Neurosteroids reduce inflammation after TBI through CD55 induction. - Wikidata - wikimedia - TriplyDB

Neurosteroids reduce inflammation after TBI through CD55 induction.

Neurosteroids reduce inflammation after TBI through CD55 induction.

http://www.wikidata.org/entity/Q34682946

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Combination treatment with progesterone and vitamin D hormone may be more effective than monotherapy for nervous system injury and disease Proof-of-concept trial with the neurosteroid pregnenolone targeting cognitive and negative symptoms in schizophrenia Decay accelerating factor (CD55) protects neuronal cells from chemical hypoxia-induced injury Brain and Serum Androsterone Is Elevated in Response to Stress in Rats with Mild Traumatic Brain Injury.Progesterone in experimental permanent stroke: a dose-response and therapeutic time-window study Allopregnanolone and neuroinflammation: a focus on multiple sclerosis.Frontiers in therapeutic development of allopregnanolone for Alzheimer's disease and other neurological disorders.Allopregnanolone levels are reduced in temporal cortex in patients with Alzheimer's disease compared to cognitively intact control subjects.Is progesterone a worthy candidate as a novel therapy for traumatic brain injury?Progesterone and allopregnanolone attenuate blood-brain barrier dysfunction following permanent focal ischemia by regulating the expression of matrix metalloproteinases.Secondary insults following traumatic brain injury enhance complement activation in the human brain and release of the tissue damage marker S100B Progesterone for neuroprotection in pediatric traumatic brain injury Neuroprotective actions of neurosteroids Menopause leads to elevated expression of macrophage-associated genes in the aging frontal cortex: rat and human studies identify strikingly similar changes.Vitamin D deficiency reduces the benefits of progesterone treatment after brain injury in aged rats.Estrous cycle, pregnancy, and parity enhance performance of rats in object recognition or object placement tasks.Progesterone and vitamin d hormone as a biologic treatment of traumatic brain injury in the aged.Effects of progesterone administration on infarct volume and functional deficits following permanent focal cerebral ischemia in rats.Experimental models of status epilepticus and neuronal injury for evaluation of therapeutic interventions The neuroprotective effects of progesterone on traumatic brain injury: current status and future prospects.Pharmacotherapy of traumatic brain injury: state of the science and the road forward: report of the Department of Defense Neurotrauma Pharmacology Workgroup.Adaptation to extreme stress: post-traumatic stress disorder, neuropeptide Y and metabolic syndrome.Analgesic effects and pharmacologic mechanisms of the Gelsemium alkaloid koumine on a rat model of postoperative pain.Does progesterone improve outcome in diffuse axonal injury?

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P2860

Neurosteroids reduce inflammation after TBI through CD55 induction.

http://www.wikidata.org/entity/Q34682946

description

2007 nî lūn-bûn

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2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի օգոստոսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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Neurosteroids reduce inflammation after TBI through CD55 induction.

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J.NEULET.2007.08.045

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Neuroscience Letters

P1476

Neurosteroids reduce inflammation after TBI through CD55 induction.

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P2093

Donald G Stein

http://www.w3.org/2001/XMLSchema#string

Jacob W VanLandingham

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Milos Cekic

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Sarah Cutler

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Stuart W Hoffman

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P2860

Pregnane X receptor (PXR) activation: a mechanism for neuroprotection in a mouse model of Niemann-Pick C disease

Complement C3 and C5 play critical roles in traumatic brain cryoinjury: blocking effects on neutrophil extravasation by C5a receptor antagonist.

Edema fluid accumulation within necrotic brain tissue as a cause of the mass effect of cerebral contusion in head trauma patients.

Allopregnanolone, a progesterone metabolite, is more effective than progesterone in reducing cortical infarct volume after transient middle cerebral artery occlusion.

The immunology of pregnancy.

The PPARs and PXRs: nuclear xenobiotic receptors that define novel hormone signaling pathways.

Progesterone treatment inhibits the inflammatory agents that accompany traumatic brain injury.

Progesterone protects against lipid peroxidation following traumatic brain injury in rats.

Progesterone administration modulates AQP4 expression and edema after traumatic brain injury in male rats.

ProTECT: a randomized clinical trial of progesterone for acute traumatic brain injury.

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