Organ-specific innate immune responses in a mouse model of invasive candidiasis. - Wikidata - wikimedia - TriplyDB

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

http://www.wikidata.org/entity/Q34769434

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Mechanistic Insights into the Role of C-Type Lectin Receptor/CARD9 Signaling in Human Antifungal Immunity New insights into innate immune control of systemic candidiasis Host response to Candida albicans bloodstream infection and sepsis Neutrophil Attack Triggers Extracellular Trap-Dependent Candida Cell Wall Remodeling and Altered Immune Recognition CARD9-Dependent Neutrophil Recruitment Protects against Fungal Invasion of the Central Nervous System NADPH oxidase-driven phagocyte recruitment controls Candida albicans filamentous growth and prevents mortality Redefined clinical features and diagnostic criteria in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy Type I interferons promote fatal immunopathology by regulating inflammatory monocytes and neutrophils during Candida infections Urea amidolyase (DUR1,2) contributes to virulence and kidney pathogenesis of Candida albicans The Candida albicans Histone Acetyltransferase Hat1 Regulates Stress Resistance and Virulence via Distinct Chromatin Assembly Pathways Jagunal homolog 1 is a critical regulator of neutrophil function in fungal host defense Live imaging of disseminated candidiasis in zebrafish reveals role of phagocyte oxidase in limiting filamentous growth.The loss of RGS protein-Gα(i2) interactions results in markedly impaired mouse neutrophil trafficking to inflammatory sites.MRI confirms loss of blood-brain barrier integrity in a mouse model of disseminated candidiasis Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of Candida by Modulating CR3 Downstream Syk Activation.Mechanisms of Candida albicans trafficking to the brain.Animal models for candidiasis Wild-type Drosophila melanogaster as a model host to analyze nitrogen source dependent virulence of Candida albicans.Drosophila and Galleria insect model hosts: new tools for the study of fungal virulence, pharmacology and immunology Overview of vertebrate animal models of fungal infection.Chemokine receptor Ccr1 drives neutrophil-mediated kidney immunopathology and mortality in invasive candidiasis Candida and host determinants of susceptibility to invasive candidiasis Cutting edge: Failure of antigen-specific CD4+ T cell recruitment to the kidney during systemic candidiasis.Trafficking of phagocytic peritoneal cells in hypoinsulinemic-hyperglycemic mice with systemic candidiasis Serial passaging of Candida albicans in systemic murine infection suggests that the wild type strain SC5314 is well adapted to the murine kidney.Protection from systemic Candida albicans infection by inactivation of the Sts phosphatases Catching fire: Candida albicans, macrophages, and pyroptosis.Syk signaling in dendritic cells orchestrates innate resistance to systemic fungal infection.Pathogenic fungi regulate immunity by inducing neutrophilic myeloid-derived suppressor cells.Experimental autoimmune encephalomyelitis development is aggravated by Candida albicans infection Mononuclear phagocyte-mediated antifungal immunity: the role of chemotactic receptors and ligands.CD47 Promotes Protective Innate and Adaptive Immunity in a Mouse Model of Disseminated Candidiasis.Oral infection with signature-tagged Listeria monocytogenes reveals organ-specific growth and dissemination routes in guinea pigs.Ibuprofen potentiates the in vivo antifungal activity of fluconazole against Candida albicans murine infection Flavodoxin-Like Proteins Protect Candida albicans from Oxidative Stress and Promote Virulence.Isolation of Mouse Neutrophils Immunosuppression, peripheral inflammation and invasive infection from endogenous gut microbiota activate retinal microglia in mouse models.The role of pattern recognition receptors in the innate recognition of Candida albicans.Candida albicans ISW2 Regulates Chlamydospore Suspensor Cell Formation and Virulence In Vivo in a Mouse Model of Disseminated Candidiasis.Differential Roles of Chemokines CCL2 and CCL7 in Monocytosis and Leukocyte Migration during West Nile Virus Infection.

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Organ-specific innate immune responses in a mouse model of invasive candidiasis.

http://www.wikidata.org/entity/Q34769434

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

@wuu

name

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@ast

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

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Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@nl

type

label

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

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Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@en

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@nl

prefLabel

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@ast

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@en

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@nl

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P1433

Journal of Innate Immunity

P1476

Organ-specific innate immune responses in a mouse model of invasive candidiasis.

@en

P2093

Chyi-Chia Richard Lee

http://www.w3.org/2001/XMLSchema#string

Jean K Lim

http://www.w3.org/2001/XMLSchema#string

Michail S Lionakis

http://www.w3.org/2001/XMLSchema#string

Philip M Murphy

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P2860

Chemokine receptor CCR5 promotes leukocyte trafficking to the brain and survival in West Nile virus infection

Ly6c+ "inflammatory monocytes" are microglial precursors recruited in a pathogenic manner in West Nile virus encephalitis

Nonfilamentous C. albicans mutants are avirulent

Natural killer cells do not play a dominant role in CD4+ subset differentiation in Candida albicans-infected mice.

Monocyte-mediated defense against microbial pathogens.

Phagocytosis and intracellular killing of pathogenic yeasts by human monocytes and neutrophils.

The distinct morphogenic states of Candida albicans.

Turnover of resident microglia in the normal adult mouse brain.

RORgammat and commensal microflora are required for the differentiation of mucosal interleukin 22-producing NKp46+ cells

Critical roles for CCR2 and MCP-3 in monocyte mobilization from bone marrow and recruitment to inflammatory sites

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180-199

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scholarly article

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10.1159/000321157

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2

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3

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2010-11-09T00:00:00Z

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47729492

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21063074

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3072204

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