Vitamin D induces interleukin-1β expression: paracrine macrophage epithelial signaling controls M. tuberculosis infection. - Wikidata - wikimedia - TriplyDB

Vitamin D induces interleukin-1β expression: paracrine macrophage epithelial signaling controls M. tuberculosis infection.

Vitamin D induces interleukin-1β expression: paracrine macrophage epithelial signaling controls M. tuberculosis infection.

http://www.wikidata.org/entity/Q34769278

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An update on the association of vitamin D deficiency with common infectious diseases 1 Innate Immunity Holding the Flanks until Reinforced by Adaptive Immunity against Mycobacterium tuberculosis Infection Versatile myeloid cell subsets contribute to tuberculosis-associated inflammation Vitamin D every day to keep the infection away?Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility Association of vitamin D deficiency, season of the year, and latent tuberculosis infection among household contacts Vitamin D in inflammatory diseases.Insights into battles between Mycobacterium tuberculosis and macrophages.Vitamin D in Autoimmunity: Molecular Mechanisms and Therapeutic Potential.A rho GDP dissociation inhibitor produced by apoptotic T-cells inhibits growth of Mycobacterium tuberculosis.A serum vitamin D level <25nmol/l pose high tuberculosis risk: a meta-analysis Vitamin D regulates cytokine patterns secreted by dendritic cells to promote differentiation of IL-22-producing T cells.The human cytomegalovirus lytic cycle is induced by 1,25-dihydroxyvitamin D3 in peripheral blood monocytes and in the THP-1 monocytic cell line The bacterial and host factors associated with extrapulmonary dissemination of Mycobacterium tuberculosis Toxoplasma gondii GRA7-Targeted ASC and PLD1 Promote Antibacterial Host Defense via PKCα Mycobacterial infection induces a specific human innate immune response.Patterns of Transcriptional Response to 1,25-Dihydroxyvitamin D3 and Bacterial Lipopolysaccharide in Primary Human Monocytes."Let there be light": the role of vitamin D in the immune response to vaccines.1,25 (OH)2D3 treatment alters the granulomatous response in M. tuberculosis infected mice Alu repeats as transcriptional regulatory platforms in macrophage responses to M. tuberculosis infection.Anti-Inflammatory Effects of Vitamin D on Human Immune Cells in the Context of Bacterial Infection.Vitamin D both facilitates and attenuates the cellular response to lipopolysaccharide.In search of a new paradigm for protective immunity to TB Genomic binding sites and biological effects of the vitamin D--VDR complex in multiple sclerosis [corrected].Inflammation and tuberculosis: host-directed therapies.Host-Directed Therapies for Tuberculosis.Vitamin D and tuberculosis: a review on a hot topic.Role of vitamin D in cytotoxic T lymphocyte immunity to pathogens and cancer.Effect of Severe Vitamin D Deficiency at Admission on Shock Reversal in Children With Septic Shock.Impact of Childhood Malnutrition on Host Defense and Infection.MIR144* inhibits antimicrobial responses against Mycobacterium tuberculosis in human monocytes and macrophages by targeting the autophagy protein DRAM2.Heightened circulating levels of antimicrobial peptides in tuberculosis-Diabetes co-morbidity and reversal upon treatment.Innate activation of human primary epithelial cells broadens the host response to Mycobacterium tuberculosis in the airways.The dynamic immunomodulatory effects of vitamin D3 during Mycobacterium infection.Vitamin D: a new anti-infective agent?Elevated serum 25-hydroxy (OH) vitamin D levels are associated with risk of TB progression in Gambian adults Vitamin D₃ metabolites enhance the NLRP3-dependent secretion of IL-1β from human THP-1 monocytic cells.Host-Directed Therapeutic Strategies for Tuberculosis.Hormonal vitamin D up-regulates tissue-specific PD-L1 and PD-L2 surface glycoprotein expression in humans but not mice.Pathway analysis of transcriptomic data shows immunometabolic effects of vitamin D.

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Vitamin D induces interleukin-1β expression: paracrine macrophage epithelial signaling controls M. tuberculosis infection.

http://www.wikidata.org/entity/Q34769278

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Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

The inflammasomes

NOD2 controls the nature of the inflammatory response and subsequent fate of Mycobacterium tuberculosis and M. bovis BCG in human macrophages

AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC

The human genome browser at UCSC

Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3

Toll-like receptor triggering of a vitamin D-mediated human antimicrobial response

Activation of inflammasomes requires intracellular redistribution of the apoptotic speck-like protein containing a caspase recruitment domain

Vitamin D inhibits human immunodeficiency virus type 1 and Mycobacterium tuberculosis infection in macrophages through the induction of autophagy

The inflammasome adaptor ASC regulates the function of adaptive immune cells by controlling Dock2-mediated Rac activation and actin polymerization

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Akafuba bulwadde

Mycobacterium tuberculosis

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