DNA hypomethylation causes bile duct defects in zebrafish and is a distinguishing feature of infantile biliary atresia.
about
Zebrafish: an important tool for liver disease researchBiliary atresia: cellular dynamics and immune dysregulationClinical implications of interferon-γ genetic and epigenetic variantsGlobal methylation, oxidative stress, and relative telomere length in biliary atresia patientsEvidence from human and zebrafish that GPC1 is a biliary atresia susceptibility gene.Identification of Annexin A4 as a hepatopancreas factor involved in liver cell survival.MicroRNA profiling identifies miR-29 as a regulator of disease-associated pathways in experimental biliary atresiaMethylation Microarray Studies Highlight PDGFA Expression as a Factor in Biliary AtresiaAssociation between Promoter Hypomethylation and Overexpression of Autotaxin with Outcome Parameters in Biliary AtresiaZebrafish models of human liver development and diseaseAetiology of biliary atresia: what is actually known?Imaging vertebrate digestive function and lipid metabolism in vivoClues to the etiology of bile duct injury in biliary atresiaExtrahepatic anomalies in infants with biliary atresia: results of a large prospective North American multicenter study.Etiology of biliary atresia as a developmental anomaly: recent advances.Recent Advances in Etiology of Biliary Atresia.Changes in epigenetic regulation of CD4+ T lymphocytesin biliary atresia.The lure of zebrafish in liver research: regulation of hepatic growth in development and regeneration.Loss of DNA methylation in zebrafish embryos activates retrotransposons to trigger antiviral signaling.Foxp3 promoter methylation impairs suppressive function of regulatory T cells in biliary atresia.Mutations in vacuolar H+ -ATPase subunits lead to biliary developmental defects in zebrafish.Interferon-gamma directly mediates developmental biliary defectsThree-dimensional structural analysis reveals a Cdk5-mediated kinase cascade regulating hepatic biliary network branching in zebrafish.Using zebrafish to model liver diseases-Where do we stand?MicroRNA-29b/142-5p contribute to the pathogenesis of biliary atresia by regulating the IFN-γ gene.
P2860
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P2860
DNA hypomethylation causes bile duct defects in zebrafish and is a distinguishing feature of infantile biliary atresia.
description
2011 nî lūn-bûn
@nan
2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@ast
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@en
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@nl
type
label
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@ast
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@en
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@nl
prefLabel
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@ast
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@en
DNA hypomethylation causes bil ...... of infantile biliary atresia.
@nl
P2093
P2860
P356
P1433
P1476
DNA hypomethylation causes bil ...... e of infantile biliary atresia
@en
P2093
Kristin Lorent
Megan M Ross
Michael Pack
Pierre Russo
Randolph P Matthews
Shuang Cui
P2860
P304
P356
10.1002/HEP.24106
P407
P577
2011-02-11T00:00:00Z