Ventral tegmental afferents in stress-induced reinstatement: the role of cAMP response element-binding protein - Wikidata - wikimedia - TriplyDB

Ventral tegmental afferents in stress-induced reinstatement: the role of cAMP response element-binding protein

Ventral tegmental afferents in stress-induced reinstatement: the role of cAMP response element-binding protein

http://www.wikidata.org/entity/Q34786921

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MicroRNAs Modulate Interactions between Stress and Risk for Cocaine Addiction Ventral Tegmental Area Afferents and Drug-Dependent Behaviors Stress and VTA synapses: implications for addiction and depression Optogenetic strategies to investigate neural circuitry engaged by stress.Maternally responsive neurons in the bed nucleus of the stria terminalis and medial preoptic area: Putative circuits for regulating anxiety and reward Stress-Induced Reinstatement of Drug Seeking: 20 Years of Progress.Amygdala and bed nucleus of the stria terminalis circuitry: Implications for addiction-related behaviors.Loss of environmental enrichment increases vulnerability to cocaine addiction.Distinct extended amygdala circuits for divergent motivational states Constitutive activation of kappa opioid receptors at ventral tegmental area inhibitory synapses following acute stress.Stress-induced cocaine seeking requires a beta-2 adrenergic receptor-regulated pathway from the ventral bed nucleus of the stria terminalis that regulates CRF actions in the ventral tegmental area.Poststress block of kappa opioid receptors rescues long-term potentiation of inhibitory synapses and prevents reinstatement of cocaine seeking.Identifying candidate drivers of alcohol dependence-induced excessive drinking by assembly and interrogation of brain-specific regulatory networks GluN2B subunit deletion reveals key role in acute and chronic ethanol sensitivity of glutamate synapses in bed nucleus of the stria terminalis.Morphine history sensitizes postsynaptic GABA receptors on dorsal raphe serotonin neurons in a stress-induced relapse model in rats β-adrenergic receptor mediation of stress-induced reinstatement of extinguished cocaine-induced conditioned place preference in mice: roles for β1 and β2 adrenergic receptors.Regulation of the ventral tegmental area by the bed nucleus of the stria terminalis is required for expression of cocaine preference.New insights on neurobiological mechanisms underlying alcohol addiction.A corticotropin releasing factor pathway for ethanol regulation of the ventral tegmental area in the bed nucleus of the stria terminalis.Not all stress is equal: CREB is not necessary for restraint stress reinstatement of cocaine-conditioned reward.Emerging role for corticotropin releasing factor signaling in the bed nucleus of the stria terminalis at the intersection of stress and reward.Changes in c-Fos Expression in the Forced Swimming Test: Common and Distinct Modulation in Rat Brain by Desipramine and Citalopram.Developmental changes in the acute ethanol sensitivity of glutamatergic and GABAergic transmission in the BNST.Neurobiological mechanisms that contribute to stress-related cocaine use.Neurobiological mechanisms underlying relapse to cocaine use: contributions of CRF and noradrenergic systems and regulation by glucocorticoids.Role of κ-Opioid Receptors in the Bed Nucleus of Stria Terminalis in Reinstatement of Alcohol Seeking.Substance use modulates stress reactivity: Behavioral and physiological outcomes.The bed nucleus of the stria terminalis in drug-associated behavior and affect: A circuit-based perspective.Effect of baclofen on morphine-induced conditioned place preference, extinction, and stress-induced reinstatement in chronically stressed mice Fos activation of selective afferents to ventral tegmental area during cue-induced reinstatement of cocaine seeking in rats.Ethanol induced adaptations in 5-HT2c receptor signaling in the bed nucleus of the stria terminalis: implications for anxiety during ethanol withdrawal.Cue-induced reinstatement of alcohol-seeking behavior is associated with increased CaMKII T286 phosphorylation in the reward pathway of mice.Beta-2 adrenergic receptors mediate stress-evoked reinstatement of cocaine-induced conditioned place preference and increases in CRF mRNA in the bed nucleus of the stria terminalis in mice.

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Ventral tegmental afferents in stress-induced reinstatement: the role of cAMP response element-binding protein

http://www.wikidata.org/entity/Q34786921

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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JNEUROSCI.2827-10.2010

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Journal of Neuroscience

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Fair M Vassoler

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Julie A Blendy

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Lisa A Briand

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R Christopher Pierce

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Rita J Valentino

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P2860

Review. Transcriptional mechanisms of addiction: role of DeltaFosB

A role for the bed nucleus of the stria terminalis, but not the amygdala, in the effects of corticotropin-releasing factor on stress-induced reinstatement of cocaine seeking

Prior exposure to chronic stress and MDMA potentiates mesoaccumbens dopamine release mediated by the 5-HT(1B) receptor

The neural basis of addiction: a pathology of motivation and choice

The reinstatement model of drug relapse: history, methodology and major findings

Stress-induced potentiation of cocaine reward: a role for CRF R1 and CREB.

A role for the CRF-containing pathway from central nucleus of the amygdala to bed nucleus of the stria terminalis in the stress-induced reinstatement of cocaine seeking in rats.

Role for hypocretin in mediating stress-induced reinstatement of cocaine-seeking behavior.

Is there a common molecular pathway for addiction?

Curcumin reverses the effects of chronic stress on behavior, the HPA axis, BDNF expression and phosphorylation of CREB.

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10.1523/JNEUROSCI.2827-10.2010

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48

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21123561

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3075606

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