Effects of traditional Chinese medicine Xin-Ji-Er-Kang formula on 2K1C hypertensive rats: role of oxidative stress and endothelial dysfunction - Wikidata - wikimedia - TriplyDB

Effects of traditional Chinese medicine Xin-Ji-Er-Kang formula on 2K1C hypertensive rats: role of oxidative stress and endothelial dysfunction

Effects of traditional Chinese medicine Xin-Ji-Er-Kang formula on 2K1C hypertensive rats: role of oxidative stress and endothelial dysfunction

http://www.wikidata.org/entity/Q34816253

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Protective effects of Xinji'erkang on myocardial infarction induced cardiac injury in mice Benefical therapeutic effect of Chinese Herbal Xinji'erkang formula on hypertension-induced renal injury in the 2-kidney-1-clip hypertensive rats.Combined aliskiren and L-arginine treatment has antihypertensive effects and prevents vascular endothelial dysfunction in a model of renovascular hypertension.β-Catenin-Dependent Signaling Pathway Contributes to Renal Fibrosis in Hypertensive Rats.Radix Cyathula officinalis Kuan inhibits arterial remodeling in spontaneously hypertensive rats.

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Effects of traditional Chinese medicine Xin-Ji-Er-Kang formula on 2K1C hypertensive rats: role of oxidative stress and endothelial dysfunction

http://www.wikidata.org/entity/Q34816253

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2013 nî lūn-bûn

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2013年の論文

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Chao-zong Lan

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Han-chun Chen

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Jian Wang

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Kun Guo

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Ling-ling Huang

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Shan Gao

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Ting-ting Yu

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Xing-hui Wang

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Zhen Zhang

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P2860

Endothelium-dependent contractions and endothelial dysfunction in human hypertension

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.

State-of-the-Art lecture. Role of angiotensin and oxidative stress in essential hypertension.

Molecular and cellular mechanisms of angiotensin II-mediated cardiovascular and renal diseases.

Global burden of blood-pressure-related disease, 2001.

Nebivolol: impact on cardiac and endothelial function and clinical utility.

NADPH oxidase-dependent redox signalling in cardiac hypertrophy, remodelling and failure.

Pharmacodynamics of ginsenosides: antioxidant activities, activation of Nrf2, and potential synergistic effects of combinations.

Cardiac hypertrophy: mechanisms and therapeutic opportunities.

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