Proinflammatory and profibrotic mediators: principal effectors of leiomyoma development as a fibrotic disorder.
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Proceedings from the Third National Institutes of Health International Congress on Advances in Uterine Leiomyoma Research: comprehensive review, conference summary and future recommendationsRole of activin-A and myostatin and their signaling pathway in human myometrial and leiomyoma cell function.Fibroid-associated heavy menstrual bleeding: correlation between clinical features, Doppler ultrasound assessment of vasculature, and tissue gene expression profiles.Examining the relationship between uterine fibroids and dyspareunia among premenopausal women in the United States.Use of dietary phytochemicals to target inflammation, fibrosis, proliferation, and angiogenesis in uterine tissues: promising options for prevention and treatment of uterine fibroids?Liarozole inhibits transforming growth factor-β3--mediated extracellular matrix formation in human three-dimensional leiomyoma cultures.Racial and ethnic differences in the pathogenesis and clinical manifestations of uterine leiomyoma.Increased serum oxidative stress markers in women with uterine leiomyomamiR-200c regulates IL8 expression by targeting IKBKB: a potential mediator of inflammation in leiomyoma pathogenesis.Leiomyoma-derived transforming growth factor-β impairs bone morphogenetic protein-2-mediated endometrial receptivity.The role of progesterone signaling in the pathogenesis of uterine leiomyomaGrowth factors and myometrium: biological effects in uterine fibroid and possible clinical implicationsmiR-93/106b and their host gene, MCM7, are differentially expressed in leiomyomas and functionally target F3 and IL-8.miR-200c is aberrantly expressed in leiomyomas in an ethnic-dependent manner and targets ZEBs, VEGFA, TIMP2, and FBLN5.Leiomyoma: genetics, assisted reproduction, pregnancy and therapeutic advancesLipopolysaccharide activated TLR4/NF-κB signaling pathway of fibroblasts from uterine fibroids.Epidemiology of Uterine Fibroids: From Menarche to Menopause.In Thyroidectomized Thyroid Cancer Patients, False-Positive I-131 Whole Body Scans Are Often Caused by Inflammation Rather Than Thyroid Cancer.Expression profiling of nuclear receptors identifies key roles of NR4A subfamily in uterine fibroids.Abnormal uterine bleeding as a presenting symptom is related to multiple uterine leiomyoma: an ultrasound-based study.The endocannabinoid system and sex steroid hormone-dependent cancers.Uterine fibroids and current clinical challenges.Ulipristal acetate: in uterine fibroids.Uterine fibroids: clinical manifestations and contemporary management.Molecular targets of dietary phytochemicals for possible prevention and therapy of uterine fibroids: Focus on fibrosis.Profibrotic interleukin-33 is correlated with uterine leiomyoma tumour burden.Transforming growth factor-β signaling pathway cross-talking with ERα signaling pathway on regulating the growth of uterine leiomyoma activated by phenolic environmental estrogens in vitro.Overhydroxylation of Lysine of Collagen Increases Uterine Fibroids Proliferation: Roles of Lysyl Hydroxylases, Lysyl Oxidases, and Matrix Metalloproteinases.Role of Transforming Growth Factor β in Uterine Fibroid Biology.Endothelin-1-induced expression of α-smooth muscle actin in human myometrial fibroblasts.The Rising Phoenix-Progesterone as the Main Target of the Medical Therapy for Leiomyoma.Ulipristal Acetate and Extracellular Matrix Production in Human Leiomyomas In Vivo: A Laboratory Analysis of a Randomized Placebo Controlled Trial.Pathobiology of myomatosis uteri: the underlying knowledge to support our clinical practice.Effects of TGF-β on uterine fibroids of women of childbearing age and uterine artery embolization.Possible involvement of inflammatory/reparative processes in the development of uterine fibroids.Tranilast Inhibits Genes Functionally Involved in Cell Proliferation, Fibrosis, and Epigenetic Regulation and Epigenetically Induces miR-29c Expression in Leiomyoma Cells.Expression Profiling of lncRNAs, miRNAs, and mRNAs and Their Differential Expression in Leiomyoma Using Next-Generation RNA Sequencing.MicroRNA 21a-5p overexpression impacts mediators of extracellular matrix formation in uterine leiomyoma.Chronic Inflammation May Enhance Leiomyoma Development by the Involvement of Progenitor Cells.
P2860
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P2860
Proinflammatory and profibrotic mediators: principal effectors of leiomyoma development as a fibrotic disorder.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@ast
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@en
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@nl
type
label
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@ast
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@en
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@nl
prefLabel
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@ast
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@en
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@nl
P2860
P356
P1476
Proinflammatory and profibroti ...... opment as a fibrotic disorder.
@en
P2093
Nasser Chegini
P2860
P304
P356
10.1055/S-0030-1251476
P577
2010-04-22T00:00:00Z