HilE interacts with HilD and negatively regulates hilA transcription and expression of the Salmonella enterica serovar Typhimurium invasive phenotype.
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sRNAs and the virulence of Salmonella enterica serovar TyphimuriumRegulatory principles governing Salmonella and Yersinia virulenceA Salmonella small non-coding RNA facilitates bacterial invasion and intracellular replication by modulating the expression of virulence factorsSystematic analysis of the regulation of type three secreted effectors in Salmonella enterica serovar Typhimurium.Comparative genomic analysis uncovers 3 novel loci encoding type six secretion systems differentially distributed in Salmonella serotypes.Gre factors-mediated control of hilD transcription is essential for the invasion of epithelial cells by Salmonella enterica serovar Typhimurium.The role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella.Signal transduction pathway mediated by the novel regulator LoiA for low oxygen tension induced Salmonella Typhimurium invasion.Microarray analysis and motif detection reveal new targets of the Salmonella enterica serovar Typhimurium HilA regulatory protein, including hilA itself.Fur activates the expression of Salmonella enterica pathogenicity island 1 by directly interacting with the hilD operator in vivo and in vitroA eukaryotic-like 3' untranslated region in Salmonella enterica hilD mRNA.Crosstalk between virulence loci: regulation of Salmonella enterica pathogenicity island 1 (SPI-1) by products of the std fimbrial operon.Negative regulation of Salmonella pathogenicity island 2 is required for contextual control of virulence during typhoid.The flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.Pathways leading from BarA/SirA to motility and virulence gene expression in Salmonella.Salmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Salmonella enterica highly expressed genes are disease specific.Mechanism of hilA repression by 1,2-propanediol consists of two distinct pathways, one dependent on and the other independent of catabolic production of propionate, in Salmonella enterica serovar Typhimurium.Patterns of expression and translocation of the ubiquitin ligase SlrP in Salmonella enterica serovar Typhimurium.Integration of a complex regulatory cascade involving the SirA/BarA and Csr global regulatory systems that controls expression of the Salmonella SPI-1 and SPI-2 virulence regulons through HilDRtsA and RtsB coordinately regulate expression of the invasion and flagellar genes in Salmonella enterica serovar Typhimurium.Mlc regulation of Salmonella pathogenicity island I gene expression via hilE repression.The two-component system CpxR/A represses the expression of Salmonella virulence genes by affecting the stability of the transcriptional regulator HilDThe Impact of 18 Ancestral and Horizontally-Acquired Regulatory Proteins upon the Transcriptome and sRNA Landscape of Salmonella enterica serovar Typhimurium.Coordinate regulation of Salmonella pathogenicity island 1 (SPI1) and SPI4 in Salmonella enterica serovar TyphimuriumThe intestinal fatty acid propionate inhibits Salmonella invasion through the post-translational control of HilDLeucine-responsive regulatory protein (Lrp) acts as a virulence repressor in Salmonella enterica serovar Typhimurium.Role of FimW, FimY, and FimZ in regulating the expression of type i fimbriae in Salmonella enterica serovar Typhimurium.Mapping the Regulatory Network for Salmonella enterica Serovar Typhimurium Invasion.Hyperinvasiveness of Salmonella enterica serovar Choleraesuis linked to hyperexpression of type III secretion systems in vitro.Interplay of regulatory RNAs and mobile genetic elements in enteric pathogens.Effect of bacteriophage on the susceptibility, motility, invasion, and survival of Salmonella Typhimurium exposed to the simulated intestinal conditions.Potassium transport of Salmonella is important for type III secretion and pathogenesis.Integrating global regulatory input into the Salmonella pathogenicity island 1 type III secretion system.Signal pathway in salt-activated expression of the Salmonella pathogenicity island 1 type III secretion system in Salmonella enterica serovar Typhimurium.Bile Acids Function Synergistically To Repress Invasion Gene Expression in Salmonella by Destabilizing the Invasion Regulator HilD.Regulation of Salmonella enterica pathogenicity island 1 by DNA adenine methylationThe TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarityThe small nucleoid-binding proteins H-NS, HU, and Fis affect hilA expression in Salmonella enterica serovar Typhimurium.Roles for DNA supercoiling and the Fis protein in modulating expression of virulence genes during intracellular growth of Salmonella enterica serovar Typhimurium.
P2860
Q24597091-FBB2F786-3444-4519-AF62-73098A7C487BQ26781384-426DC37C-BAC9-473C-90CF-F7C51CA5CFC8Q27349443-59057062-C788-4795-88F5-F680FC15F313Q33269622-A5DEF92C-8541-437B-97A1-9F3511E276B6Q33490263-5003B8D9-840A-458F-86A7-3F1FDB1732CCQ33585337-60313EFA-EDB7-4AC0-8C67-A0C8DB93F51AQ33649749-82D61DD0-A9B1-436E-B603-2E21AB08E16EQ33811877-44A3E8E3-E972-4A66-8DE0-B6B537555C78Q33855706-55A99F2E-7673-4F5C-9AEB-4A5C11DD1881Q33900474-21484E10-893C-456D-9C34-8E29FB16E89EQ34040392-7EEA90E1-44FD-497D-8DA9-2990E75CDB97Q34146148-D0398126-399F-4447-82E4-C814CFCC7559Q34161056-867EB54B-C982-439B-B21C-3EE0648450BCQ34222261-479E63E8-55D7-4EB5-ACC7-72BD50CFB399Q34232554-F147735E-9FA2-4F03-8637-3F5A82478421Q34336985-1F6FAAA3-B503-4523-A3A8-181004A14EC7Q34491811-7418A621-0F49-41B3-B3C4-CD16C1BF0941Q34563390-99CE205E-896E-473A-A4B2-771364CD6929Q34593329-CBC4886B-525E-4661-B271-01AEDBA228B8Q35049823-3342BF86-8BEC-416B-96F7-2CEFF254AA74Q35662912-C0E0C330-6A72-4B14-A772-A443084A56C9Q35804589-A9D48C33-A6F4-4679-BCC4-27394E564F7EQ35921961-62E72952-83EA-4428-A41B-D01F6BA8AFFAQ36114219-86B19B48-7AFF-4D12-BFBB-48AC822C12B0Q36483666-A6F4471F-23AA-4F77-922A-EF36510909A5Q36636799-5AE3B820-974C-4EDE-8C75-C64DFA2F89A1Q37075381-D33A8B11-F95A-43BF-962F-850BB8FE14F6Q37191525-42B97E9D-DB7E-4452-B02E-52E48F8E0995Q37236308-954F5663-B572-40DD-AE74-0B650CE56579Q37439093-78048DFA-6A5F-496F-BDF1-9C4FD8FCB42BQ38846031-9B13F1B0-74EB-4A19-A5BC-251431905EC1Q39026784-071257D5-906A-4785-8BD1-BA1137DD945BQ39144745-8EC0C690-77D2-46D9-AF2D-0FD66B3493C3Q39694438-12BD1C56-E495-4289-AB73-EAE7399C074BQ39759687-F58E6383-F5B5-4C9E-9134-ACEB630A8569Q39761480-5CD7DAA8-5BDD-46F0-9CD1-35944A365159Q39792027-75C8C70E-3EE8-4CA2-BE0F-D96D2FB47EF1Q39809027-E0B1FFF8-E7C0-4849-98C0-A0264C8101F2Q39863987-94E64741-7810-4F53-A436-68DFD58FA253Q40227942-6DE4E744-5BD8-417D-A9B0-703070ED65CA
P2860
HilE interacts with HilD and negatively regulates hilA transcription and expression of the Salmonella enterica serovar Typhimurium invasive phenotype.
description
2003 nî lūn-bûn
@nan
2003 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մարտին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@ast
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@en
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@nl
type
label
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@ast
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@en
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@nl
prefLabel
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@ast
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@en
HilE interacts with HilD and n ...... yphimurium invasive phenotype.
@nl
P2093
P2860
P1476
HilE interacts with HilD and n ...... Typhimurium invasive phenotype
@en
P2093
M Aaron Baxter
Rebecca L Wilson
Thomas F Fahlen
P2860
P304
P356
10.1128/IAI.71.3.1295-1305.2003
P407
P577
2003-03-01T00:00:00Z