The Th17-defining transcription factor RORγt promotes glomerulonephritis - Wikidata - wikimedia - TriplyDB

The Th17-defining transcription factor RORγt promotes glomerulonephritis

The Th17-defining transcription factor RORγt promotes glomerulonephritis

http://www.wikidata.org/entity/Q34695374

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CD4+ T Cell Fate in Glomerulonephritis: A Tale of Th1, Th17, and Novel Treg Subtypes Stat3 programs Th17-specific regulatory T cells to control GN.Interleukin-17A promotes early but attenuates established disease in crescentic glomerulonephritis in mice.Inflammation-Induced IL-6 Functions as a Natural Brake on Macrophages and Limits GN.IL-17 Expression in the Time Course of Acute Anti-Thy1 Glomerulonephritis.Absence of MyD88 signaling induces donor-specific kidney allograft tolerance The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis.IL-6 Trans-Signaling Drives Murine Crescentic GN RORγt(+)Foxp3(+) Cells are an Independent Bifunctional Regulatory T Cell Lineage and Mediate Crescentic GN Endogenous Toll-Like Receptor 9 Regulates AKI by Promoting Regulatory T Cell Recruitment Complement receptor 3 mediates renal protection in experimental C3 glomerulopathy.Regulation of pathogenic Th17 cell differentiation by IL-10 in the development of glomerulonephritis.Update on crescentic glomerulonephritis.Cytokines: Names and Numbers You Should Care About.Regulatory T cells in immune-mediated renal disease.T-Bet Enhances Regulatory T Cell Fitness and Directs Control of Th1 Responses in Crescentic GN.Plasticity of Th17 Cells in Autoimmune Kidney Diseases.B-cell-derived IL-10 does not vitally contribute to the clinical course of glomerulonephritis.Renal IL-17 expression in human ANCA-associated glomerulonephritis.Decoy receptor 3 inhibits renal mononuclear leukocyte infiltration and apoptosis and prevents progression of IgA nephropathy in mice.T helper type 17 cells in immune-mediated glomerular disease.Abnormal CD161+ immune cells and retinoic acid receptor-related orphan receptor γt-mediate enhanced IL-17F expression in the setting of genetic hypertension.IL-17F Promotes Tissue Injury in Autoimmune Kidney Diseases.Autonomic regulation of the immune system in cardiovascular diseases.Signal transducer and activation of transcription 6 (STAT6) regulates T helper type 1 (Th1) and Th17 nephritogenic immunity in experimental crescentic glomerulonephritis.RORγt expression in Tregs promotes systemic lupus erythematosus via IL-17 secretion, alteration of Treg phenotype and suppression of Th2 responses.Induced regulatory T cells are phenotypically unstable and do not protect mice from rapidly progressive glomerulonephritis.Cln 3-requiring 9 is a negative regulator of Th17 pathway-driven inflammation in anti-glomerular basement membrane glomerulonephritis.Down-regulation of expression of retinoid acid-related orphan receptor C (RORC) in systemic lupus erythematosus.

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P2860

The Th17-defining transcription factor RORγt promotes glomerulonephritis

http://www.wikidata.org/entity/Q34695374

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年学术文章

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2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

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2010年學術文章

@yue

name

The Th17-defining transcription factor RORγt promotes glomerulonephritis

@ast

The Th17-defining transcription factor RORγt promotes glomerulonephritis

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The Th17-defining transcription factor RORγt promotes glomerulonephritis

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type

label

The Th17-defining transcription factor RORγt promotes glomerulonephritis

@ast

The Th17-defining transcription factor RORγt promotes glomerulonephritis

@en

The Th17-defining transcription factor RORγt promotes glomerulonephritis

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prefLabel

The Th17-defining transcription factor RORγt promotes glomerulonephritis

@ast

The Th17-defining transcription factor RORγt promotes glomerulonephritis

@en

The Th17-defining transcription factor RORγt promotes glomerulonephritis

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Journal of the American Society of Nephrology

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The Th17-defining transcription factor RORγt promotes glomerulonephritis

@en

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A Richard Kitching

http://www.w3.org/2001/XMLSchema#string

Oliver M Steinmetz

http://www.w3.org/2001/XMLSchema#string

Poh-Yi Gan

http://www.w3.org/2001/XMLSchema#string

Shaun A Summers

http://www.w3.org/2001/XMLSchema#string

Stephen R Holdsworth

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Timothy Semple

http://www.w3.org/2001/XMLSchema#string

P2860

A novel transcription factor, T-bet, directs Th1 lineage commitment

Retinoid-related orphan receptor gamma (RORgamma) is essential for lymphoid organogenesis and controls apoptosis during thymopoiesis

The differentiation of human T(H)-17 cells requires transforming growth factor-beta and induction of the nuclear receptor RORgammat

Interleukin-23 drives innate and T cell-mediated intestinal inflammation

Th17 and regulatory T cells in mediating and restraining inflammation.

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

IL-17 and Th17 Cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Requirement for RORgamma in thymocyte survival and lymphoid organ development

The role of the nuclear hormone receptor RORγt in the development of lymph nodes and Peyer's patches

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472-483

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scholarly article

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10.1681/ASN.2010040435

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3

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22

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2010-12-23T00:00:00Z

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21183590

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glomerulonephritis

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3060441

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