FHIT gene therapy prevents tumor development in Fhit-deficient mice. - Wikidata - wikimedia - TriplyDB

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

http://www.wikidata.org/entity/Q34729169

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Fhit interaction with ferredoxin reductase triggers generation of reactive oxygen species and apoptosis of cancer cells Designed FHIT alleles establish that Fhit-induced apoptosis in cancer cells is limited by substrate binding The tumor spectrum in FHIT-deficient mice Adenosine monophosphoramidase activity of Hint and Hnt1 supports function of Kin28, Ccl1, and Tfb3 Hint, Fhit, and GalT: function, structure, evolution, and mechanism of three branches of the histidine triad superfamily of nucleotide hydrolases and transferases Di-, tri- and tetra-5'-O-phosphorothioadenosyl substituted polyols as inhibitors of Fhit: Importance of the alpha-beta bridging oxygen and beta phosphorus replacement Control of dinucleoside polyphosphates by the FHIT-homologous HNT2 gene, adenine biosynthesis and heat shock in Saccharomyces cerevisiae.A hybrid machine learning-based method for classifying the Cushing's Syndrome with comorbid adrenocortical lesions.Fhit, a tumor suppressor protein, induces autophagy via 14-3-3τ in non-small cell lung cancer cells.Replicative Stress and the FHIT Gene: Roles in Tumor Suppression, Genome Stability and Prevention of Carcinogenesis.How can we prevent cancer?Association of the promoter methylation and protein expression of Fragile Histidine Triad (FHIT) gene with the progression of differentiated thyroid carcinoma.Restoration of fragile histidine triad (FHIT) expression induces apoptosis and suppresses tumorigenicity in lung and cervical cancer cell lines.The tumour suppressor Fhit positively regulates MHC class I expression on cancer cells.Premature condensation induces breaks at the interface of early and late replicating chromosome bands bearing common fragile sites.Fragile site orthologs FHIT/FRA3B and Fhit/Fra14A2: evolutionarily conserved but highly recombinogenic FHIT suppresses epithelial-mesenchymal transition (EMT) and metastasis in lung cancer through modulation of microRNAs Influence of FHIT on benzo[a]pyrene-induced tumors and alopecia in mice: chemoprevention by budesonide and N-acetylcysteine.Fhit delocalizes annexin a4 from plasma membrane to cytosol and sensitizes lung cancer cells to paclitaxel.Deletion of histidine triad nucleotide-binding protein 1/PKC-interacting protein in mice enhances cell growth and carcinogenesis.Tumor suppressor genes FHIT and WWOX are deleted in primary effusion lymphoma (PEL) cell lines.FHIT-proteasome degradation caused by mitogenic stimulation of the EGF receptor family in cancer cells.A knockdown with smoke model reveals FHIT as a repressor of Heme oxygenase 1 Cancer prevention and therapy: strategies and problems.Alteration of the fragile histidine triad gene early in carcinogenesis: an update.Induction of apoptosis by tumor suppressor FHIT via death receptor signaling pathway in human lung cancer cells.The effect of adenovirus-mediated gene expression of FHIT in small cell lung cancer cells.Oral administration of recombinant adeno-associated virus-mediated bone morphogenetic protein-7 suppresses CCl(4)-induced hepatic fibrosis in mice.Characterization of the role of Fhit in suppression of DNA damage.Cancer and the FRA3B/FHIT fragile locus: it's a HIT Development of spontaneous tumours and intestinal lesions in Fhit gene knockout mice.Fhit-deficient normal and cancer cells are mitomycin C and UVC resistant.FOXP1: a potential therapeutic target in cancer.Gene therapy approaches for stem cell protection.Intramitochondrial calcium regulation by the FHIT gene product sensitizes to apoptosis.A Fhit-mimetic peptide suppresses annexin A4-mediated chemoresistance to paclitaxel in lung cancer cells.Epigenetics of renal cell carcinoma: the path towards new diagnostics and therapeutics Very large common fragile site genes and their potential role in cancer development.Increased sensitivity to cisplatin in non-small cell lung cancer cell lines after FHIT gene transfer.WWOX, large common fragile site genes, and cancer.

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P2860

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

http://www.wikidata.org/entity/Q34729169

description

2001 nî lūn-bûn

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2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2001 թվականի փետրվարին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

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FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@en

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

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type

label

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@ast

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@en

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@nl

prefLabel

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@ast

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@en

FHIT gene therapy prevents tumor development in Fhit-deficient mice.

@nl

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Proceedings of the National Academy of Sciences of the United States of America

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FHIT gene therapy prevents tumor development in Fhit-deficient mice.

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A Vecchione

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F Trapasso

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P2860

The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers

Muir-Torre-like syndrome in Fhit-deficient mice

The murine Fhit locus: isolation, characterization, and expression in normal and tumor cells

Replacement of Fhit in cancer cells suppresses tumorigenicity

a controlled trial of intratumoral ONYX-015, a selectively-replicating adenovirus, in combination with cisplatin and 5-fluorouracil in patients with recurrent head and neck cancer.

Research on esophageal cancer in China: a review.

Cellular immunity to viral antigens limits E1-deleted adenoviruses for gene therapy

The tumor-suppressor gene FHIT is involved in the regulation of apoptosis and in cell cycle control

Enhancement of in vivo adenovirus-mediated gene transfer and expression by prior depletion of tissue macrophages in the target organ.

Adeno-associated viruses.

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3346-3351

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6

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Carlo Maria Croce

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