by handgrip strength and postoperative outcomes: a systematic review.Action potential-evoked calcium release is impaired in single skeletal muscle fibers from heart failure patients.Management of chronic heart failure in the older population.Effects of exercise training on neurovascular control and skeletal myopathy in systolic heart failure.Altered systemic ketone body metabolism in advanced heart failure.Exercise and the cardiovascular system: clinical science and cardiovascular outcomes.Lack of β2 -adrenoceptors aggravates heart failure-induced skeletal muscle myopathy in miceTestosterone deficiency increases hospital readmission and mortality rates in male patients with heart failure.Maximal oxygen uptake and exercise tolerance are improved in rats with heart failure subjected to low-level laser therapy associated with resistance training.Abnormalities of calcium handling proteins in skeletal muscle mirror those of the heart in humans with heart failure: a shared mechanism?Supervised exercise training versus usual care in ambulatory patients with left ventricular assist devices: A systematic reviewExercise training in chronic heart failure: improving skeletal muscle O2 transport and utilization.S-nitrosylation: integrator of cardiovascular performance and oxygen delivery.Iron deficiency and heart failure: diagnostic dilemmas and therapeutic perspectives.Sarcopenic obesity and the pathogenesis of exercise intolerance in heart failure with preserved ejection fraction.Nocturnal CPAP improves walking capacity in COPD patients with obstructive sleep apnoeaIntact skeletal muscle mitochondrial enzyme activity but diminished exercise capacity in advanced heart failure patients on optimal medical and device therapy.The therapeutic potential of IGF-I in skeletal muscle repair.Defining cardiac adaptations and safety of endurance training in patients with m.3243A>G-related mitochondrial disease.Comparative assessment of knee extensor and flexor muscle strength measured using a hand-held vs. isokinetic dynamometer.Exercise after heart transplantation: An overview.Heart failure with preserved ejection fraction in the elderly: scope of the problem.The cardiorenal syndrome in heart failure: cardiac? renal? syndrome?Unraveling new mechanisms of exercise intolerance in chronic heart failure: role of exercise training.Proteomic identification of biomarkers of skeletal muscle disorders.Skeletal myopathy in heart failure: effects of aerobic exercise training.Exercise intolerance in chronic heart failure: the role of cortisol and the catabolic state.Possible synergism of physical exercise and ghrelin-agonists in patients with cachexia associated with chronic heart failure.Heart Failure with Preserved Ejection Fraction in Older Adults.Heart failure with preserved ejection fraction induces molecular, mitochondrial, histological, and functional alterations in rat respiratory and limb skeletal muscle.Autophagy signaling in skeletal muscle of infarcted rats.Skeletal Muscle Mitochondrial Content, Oxidative Capacity, and Mfn2 Expression Are Reduced in Older Patients With Heart Failure and Preserved Ejection Fraction and Are Related to Exercise Intolerance.
P2860
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P2860
Making the case for skeletal myopathy as the major limitation of exercise capacity in heart failure
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Making the case for skeletal m ...... cise capacity in heart failure
@ast
Making the case for skeletal m ...... cise capacity in heart failure
@en
Making the case for skeletal m ...... cise capacity in heart failure
@nl
type
label
Making the case for skeletal m ...... cise capacity in heart failure
@ast
Making the case for skeletal m ...... cise capacity in heart failure
@en
Making the case for skeletal m ...... cise capacity in heart failure
@nl
prefLabel
Making the case for skeletal m ...... cise capacity in heart failure
@ast
Making the case for skeletal m ...... cise capacity in heart failure
@en
Making the case for skeletal m ...... cise capacity in heart failure
@nl
P2860
P1476
Making the case for skeletal m ...... cise capacity in heart failure
@en
P2093
Holly R Middlekauff
P2860
P304
P356
10.1161/CIRCHEARTFAILURE.109.903773
P577
2010-07-01T00:00:00Z