Deletion of the G protein-coupled receptor 30 impairs glucose tolerance, reduces bone growth, increases blood pressure, and eliminates estradiol-stimulated insulin release in female mice.
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Activation of GPER Induces Differentiation and Inhibition of Coronary Artery Smooth Muscle Cell ProliferationConserved estrogen binding and signaling functions of the G protein-coupled estrogen receptor 1 (GPER) in mammals and fishInternational Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligandsLocalisation of GPR30, a novel G protein-coupled oestrogen receptor, suggests multiple functions in rodent brain and peripheral tissuesHeart Disease in Women: Unappreciated Challenges, GPER as a New TargetMechanisms of uterine estrogen signaling during early pregnancy in mice: an updateSex Hormones and Their Receptors Regulate Liver Energy HomeostasisProgress in the molecular understanding of central regulation of body weight by estrogensRapid steroid hormone actions initiated at the cell surface and the receptors that mediate them with an emphasis on recent progress in fish modelsG-protein-coupled estrogen receptor as a new therapeutic target for treating coronary artery diseaseThe G-protein-coupled estrogen receptor GPER in health and diseaseRegulation of Long Bone Growth in Vertebrates; It Is Time to Catch UpEstrogen biology: new insights into GPER function and clinical opportunitiesInsulinotropic effect of the non-steroidal compound STX in pancreatic β-cellsMechanisms of estradiol-induced insulin secretion by the G protein-coupled estrogen receptor GPR30/GPER in pancreatic beta-cellsG-protein-coupled estrogen receptor agonist suppresses airway inflammation in a mouse model of asthma through IL-10Estrogens in Male PhysiologyG protein-coupled estrogen receptor in energy homeostasis and obesity pathogenesis.The hedgehog signaling pathway: where did it come from?Sex differences in the pulmonary circulation: implications for pulmonary hypertensionRole of Nongenomic Signaling Pathways Activated by Aldosterone During Cardiac Reperfusion Injury.Rapid regulation of K(ATP) channel activity by 17{beta}-estradiol in pancreatic {beta}-cells involves the estrogen receptor {beta} and the atrial natriuretic peptide receptor.Sexually dimorphic role of G protein-coupled estrogen receptor (GPER) in modulating energy homeostasis.Minireview: Estrogenic protection of beta-cell failure in metabolic diseases.Mechanisms of estrogen signaling and gene expression via GPR30GPER/GPR30 and Regulation of Vascular Tone and Blood Pressure.The G protein-coupled receptor GPR30 inhibits proliferation of estrogen receptor-positive breast cancer cells.Rapid, nongenomic estrogen actions protect pancreatic islet survival.Genistein induces pancreatic beta-cell proliferation through activation of multiple signaling pathways and prevents insulin-deficient diabetes in miceInfluence of charge on cell permeability and tumor imaging of GPR30-targeted 111in-labeled nonsteroidal imaging agents.GPR30 deficiency causes increased bone mass, mineralization, and growth plate proliferative activity in male mice.The G protein-coupled oestrogen receptor 1 agonist G-1 disrupts endothelial cell microtubule structure in a receptor-independent manner.Evidence that the G protein-coupled membrane receptor GPR30 contributes to the cardiovascular actions of estrogen.Deletion of G protein-coupled estrogen receptor increases endothelial vasoconstrictionMinireview: G protein-coupled estrogen receptor-1, GPER-1: its mechanism of action and role in female reproductive cancer, renal and vascular physiologyGPER deficiency in male mice results in insulin resistance, dyslipidemia, and a proinflammatory state.Sex steroid actions in male boneEstrogen and androgen receptors: regulators of fuel homeostasis and emerging targets for diabetes and obesity.Estrogen receptor activation and cardioprotection in ischemia reperfusion injury.Emerging roles of GPER in diabetes and atherosclerosis.
P2860
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P2860
Deletion of the G protein-coupled receptor 30 impairs glucose tolerance, reduces bone growth, increases blood pressure, and eliminates estradiol-stimulated insulin release in female mice.
description
2008 nî lūn-bûn
@nan
2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Deletion of the G protein-coup ...... nsulin release in female mice.
@ast
Deletion of the G protein-coup ...... nsulin release in female mice.
@en
Deletion of the G protein-coup ...... nsulin release in female mice.
@nl
type
label
Deletion of the G protein-coup ...... nsulin release in female mice.
@ast
Deletion of the G protein-coup ...... nsulin release in female mice.
@en
Deletion of the G protein-coup ...... nsulin release in female mice.
@nl
prefLabel
Deletion of the G protein-coup ...... nsulin release in female mice.
@ast
Deletion of the G protein-coup ...... nsulin release in female mice.
@en
Deletion of the G protein-coup ...... nsulin release in female mice.
@nl
P2093
P50
P356
P1433
P1476
Deletion of the G protein-coup ...... nsulin release in female mice.
@en
P2093
Björn Olde
Christer Owman
Clifford J Rosen
Ingmar Lundquist
Joanna Daszkiewicz-Nilsson
L M Fredrik Leeb-Lundberg
Martin L Adamo
Niklas Andersson
Per Hellstrand
Per-Olof Grände
P304
P356
10.1210/EN.2008-0623
P407
P577
2008-10-09T00:00:00Z