Mice transgenically overexpressing sulfonylurea receptor 1 in forebrain resist seizure induction and excitotoxic neuron death. - Wikidata - wikimedia - TriplyDB

Mice transgenically overexpressing sulfonylurea receptor 1 in forebrain resist seizure induction and excitotoxic neuron death.

Mice transgenically overexpressing sulfonylurea receptor 1 in forebrain resist seizure induction and excitotoxic neuron death.

http://www.wikidata.org/entity/Q34749138

about

Noble gas xenon is a novel adenosine triphosphate-sensitive potassium channel opener How ATP inhibits the open K(ATP) channel Potassium Channels and Human Epileptic Phenotypes: An Updated Overview Neuroprotective role of ATP-sensitive potassium channels in cerebral ischemia ATP-sensitive potassium channelopathies: focus on insulin secretion Elevated expression of TDP-43 in the forebrain of mice is sufficient to cause neurological and pathological phenotypes mimicking FTLD-U.KATP channels modulate intrinsic firing activity of immature entorhinal cortex layer III neurons.Optical control of insulin release using a photoswitchable sulfonylurea Role of microglial IKKbeta in kainic acid-induced hippocampal neuronal cell death.Role of K(ATP) channels in protection against neuronal excitatory insults.Metabolic environment in substantia nigra reticulata is critical for the expression and control of hypoglycemia-induced seizures.Role of genetic influences in animal models of status.The opening effect of pregabalin on ATP-sensitive potassium channels in differentiated hippocampal neuron-derived H19-7 cells.Ankyrin regulates KATP channel membrane trafficking and gating in excitable cells.Single KATP channel opening in response to stimulation of AMPA/kainate receptors is mediated by Na+ accumulation and submembrane ATP and ADP changes.The pattern of ATP-sensitive K+ channel subunits, Kir6.2 and SUR1 mRNA expressions in DG region is different from those in CA1-3 regions of chronic epilepsy induced by picrotoxin in rats.The antiepileptic effect of the glycolytic inhibitor 2-deoxy-D-glucose is mediated by upregulation of K(ATP) channel subunits Kir6.1 and Kir6.2.Anticonvulsant effect of Diazoxide against Dichlorvos-induced seizures in mice.Metabolic etiologies in West syndrome.

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Mice transgenically overexpressing sulfonylurea receptor 1 in forebrain resist seizure induction and excitotoxic neuron death.

http://www.wikidata.org/entity/Q34749138

description

2001 nî lūn-bûn

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2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի փետրվարին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Mice transgenically overexpres ...... and excitotoxic neuron death.

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Proceedings of the National Academy of Sciences of the United States of America

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Mice transgenically overexpres ...... and excitotoxic neuron death.

@en

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A M Fernandez

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A S Basile

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B Stannard

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C Hernández-Sánchez

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D LeRoith

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H Arima

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I Fedorova

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Y Ito

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P2860

Reconstitution of IKATP: an inward rectifier subunit plus the sulfonylurea receptor

Cloning, tissue expression, and chromosomal localization of SUR2, the putative drug-binding subunit of cardiac, skeletal muscle, and vascular KATP channels

Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptor

ATP-regulated K+ channels in cardiac muscle

Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy

CaMKII regulates the frequency-response function of hippocampal synapses for the production of both LTD and LTP

Sulphonylurea receptor 2B and Kir6.1 form a sulphonylurea-sensitive but ATP-insensitive K+ channel

A family of sulfonylurea receptors determines the pharmacological properties of ATP-sensitive K+ channels

Genetic determinants of susceptibility to excitotoxic cell death: implications for gene targeting approaches.

Altered functional properties of KATP channel conferred by a novel splice variant of SUR1.

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3549-3554

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scholarly article

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10.1073/PNAS.051012898

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6

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98

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11248115

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