Decreased microRNA is involved in the vascular remodeling abnormalities in chronic kidney disease (CKD).
about
Systems biology combining human- and animal-data miRNA and mRNA data identifies new targets in ureteropelvic junction obstructionDeletion of the miR-143/145 cluster leads to hydronephrosis in miceMedial vascular calcification revisited: review and perspectivesSerum microRNAs are altered in various stages of chronic kidney disease: a preliminary studyRelationship between microRNA-146a expression and plasma renalase levels in hemodialyzed patients.A current understanding of vascular calcification in CKD.Cardio-miRNAs and onco-miRNAs: circulating miRNA-based diagnostics for non-cancerous and cancerous diseasesMicroRNAs Regulate Vascular Medial Calcification.Genomic analysis of host - Peste des petits ruminants vaccine viral transcriptome uncovers transcription factors modulating immune regulatory pathways.Circulating and renal vein levels of microRNAs in patients with renal artery stenosisDifferential miRNA Expression in Cells and Matrix Vesicles in Vascular Smooth Muscle Cells from Rats with Kidney DiseaseInteraction between microRNA expression and classical risk factors in the risk of coronary heart disease.Plasma levels of microRNA in chronic kidney disease: patterns in acute and chronic exercise.Immunomediated Pan-cancer Regulation Networks are Dominant Fingerprints After Treatment of Cell Lines with Demethylation.Vascular mineralocorticoid receptor regulates microRNA-155 to promote vasoconstriction and rising blood pressure with aging.MicroRNA in diabetic nephropathy: renin angiotensin, aGE/RAGE, and oxidative stress pathway.Role of microRNAs in Vascular Remodeling.Pathophysiology of Vascular Calcification.Novel Biomarkers in the Diagnosis of Chronic Kidney Disease and the Prediction of Its Outcome.Extracellular vesicles in cardiovascular calcification: expanding current paradigms.Impaired vascular function contributes to exercise intolerance in chronic kidney disease.Calcium as a cardiovascular toxin in CKD-MBD.MicroRNA-155 is upregulated in ascites in patients with spontaneous bacterial peritonitis.MiR-145-5p inhibits proliferation and inflammatory responses of RMC through regulating AKT/GSK pathway by targeting CXCL16.Intracellular calcium increases in vascular smooth muscle cells with progression of chronic kidney disease in a rat model.Initiation and Propagation of Vascular Calcification Is Regulated by a Concert of Platelet- and Smooth Muscle Cell-Derived Extracellular Vesicles.Angiotensin II prevents calcification in vascular smooth muscle cells by enhancing magnesium influx.Genome-wide Profiling of Urinary Extracellular Vesicle microRNAs Associated With Diabetic Nephropathy in Type 1 Diabetes.Vascular calcification and renal bone disorders
P2860
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P2860
Decreased microRNA is involved in the vascular remodeling abnormalities in chronic kidney disease (CKD).
description
2013 nî lūn-bûn
@nan
2013 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@ast
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@en
Decreased microRNA is involved ...... ties in chronic kidney disease
@nl
type
label
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@ast
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@en
Decreased microRNA is involved ...... ties in chronic kidney disease
@nl
prefLabel
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@ast
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@en
Decreased microRNA is involved ...... ties in chronic kidney disease
@nl
P2093
P2860
P1433
P1476
Decreased microRNA is involved ...... chronic kidney disease (CKD).
@en
P2093
Kalisha D O'Neill
Kraiwiporn Kiattisunthorn
Matthew R Allen
Neal X Chen
Ranjani N Moorthi
Sharon M Moe
Vincent H Gattone
Xianming Chen
P2860
P304
P356
10.1371/JOURNAL.PONE.0064558
P407
P577
2013-05-22T00:00:00Z