Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation - Wikidata - wikimedia - TriplyDB

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

http://www.wikidata.org/entity/Q34796730

about

Recent advances in understanding the molecular mechanisms of the development and function of Th17 cells Ubiquitin signaling in immune responses NF-κB in inflammation and renal diseases A key role for NF-κB transcription factor c-Rel in T-lymphocyte-differentiation and effector functions TCR signaling to NF-κB and mTORC1: Expanding roles of the CARMA1 complex Apilimod inhibits the production of IL-12 and IL-23 and reduces dendritic cell infiltration in psoriasis Functional imaging of Rel expression in inflammatory processes using bioluminescence imaging system in transgenic mice The ubiquitin ligase Peli1 negatively regulates T cell activation and prevents autoimmunity NOD2 is a negative regulator of Toll-like receptor 2-mediated T helper type 1 responses LRP1 expression in microglia is protective during CNS autoimmunity.Heterogeneous Network Edge Prediction: A Data Integration Approach to Prioritize Disease-Associated Genes.RNAi-mediated c-Rel silencing leads to apoptosis of B cell tumor cells and suppresses antigenic immune response in vivo.A small-molecule c-Rel inhibitor reduces alloactivation of T cells without compromising antitumor activity Differential role for c-Rel and C/EBPbeta/delta in TLR-mediated induction of proinflammatory cytokines.Development of Foxp3(+) regulatory t cells is driven by the c-Rel enhanceosome.Activation of the transcriptional function of the NF-κB protein c-Rel by O-GlcNAc glycosylation NF-kappaB1 contributes to T cell-mediated control of Toxoplasma gondii in the CNS.Critical roles of TIPE2 protein in murine experimental colitis T-cell-receptor-dependent signal intensity dominantly controls CD4(+) T cell polarization In Vivo.Treatment of murine Th1- and Th2-mediated inflammatory bowel disease with NF-kappa B decoy oligonucleotides.The clinical implication and molecular mechanism of preferential IL-4 production by modified glycolipid-stimulated NKT cells.Role of the NF-κB transcription factor c-Rel in the generation of CD8+ T-cell responses to Toxoplasma gondii.Transcriptional regulation of the Th17 immune response by IKK(alpha)The microRNA-21-PDCD4 axis prevents type 1 diabetes by blocking pancreatic beta cell death.Loss of c-REL but not NF-κB2 prevents autoimmune disease driven by FasL mutation.The Th17 immune response is controlled by the Rel-RORγ-RORγ T transcriptional axis.Protective effects of dietary curcumin in mouse model of chemically induced colitis are strain dependent.NOD2 regulation of Toll-like receptor responses and the pathogenesis of Crohn's disease Opposing roles of NF-kappaB family members in the regulation of NK cell proliferation and production of IFN-gamma.Exacerbated experimental colitis in TNFAIP8-deficient mice.Constitutive activity of NF-kappa B in myeloid cells drives pathogenicity of monocytes and macrophages during autoimmune neuroinflammation.Non-viral delivery of nuclear factor-kappaB decoy ameliorates murine inflammatory bowel disease and restores tissue homeostasis.Genetic Variation in the REL Gene Increases Risk of Behcet's Disease in a Chinese Han Population but That of PRKCQ Does Not.Exogenous farnesol interferes with the normal progression of cytokine expression during candidiasis in a mouse model.[Nle4, D-Phe7]-α-MSH Inhibits Toll-Like Receptor (TLR)2- and TLR4-Induced Microglial Activation and Promotes a M2-Like Phenotype.NOD2 transgenic mice exhibit enhanced MDP-mediated down-regulation of TLR2 responses and resistance to colitis induction.Single-Cell Genomics Unveils Critical Regulators of Th17 Cell Pathogenicity.Ets-1, a functional cofactor of T-bet, is essential for Th1 inflammatory responses.Epigenetic regulation of the expression of Il12 and Il23 and autoimmune inflammation by the deubiquitinase Trabid.Systems-based medicine approaches to understand and treat complex diseases. The example of multiple sclerosis.

P2860

Q24626339-7A59CF61-1D57-47B3-AEB5-97AA60C1DCBB Q26751376-19C88F9D-5A5F-43F8-A847-3638E5F62C61 Q27021276-AF3A4112-1114-4E94-B544-5A9449FB9BDA Q27026685-D3C37992-E097-4D45-97D3-C62B536EAF62 Q28085394-081A6D2A-91E9-4D37-923F-B4ED3D74D58F Q28482161-C1C5D647-7F0C-4170-8520-9DFA44479196 Q28487261-1CAC986D-D708-4D40-B61F-89753074FBCE Q28505401-D7D5C081-FE47-4043-A0EC-74A376BB8A18 Q28588300-087D10D4-316D-4753-BBA1-65D4AC9D5558 Q30276379-E57165E3-62F7-402F-A34F-38A4F0089AD3 Q30979471-F68C4D35-65A5-47D9-9C9F-70400939263E Q33426864-F0151798-F216-4DD6-BB2B-D5762FA0AC0C Q33573863-10316E6E-F4E4-4D4F-B769-35DB23BF691E Q33588112-6BDD57BC-9454-4632-9849-F6D937B476C2 Q33596681-D910A2A7-4CCB-4794-A34F-FDD781EE0D75 Q33791683-25690D30-1F63-4CBE-95EA-126FDBB57A02 Q33814730-E337A42B-E6BE-40FA-9AF1-7A9095CC5360 Q33940549-EFEA711C-D2BB-43BE-8B5B-1C7910FB3CA4 Q33964858-B29439AC-9526-404D-9C6E-C6A2A98E378E Q34084499-696B2E90-0EB3-46F7-B6C1-E77F8A7982F4 Q34328138-2400B9DE-AEE2-4B18-BE37-3C66C3A491D1 Q34365047-684EF6F1-B273-4882-A068-52E10FED1DF7 Q35102214-85C7F8D3-DFCD-445A-9830-71BDA7E76BBC Q35123062-13162851-20B1-44A6-9812-3A3489751025 Q35349474-702B3EA5-7846-4254-AE40-5014B10F4394 Q35441559-3C7A1F1F-C414-4226-AFBB-AF9C783DC0F8 Q35593759-20ABDAC2-D768-4FCD-BABE-56463BE42DC3 Q35597934-28F62644-FDD2-4EBD-86FD-AAB991D97C57 Q35641142-B1D2F456-A59F-4B8E-9F84-DE6774311435 Q35691053-8B458ACD-37E2-4BCF-9729-CD294790221E Q35742153-E27CC425-819C-4922-9274-D3419CC64623 Q35761406-EC1360DE-91E0-4344-991B-83E87D0BDB84 Q35897813-90D1595B-6E33-4890-B81D-8CDFDE3A7B4A Q35949651-023BC677-F7E9-4DAC-B781-2AB637B004B1 Q36064675-8B651308-2149-48BB-9DD2-583D9CEAC31B Q36259779-7874BF41-0D63-4988-B7A4-BEF1E0AC8641 Q36352374-7354D9BD-1D59-4052-83E0-D5FF19226DE9 Q36403225-FB80F94A-9F82-48E2-BDCA-BB864DA4633D Q36586330-329DAE13-273C-4E03-838C-9FA0F29F6184 Q36687908-02002569-623B-46EB-B8CC-E9C7513FEAEC

P2860

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

http://www.wikidata.org/entity/Q34796730

description

2002 nî lūn-bûn

@nan

2002 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2002年の論文

@ja

2002年論文

@yue

2002年論文

@zh-hant

2002年論文

@zh-hk

2002年論文

@zh-mo

2002年論文

@zh-tw

2002年论文

@wuu

name

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@ast

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@en

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@nl

type

label

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@ast

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@en

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@nl

prefLabel

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@ast

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@en

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@nl

P1433

Q34796730-22F6A025-43C9-468C-9DDA-28EC4F265FA5

P1476

Q34796730-6323CE62-9F0C-458D-864C-3245E89F781F

P2093

Q34796730-01C12B68-BC72-4559-919E-09D133CE4922

Q34796730-629B6E3B-B19F-44F0-82F7-87B7B6E2ACBA

Q34796730-6F7E1284-9EC8-47E0-8442-43ECF5FC5742

Q34796730-B635871C-B6D1-45CE-85FC-DF85821066D0

Q34796730-B70FD497-615F-46B7-B21C-475D5EDE5F33

Q34796730-E020350A-F2AC-4052-9D7D-4B06AA3862C5

Q34796730-F637E6E4-2815-4F8C-B4DE-88A7E273CD86

Q34796730-F988615C-A0D4-460D-A334-45D090CC83F0

P2860

Q34796730-003EFC6E-AACF-41E2-8EF0-8966A31D03AC

Q34796730-073F31BA-5C5B-4817-BA55-9B93CEEA8976

Q34796730-0930BEF7-F0C1-4576-9404-AABDD50E74A7

Q34796730-0C18F6FE-2B6E-4243-8D60-B47378024984

Q34796730-1003BB91-6180-41D5-8446-709F14EB7741

Q34796730-1EBA9BA9-18F7-4D73-9537-230B8D640883

Q34796730-33793EAE-9CED-4832-836D-088CF143DC02

Q34796730-3DB7D389-FD52-4BE7-84D1-64431FAC44EA

Q34796730-3F768CA5-7A73-4C93-B420-D5D693227B0D

Q34796730-44EFC696-F20A-4F2D-A4EE-020D1B82DB99

P304

Q34796730-781A811D-5C73-449E-9CCC-713F289D685B

P31

Q34796730-67BC7783-6794-4671-8F35-B4987784E45C

P356

Q34796730-74FA9205-A0E7-4C2A-92CE-12052155B0A6

P407

Q34796730-3E0C69A7-F72D-4F94-9861-632DE8412AB6

P433

Q34796730-BBA335D8-723C-47FA-A35B-704663F2BDA4

P478

Q34796730-7ECE1AD6-1DEA-4522-9315-BA9F519BFB18

P577

Q34796730-D1D13A48-D3FE-4100-BA8C-1EC6E98F5F00

P698

Q34796730-B3CDAA9E-793B-479E-9089-4BE3A8B2DE2A

P932

Q34796730-D08DADE7-3433-467B-8F6E-9CE09AEEF1EC

P356

P1433

Journal of Clinical Investigation

P1476

Critical roles of c-Rel in autoimmune inflammation and helper T cell differentiation

@en

P2093

Brendan A Hilliard

http://www.w3.org/2001/XMLSchema#string

Christopher Hunter

http://www.w3.org/2001/XMLSchema#string

Hsiou-Chi Liou

http://www.w3.org/2001/XMLSchema#string

Jing Sun

http://www.w3.org/2001/XMLSchema#string

Lingyun Xu

http://www.w3.org/2001/XMLSchema#string

Nicola Mason

http://www.w3.org/2001/XMLSchema#string

Salah-Eddine Lamhamedi-Cherradi

http://www.w3.org/2001/XMLSchema#string

Youhai H Chen

http://www.w3.org/2001/XMLSchema#string

P2860

A novel transcription factor, T-bet, directs Th1 lineage commitment

Respiratory syncytial virus-induced RANTES production from human bronchial epithelial cells is dependent on nuclear factor-kappa B nuclear binding and is inhibited by adenovirus-mediated expression of inhibitor of kappa B alpha

NF-kappa B RelA-deficient lymphocytes: normal development of T cells and B cells, impaired production of IgA and IgG1 and reduced proliferative responses

Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

Constitutive NF-kappa B activity in neurons

Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases

An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

Tissue-specific regulation of cytokine gene expression.

Multiorgan inflammation and hematopoietic abnormalities in mice with a targeted disruption of RelB, a member of the NF-kappa B/Rel family.

Rel-dependent induction of A1 transcription is required to protect B cells from antigen receptor ligation-induced apoptosis

P304

843-850

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1172/JCI0215254

http://www.w3.org/2001/XMLSchema#string

P407

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

110

http://www.w3.org/2001/XMLSchema#string

P577

2002-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

12235116

http://www.w3.org/2001/XMLSchema#string

P932

151124

http://www.w3.org/2001/XMLSchema#string