Molecular clustering identifies complement and endothelin induction as early events in a mouse model of glaucoma - Wikidata - wikimedia - TriplyDB

Molecular clustering identifies complement and endothelin induction as early events in a mouse model of glaucoma

Molecular clustering identifies complement and endothelin induction as early events in a mouse model of glaucoma

http://www.wikidata.org/entity/Q34755301

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Retinal Macroglial Responses in Health and Disease Oral microbiome link to neurodegeneration in glaucoma Retinal ganglion cell loss is accompanied by antibody depositions and increased levels of microglia after immunization with retinal antigens Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner Silencing of Fem1cR3 gene expression in the DBA/2J mouse precedes retinal ganglion cell death and is associated with histone deacetylase activity Endothelin-2 deficiency causes growth retardation, hypothermia, and emphysema in mice Endothelin-Mediated Changes in Gene Expression in Isolated Purified Rat Retinal Ganglion Cells Loss of function of endothelin-2 leads to reduced ovulation and CL formation.Generating embryonic stem cells from the inbred mouse strain DBA/2J, a model of glaucoma and other complex diseases Biomechanical aspects of axonal damage in glaucoma: A brief review.Involvement of EphB/Ephrin-B signaling in axonal survival in mouse experimental glaucoma.The Time Course of Gene Expression during Reactive Gliosis in the Optic Nerve.A lack of immune system genes causes loss in high frequency hearing but does not disrupt cochlear synapse maturation in mice.Sialic acid on the neuronal glycocalyx prevents complement C1 binding and complement receptor-3-mediated removal by microglia Early immune responses are independent of RGC dysfunction in glaucoma with complement component C3 being protective.Differential gene expression in glaucoma DLK-dependent signaling is important for somal but not axonal degeneration of retinal ganglion cells following axonal injury Datgan, a reusable software system for facile interrogation and visualization of complex transcription profiling data.Common variants at 9p21 and 8q22 are associated with increased susceptibility to optic nerve degeneration in glaucoma.Complement in animal development: unexpected roles of a highly conserved pathway.Endothelin B receptors contribute to retinal ganglion cell loss in a rat model of glaucoma.Early reduction of microglia activation by irradiation in a model of chronic glaucoma Rod photoreceptor ribbon synapses in DBA/2J mice show progressive age-related structural changes.Astrocyte processes label for filamentous actin and reorient early within the optic nerve head in a rat glaucoma model.Versatility of the complement system in neuroinflammation, neurodegeneration and brain homeostasis Vitamin B3 modulates mitochondrial vulnerability and prevents glaucoma in aged mice Disease progression in iridocorneal angle tissues of BMP2-induced ocular hypertensive mice with optical coherence tomography.Cardioembolic and small vessel disease stroke show differences in associations between systemic C3 levels and outcome Retinal ganglion cell dendritic atrophy in DBA/2J glaucoma Radiation pretreatment does not protect the rat optic nerve from elevated intraocular pressure-induced injury.Involvement of AP-1 and C/EBPβ in upregulation of endothelin B (ETB) receptor expression in a rodent model of glaucoma Clustering of transcriptional profiles identifies changes to insulin signaling as an early event in a mouse model of Alzheimer's disease.Combinatorial targeting of early pathways profoundly inhibits neurodegeneration in a mouse model of glaucoma.Effect of Lycium barbarum Polysaccharides on the expression of endothelin-1 and its receptors in an ocular hypertension model of rat glaucoma.Neurodegeneration severity can be predicted from early microglia alterations monitored in vivo in a mouse model of chronic glaucoma.Localization and phenotype-specific expression of ryanodine calcium release channels in C57BL6 and DBA/2J mouse strains Microglia: scapegoat, saboteur, or something else?APOE Stabilization by Exercise Prevents Aging Neurovascular Dysfunction and Complement Induction.Astrocytes in the optic nerve head express putative mechanosensitive channels.Radiation treatment inhibits monocyte entry into the optic nerve head and prevents neuronal damage in a mouse model of glaucoma

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Molecular clustering identifies complement and endothelin induction as early events in a mouse model of glaucoma

http://www.wikidata.org/entity/Q34755301

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2011 nî lūn-bûn

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Molecular clustering identifie ...... s in a mouse model of glaucoma

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Molecular clustering identifie ...... s in a mouse model of glaucoma

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Abbot F Clark

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Ben A Barres

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Beth Stevens

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Danilo G Macalinao

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Gareth R Howell

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Gregory L Sousa

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Jeffrey K Marchant

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Jessica M Barbay

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Matthew Hibbs

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Michael Walden

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P2860

DAVID Knowledgebase: a gene-centered database integrating heterogeneous gene annotation resources to facilitate high-throughput gene functional analysis

Laser trabeculoplasty for open angle glaucoma

KEGG for linking genomes to life and the environment

Statistical significance for genomewide studies

Intraocular pressure in genetically distinct mice: an update and strain survey

Susceptibility to neurodegeneration in a glaucoma is modified by Bax gene dosage

The Gene Ontology (GO) project in 2006

The classical complement cascade mediates CNS synapse elimination

Haploinsufficiency of the transcription factors FOXC1 and FOXC2 results in aberrant ocular development

A transcriptome database for astrocytes, neurons, and oligodendrocytes: a new resource for understanding brain development and function

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