PDL1 is required for peripheral transplantation tolerance and protection from chronic allograft rejection. - Wikidata - wikimedia - TriplyDB

PDL1 is required for peripheral transplantation tolerance and protection from chronic allograft rejection.

PDL1 is required for peripheral transplantation tolerance and protection from chronic allograft rejection.

http://www.wikidata.org/entity/Q34697293

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Role of the PD-1 pathway in the immune response Transplantation tolerance and its outcome during infections and inflammation Tempering allorecognition to induce transplant tolerance with chemically modified apoptotic donor cells The CD8 T-cell response during tolerance induction in liver transplantation Infection with the intracellular bacterium, Listeria monocytogenes, overrides established tolerance in a mouse cardiac allograft model PD-1 regulates T cell proliferation in a tissue and subset-specific manner during normal mouse pregnancy Alloantigen expression on non-hematopoietic cells reduces graft-versus-leukemia effects in mice.Programmed death-1/B7-H1 negative costimulation protects mouse liver against ischemia and reperfusion injury.LAG-3, TGF-β, and cell-intrinsic PD-1 inhibitory pathways contribute to CD8 but not CD4 T-cell tolerance induced by allogeneic BMT with anti-CD40L.Program death-1 signaling and regulatory T cells collaborate to resist the function of adoptively transferred cytotoxic T lymphocytes in advanced acute myeloid leukemia.Peripheral deletional tolerance of alloreactive CD8 but not CD4 T cells is dependent on the PD-1/PD-L1 pathway PD-1-dependent mechanisms maintain peripheral tolerance of donor-reactive CD8+ T cells to transplanted tissue.The programmed death (PD)-1/PD-ligand 1 pathway regulates graft-versus-host-reactive CD8 T cells after liver transplantation.Soluble co-signaling molecules predict long-term graft outcome in kidney-transplanted patients The role of programmed cell death ligand-1 (PD-L1/CD274) in the development of graft versus host disease TIM4 Regulates the Anti-Islet Th2 Alloimmune Response The novel costimulatory programmed death ligand 1/B7.1 pathway is functional in inhibiting alloimmune responses in vivo.Cardiac allograft vasculopathy: a donor or recipient induced pathology?The link between the PDL1 costimulatory pathway and Th17 in fetomaternal tolerance.Influence of Interferon-γ Deficiency in Immune Tolerance Induced by Male Islet Transplantation Interactions between NKT cells and Tregs are required for tolerance to combined bone marrow and organ transplants.Increased circulating follicular helper T cells with decreased programmed death-1 in chronic renal allograft rejection.CD28 family and chronic rejection: "to belatacept...And beyond!".The emerging role of T cell Ig mucin 1 in alloimmune responses in an experimental mouse transplant model.Tolerance induction using nanoparticles bearing HY peptides in bone marrow transplantation.Variability in Immunohistochemical Detection of Programmed Death Ligand 1 (PD-L1) in Cancer Tissue Types.Programmed death ligand 1 regulates a critical checkpoint for autoimmune myocarditis and pneumonitis in MRL mice.The PD-1/PD-L1 inhibitory pathway is altered in pre-eclampsia and regulates T cell responses in pre-eclamptic rats.Harnessing negative T-cell costimulatory pathways to promote engraftment.Immune Tolerance for Autoimmune Disease and Cell Transplantation Divergent role of donor dendritic cells in rejection versus tolerance of allografts.PD-1/PD-L1 Interaction Maintains Allogeneic Immune Tolerance Induced by Administration of Ultraviolet B-Irradiated Immature Dendritic Cells.Expression and function of PDCD1 at the human maternal-fetal interface.PD-1/PD-L1, PD-1/PD-L2, and other co-inhibitory signaling pathways in transplantation.Prevention of allograft rejection in heart transplantation through concurrent gene silencing of TLR and Kinase signaling pathways.Maternal PD-1 regulates accumulation of fetal antigen-specific CD8+ T cells in pregnancy.Bacterial infections, alloimmunity, and transplantation tolerance.T-cell co-stimulatory blockade in transplantation: two steps forward one step back!Tolerogenic dendritic cells: role and therapeutic implications in systemic lupus erythematosus.T Cell Cosignaling Molecules in Transplantation.

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PDL1 is required for peripheral transplantation tolerance and protection from chronic allograft rejection.

http://www.wikidata.org/entity/Q34697293

description

2007 nî lūn-bûn

@nan

2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

@zh-hans

2007年学术文章

@zh-my

2007年学术文章

@zh-sg

2007年學術文章

@yue

name

PDL1 is required for periphera ...... m chronic allograft rejection.

@ast

PDL1 is required for periphera ...... m chronic allograft rejection.

@en

PDL1 is required for periphera ...... m chronic allograft rejection.

@nl

type

label

PDL1 is required for periphera ...... m chronic allograft rejection.

@ast

PDL1 is required for periphera ...... m chronic allograft rejection.

@en

PDL1 is required for periphera ...... m chronic allograft rejection.

@nl

prefLabel

PDL1 is required for periphera ...... m chronic allograft rejection.

@ast

PDL1 is required for periphera ...... m chronic allograft rejection.

@en

PDL1 is required for periphera ...... m chronic allograft rejection.

@nl

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JIMMUNOL.179.8.5204

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Journal of Immunology

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PDL1 is required for periphera ...... m chronic allograft rejection.

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Ana Maria Waaga

http://www.w3.org/2001/XMLSchema#string

Antje Habicht

http://www.w3.org/2001/XMLSchema#string

David M Rothstein

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Hideo Yagita

http://www.w3.org/2001/XMLSchema#string

Katsunori Tanaka

http://www.w3.org/2001/XMLSchema#string

Kazuhiro Yamaura

http://www.w3.org/2001/XMLSchema#string

Martin Grimm

http://www.w3.org/2001/XMLSchema#string

Miyuki Azuma

http://www.w3.org/2001/XMLSchema#string

Mohamed H Sayegh

http://www.w3.org/2001/XMLSchema#string

Monica J Albin

http://www.w3.org/2001/XMLSchema#string

P2860

Induced expression of PD-1, a novel member of the immunoglobulin gene superfamily, upon programmed cell death

The programmed death-1 (PD-1) pathway regulates autoimmune diabetes in nonobese diabetic (NOD) mice

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

Tumor-associated B7-H1 promotes T-cell apoptosis: a potential mechanism of immune evasion

PD-L2 is a second ligand for PD-1 and inhibits T cell activation

PD-1:PD-L inhibitory pathway affects both CD4(+) and CD8(+) T cells and is overcome by IL-2

The B7 family revisited

Central tolerance: learning self-control in the thymus

Expression of the PD-1 antigen on the surface of stimulated mouse T and B lymphocytes

Recruitment of Foxp3+ T regulatory cells mediating allograft tolerance depends on the CCR4 chemokine receptor

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5204-5210

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10.4049/JIMMUNOL.179.8.5204

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8

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