Biofilm formation is not required for early-phase transmission of Yersinia pestis - Wikidata - wikimedia - TriplyDB

Biofilm formation is not required for early-phase transmission of Yersinia pestis

Biofilm formation is not required for early-phase transmission of Yersinia pestis

http://www.wikidata.org/entity/Q34747000

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Flea diversity as an element for persistence of plague bacteria in an East African plague focus The Role of Early-Phase Transmission in the Spread of Yersinia pestis Feeding Behavior Modulates Biofilm-Mediated Transmission of Yersinia pestis by the Cat Flea, Ctenocephalides felis Ecological Opportunity, Evolution, and the Emergence of Flea-Borne Plague The role of transition metal transporters for iron, zinc, manganese, and copper in the pathogenesis of Yersinia pestis Retracing the evolutionary path that led to flea-borne transmission of Yersinia pestis.Differential control of Yersinia pestis biofilm formation in vitro and in the flea vector by two c-di-GMP diguanylate cyclases.YfbA, a Yersinia pestis regulator required for colonization and biofilm formation in the gut of cat fleas Effects of temperature on the transmission of Yersinia Pestis by the flea, Xenopsylla Cheopis, in the late phase period.Resistance to Innate Immunity Contributes to Colonization of the Insect Gut by Yersinia pestis.Yersinia--flea interactions and the evolution of the arthropod-borne transmission route of plague Poly-N-acetylglucosamine expression by wild-type Yersinia pestis is maximal at mammalian, not flea, temperatures.Yersinia murine toxin is not required for early-phase transmission of Yersinia pestis by Oropsylla montana (Siphonaptera: Ceratophyllidae) or Xenopsylla cheopis (Siphonaptera: Pulicidae).Comparative Ability of Oropsylla montana and Xenopsylla cheopis Fleas to Transmit Yersinia pestis by Two Different Mechanisms.Transmission mechanisms of an emerging insect-borne rickettsial pathogen Formation and regulation of Yersinia biofilms.Plague: Infections of Companion Animals and Opportunities for Intervention.Comparative Proteomic Analysis Provides insight into the Key Proteins as Possible Targets Involved in Aspirin Inhibiting Biofilm Formation of Staphylococcus xylosus.Infection Prevalence, Bacterial Loads, and Transmission Efficiency in Oropsylla montana (Siphonaptera: Ceratophyllidae) One Day After Exposure to Varying Concentrations of Yersinia pestis in Blood.LPS modification promotes maintenance of Yersinia pestis in fleas.Infectious blood source alters early foregut infection and regurgitative transmission of Yersinia pestis by rodent fleas.

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P2860

Biofilm formation is not required for early-phase transmission of Yersinia pestis

http://www.wikidata.org/entity/Q34747000

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@ast

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@en

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@nl

type

label

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@ast

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@en

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@nl

prefLabel

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@ast

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@en

Biofilm formation is not required for early-phase transmission of Yersinia pestis

@nl

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Biofilm formation is not required for early-phase transmission of Yersinia pestis

@en

P2093

Alexander G Bobrov

http://www.w3.org/2001/XMLSchema#string

Anna M Schotthoefer

http://www.w3.org/2001/XMLSchema#string

Jennifer L Holmes

http://www.w3.org/2001/XMLSchema#string

Robert D Perry

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Sara M Vetter

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Scott W Bearden

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P2860

Depolymerization of beta-1,6-N-acetyl-D-glucosamine disrupts the integrity of diverse bacterial biofilms

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

Gene disruption in Escherichia coli: TcR and KmR cassettes with the option of Flp-catalyzed excision of the antibiotic-resistance determinant

Yersinia pestis--etiologic agent of plague

Analysis of HmsH and its role in plague biofilm formation.

Identification of critical amino acid residues in the plague biofilm Hms proteins.

Early-phase transmission of Yersinia pestis by unblocked fleas as a mechanism explaining rapidly spreading plague epizootics.

HmsP, a putative phosphodiesterase, and HmsT, a putative diguanylate cyclase, control Hms-dependent biofilm formation in Yersinia pestis.

Characterization of the Yersinia pestis Yfu ABC inorganic iron transport system.

Phenotypic convergence mediated by GGDEF-domain-containing proteins

P304

2216-2225

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scholarly article

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10.1099/MIC.0.037952-0

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P433

Pt 7

http://www.w3.org/2001/XMLSchema#string

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156

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P50

Rebecca J. Eisen

John A Montenieri

P577

2010-04-15T00:00:00Z

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P5875

43159804

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P698

20395271

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P921

Yersinia pestis

P932

3068684

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