Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice. - Wikidata - wikimedia - TriplyDB

Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.

Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.

http://www.wikidata.org/entity/Q34794990

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Post-translational regulation of endothelial nitric oxide synthase in vascular endothelium Critical role for tetrahydrobiopterin recycling by dihydrofolate reductase in regulation of endothelial nitric-oxide synthase coupling: relative importance of the de novo biopterin synthesis versus salvage pathways The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans Role for Tetrahydrobiopterin in the Fetoplacental Endothelial Dysfunction in Maternal Supraphysiological Hypercholesterolemia Recoupling the cardiac nitric oxide synthases: tetrahydrobiopterin synthesis and recycling Free radical production by dysfunctional eNOS Reactive oxygen species in inflammation and tissue injury Bidirectional regulation of monocyte chemoattractant protein-1 gene at distinct sites of its promoter by nitric oxide in vascular smooth muscle cells Oxidative stress and compartment of gene expression determine proatherosclerotic effects of inducible nitric oxide synthase.Calcific aortic valve stenosis: methods, models, and mechanisms The Subcellular Localisation of Neuronal Nitric Oxide Synthase Determines the Downstream Effects of NO on Myocardial Function Oxidative risk for atherothrombotic cardiovascular disease.Advanced glycation end-products decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cells Tetrahydrobiopterin in cardiovascular health and disease.Chronic ingestion of H1-antihistamines increase progression of atherosclerosis in apolipoprotein E-/- mice.Caveolae structure and function Genetic deletion of apolipoprotein A-I increases airway hyperresponsiveness, inflammation, and collagen deposition in the lung.eNOS protects from atherosclerosis despite relevant superoxide production by the enzyme in apoE mice.Vasodilator-stimulated phosphoprotein protects against vascular inflammation and insulin resistance Insulin resistance reduces arterial prostacyclin synthase and eNOS activities by increasing endothelial fatty acid oxidation.Endothelial dysfunction abrogates the efficacy of normobaric hyperoxia in stroke.Mechanisms related to NO-induced motility in differentiated rat aortic smooth muscle cells.Endothelial dysfunction in the microcirculation of patients with obstructive sleep apnea.Circulating tetrahydrobiopterin as a novel biomarker for abdominal aortic aneurysm Arginase in retinopathy.Endothelial nitric oxide synthase uncoupling: a novel pathway in OSA induced vascular endothelial dysfunction.Reactive oxygen species in cardiovascular disease.Sepiapterin improves angiogenesis of pulmonary artery endothelial cells with in utero pulmonary hypertension by recoupling endothelial nitric oxide synthase.PPARγ ligands regulate NADPH oxidase, eNOS, and barrier function in the lung following chronic alcohol ingestion Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse Fetoplacental vascular endothelial dysfunction as an early phenomenon in the programming of human adult diseases in subjects born from gestational diabetes mellitus or obesity in pregnancy.PPAR-α Agonist Fenofibrate Upregulates Tetrahydrobiopterin Level through Increasing the Expression of Guanosine 5'-Triphosphate Cyclohydrolase-I in Human Umbilical Vein Endothelial Cells.Perspectives of Targeting mTORC1-S6K1 in Cardiovascular Aging Phosphodiesterases Regulate BAY 41-2272-Induced VASP Phosphorylation in Vascular Smooth Muscle Cells.Hyperglycemia and endothelial dysfunction in atherosclerosis: lessons from type 1 diabetes.FoxOs integrate pleiotropic actions of insulin in vascular endothelium to protect mice from atherosclerosis.The two faces of endothelial nitric oxide synthase in the pathophysiology of atherosclerosis.Reduced vascular nitric oxide-cGMP signaling contributes to adipose tissue inflammation during high-fat feeding.Pleiotropic effects of calcium channel blockers.Nitric oxide and geriatrics: Implications in diagnostics and treatment of the elderly.

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P2860

Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice.

http://www.wikidata.org/entity/Q34794990

description

2002 nî lūn-bûn

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2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2002 թվականի օգոստոսին հրատարակված գիտական հոդված

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Overexpression of endothelial ...... mation in apoE-deficient mice.

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Journal of Clinical Investigation

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Overexpression of endothelial ...... mation in apoE-deficient mice.

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Hiromu Sakurai

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Hiroyuki Yasui

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Ken-ichi Hirata

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Masahiro Masada

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Masanori Ozaki

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Masayuki Namiki

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Mitsuhiro Yokoyama

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Nobutaka Inoue

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Seinosuke Kawashima

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Tetsuaki Hirase

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P2860

Vascular endothelial dysfunction resulting from L-arginine deficiency in a patient with lysinuric protein intolerance

L-ascorbic acid potentiates endothelial nitric oxide synthesis via a chemical stabilization of tetrahydrobiopterin.

Endothelial NO synthase overexpression inhibits lesion formation in mouse model of vascular remodeling.

Mechanisms underlying endothelial dysfunction in diabetes mellitus.

Inducible nitric oxide synthase deficiency does not affect the susceptibility of mice to atherosclerosis but increases collagen content in lesions.

Endothelial regulation of vasomotion in apoE-deficient mice: implications for interactions between peroxynitrite and tetrahydrobiopterin.

Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure.

Enhanced atherosclerosis and kidney dysfunction in eNOS(-/-)Apoe(-/-) mice are ameliorated by enalapril treatment.

Role of oxidative stress in cardiovascular diseases.

Diet-induced atherosclerosis in mice heterozygous and homozygous for apolipoprotein E gene disruption.

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331-340

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10.1172/JCI0215215

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3

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110

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2002-08-01T00:00:00Z

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12163452

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atherosclerosis

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151086

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