Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
about
Invasion by P. falciparum merozoites suggests a hierarchy of molecular interactionsThe origin of malignant malariaMechanisms of cellular invasion by intracellular parasitesAntibodies to reticulocyte binding protein-like homologue 4 inhibit invasion of Plasmodium falciparum into human erythrocytesComplement receptor 1 is a sialic acid-independent erythrocyte receptor of Plasmodium falciparumHuman erythrocyte band 3 functions as a receptor for the sialic acid-independent invasion of Plasmodium falciparum. Role of the RhopH3-MSP1 complexA Presenilin-like protease associated with Plasmodium falciparum micronemes is involved in erythrocyte invasionImmune Escape Strategies of Malaria ParasitesPfRH5: a novel reticulocyte-binding family homolog of plasmodium falciparum that binds to the erythrocyte, and an investigation of its receptorAntibodies targeting the PfRH1 binding domain inhibit invasion of Plasmodium falciparum merozoitesEpigenetic silencing of Plasmodium falciparum genes linked to erythrocyte invasion.An adaptable two-color flow cytometric assay to quantitate the invasion of erythrocytes by Plasmodium falciparum parasites.Population genetics of GYPB and association study between GYPB*S/s polymorphism and susceptibility to P. falciparum infection in the Brazilian Amazon.RBC barcoding allows for the study of erythrocyte population dynamics and P. falciparum merozoite invasionHost age and expression of genes involved in red blood cell invasion in Plasmodium falciparum field isolates.Changes in parasite virulence induced by the disruption of a single member of the 235 kDa rhoptry protein multigene family of Plasmodium yoelii.Evolution of human-chimpanzee differences in malaria susceptibility: relationship to human genetic loss of N-glycolylneuraminic acidSafety and immunogenicity of a recombinant nonglycosylated erythrocyte binding antigen 175 Region II malaria vaccine in healthy adults living in an area where malaria is not endemicSystems biology analysis of gene expression during in vivo Mycobacterium avium paratuberculosis enteric colonization reveals role for immune tolerancePlasmodium falciparum field isolates from South America use an atypical red blood cell invasion pathway associated with invasion ligand polymorphisms.Plasmodium falciparum field isolates use complement receptor 1 (CR1) as a receptor for invasion of erythrocytes.A flow cytometry-based assay for measuring invasion of red blood cells by Plasmodium falciparum.A flow cytometric assay to quantify invasion of red blood cells by rodent Plasmodium parasites in vivoMeasurement of antibody levels against region II of the erythrocyte-binding antigen 175 of Plasmodium falciparum in an area of malaria holoendemicity in western KenyaVariation in Plasmodium falciparum erythrocyte invasion phenotypes and merozoite ligand gene expression across different populations in areas of malaria endemicity.Molecular epidemiology of malaria.Functional analysis of erythrocyte determinants of Plasmodium infectionAnalysis of Erythrocyte Invasion Mechanisms of Plasmodium falciparum Clinical Isolates Across 3 Malaria-Endemic Areas in Ghana.Variation in use of erythrocyte invasion pathways by Plasmodium falciparum mediates evasion of human inhibitory antibodies.Polymorphism in the Plasmodium falciparum erythrocyte-binding ligand JESEBL/EBA-181 alters its receptor specificity.Invasion of host cells by malaria parasites: a tale of two protein families.Polymorphisms in erythrocyte binding antigens 140 and 181 affect function and binding but not receptor specificity in Plasmodium falciparumErythrocyte-binding antigens of Plasmodium falciparum are targets of human inhibitory antibodies and function to evade naturally acquired immunity.Human-specific evolution of sialic acid targets: explaining the malignant malaria mystery?Low multiplication rates of African Plasmodium falciparum isolates and lack of association of multiplication rate and red blood cell selectivity with malaria virulence.Distribution of erythrocyte binding antigen 175 (EBA-175) gene dimorphic alleles in Plasmodium falciparum field isolates from Sudan.Invasion profiles of Brazilian field isolates of Plasmodium falciparum: phenotypic and genotypic analyses.Toxoplasma gondii secretory proteins bind to sulfated heparin structures.Genetic evidence for erythrocyte receptor glycophorin B expression levels defining a dominant Plasmodium falciparum invasion pathway into human erythrocytes.Structural characterization of the erythrocyte binding domain of the reticulocyte binding protein homologue family of Plasmodium yoelii.
P2860
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P2860
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@ast
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@en
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@nl
type
label
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@ast
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@en
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@nl
prefLabel
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@ast
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@en
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@nl
P2860
P50
P1476
Erythrocyte invasion phenotypes of Plasmodium falciparum in The Gambia.
@en
P2860
P304
P356
10.1128/IAI.71.4.1856-1863.2003
P407
P577
2003-04-01T00:00:00Z