Soluble TWEAK independently predicts atherosclerosis in renal transplant patients. - Wikidata - wikimedia - TriplyDB

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

http://www.wikidata.org/entity/Q34815993

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The relationship between asymptomatic organ damage, and serum soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) and Interleukin-17A (IL-17A) levels in non-diabetic hypertensive patients A longitudinal study of inflammation, CKD-mineral bone disorder, and carotid atherosclerosis after renal transplantation.Soluble TWEAK and Major Adverse Cardiovascular Events in Patients with CKD.Is TWEAK a Biomarker for Autoimmune/Chronic Inflammatory Diseases?TWEAK/Fn14 Axis: A Promising Target for the Treatment of Cardiovascular Diseases.Neutrophil-lymphocyte ratio as predictor of mortality and morbidity in cardiovascular surgery: a systematic review.Transition from identity to bioactivity-guided proteomics for biomarker discovery with focus on the PF2D platform.Soluble TWEAK may predict carotid atherosclerosis in treated HIV infection.TWEAK protects cardiomyocyte against apoptosis in a PI3K/AKT pathway dependent manner Identifying new Risk Markers and Potential Targets: The Value of the Proteome.Serum sTWEAK and FGF-23 Levels in Hemodialysis and Renal Transplant Patients.Tweak up-regulates endothelin-1 system in mouse and human endothelial cells.

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P2860

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

http://www.wikidata.org/entity/Q34815993

description

2013 nî lūn-bûn

@nan

2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@ast

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@en

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@nl

type

label

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@ast

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@en

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@nl

prefLabel

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@ast

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@en

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients.

@nl

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P1476

Soluble TWEAK independently predicts atherosclerosis in renal transplant patients

@en

P2093

Adrian Covic

http://www.w3.org/2001/XMLSchema#string

Aysun Toker

http://www.w3.org/2001/XMLSchema#string

Elvin Enes Gul

http://www.w3.org/2001/XMLSchema#string

Fatih Mehmet Erdur

http://www.w3.org/2001/XMLSchema#string

Halil Zeki Tonbul

http://www.w3.org/2001/XMLSchema#string

Huseyin Ozbiner

http://www.w3.org/2001/XMLSchema#string

Kultigin Turkmen

http://www.w3.org/2001/XMLSchema#string

Mehmet Kayrak

http://www.w3.org/2001/XMLSchema#string

Suleyman Turk

http://www.w3.org/2001/XMLSchema#string

Yalcin Solak

http://www.w3.org/2001/XMLSchema#string

P2860

TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor

Endothelial dysfunction in renal transplant recipients maintained on cyclosporine

Identification of soluble tumor necrosis factor-like weak inducer of apoptosis (sTWEAK) as a possible biomarker of subclinical atherosclerosis.

Emerging biomarkers for evaluating cardiovascular risk in the chronic kidney disease patient: how do new pieces fit into the uremic puzzle?

TWEAK/Fn14 pathway: an immunological switch for shaping tissue responses.

Soluble TWEAK and PTX3 in nondialysis CKD patients: impact on endothelial dysfunction and cardiovascular outcomes.

The elephant in uremia: oxidant stress as a unifying concept of cardiovascular disease in uremia.

Fetuin-A, inflammation, and coronary artery calcification in hemodialysis patients

Cardiovascular mortality in end-stage renal disease.

Chronic kidney disease as cause of cardiovascular morbidity and mortality.

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144

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scholarly article

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10.1186/1471-2369-14-144

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14

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Abduzhappar Gaipov

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2013-07-12T00:00:00Z

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249318319

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23849432

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atherosclerosis

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3711729

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