Intradermal infections of mice by low numbers of african trypanosomes are controlled by innate resistance but enhance susceptibility to reinfection.

Intradermal infections of mice by low numbers of african trypanosomes are controlled by innate resistance but enhance susceptibility to reinfection.

Item
http://www.wikidata.org/entity/Q34763928
The Dermis as a Delivery Site of Trypanosoma brucei for Tsetse FliesNitric Oxide Protects against Infection-Induced Neuroinflammation by Preserving the Stability of the Blood-Brain BarrierMouse models for pathogenic African trypanosomes: unravelling the immunology of host-parasite-vector interactions.Non-invasive in vivo study of the Trypanosoma vivax infectious process consolidates the brain commitment in late infectionsTrypanosoma brucei Invasion and T-Cell Infiltration of the Brain Parenchyma in Experimental Sleeping Sickness: Timing and Correlation with Functional ChangesRegulatory T cells enhance susceptibility to experimental Trypanosoma congolense infection independent of mouse genetic backgroundImmunosuppression: cause for failures of vaccines against African TrypanosomiasesDistinct Contributions of CD4+ and CD8+ T Cells to Pathogenesis of Trypanosoma brucei Infection in the Context of Gamma Interferon and Interleukin-10Isolation, cloning, and pathologic analysis of Trypanosoma evansi field isolates.Low-dose intradermal infection with trypanosoma congolense leads to expansion of regulatory T cells and enhanced susceptibility to reinfectionModeling Rabbit Responses to Single and Multiple Aerosol Exposures of Bacillus anthracis Spores.Adenylate cyclases of Trypanosoma brucei inhibit the innate immune response of the host.Distinct Toll-like receptor signals regulate cerebral parasite load and interferon α/β and tumor necrosis factor α-dependent T-cell infiltration in the brains of Trypanosoma brucei-infected mice.Trypanosoma brucei growth control by TNF in mammalian host is independent of the soluble form of the cytokine.Chronic Trypanosoma congolense infections in mice cause a sustained disruption of the B-cell homeostasis in the bone marrow and spleen.Adenylate Cyclases of Trypanosoma brucei, Environmental Sensors and Controllers of Host Innate Immune Response.Neutrophils enhance early Trypanosoma brucei infection onset
P2860

Intradermal infections of mice by low numbers of african trypanosomes are controlled by innate resistance but enhance susceptibility to reinfection.

Item
http://www.wikidata.org/entity/Q34763928
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Intradermal infections of mice ...... susceptibility to reinfection.
@ast
Intradermal infections of mice ...... susceptibility to reinfection.
@en
Intradermal infections of mice ...... susceptibility to reinfection.
@nl
type
label
Intradermal infections of mice ...... susceptibility to reinfection.
@ast
Intradermal infections of mice ...... susceptibility to reinfection.
@en
Intradermal infections of mice ...... susceptibility to reinfection.
@nl
prefLabel
Intradermal infections of mice ...... susceptibility to reinfection.
@ast
Intradermal infections of mice ...... susceptibility to reinfection.
@en
Intradermal infections of mice ...... susceptibility to reinfection.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P932
P356
P1433
P1476
Intradermal infections of mice ...... susceptibility to reinfection.
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P932