The small envelope protein E is not essential for murine coronavirus replication.
about
Rewiring the severe acute respiratory syndrome coronavirus (SARS-CoV) transcription circuit: engineering a recombination-resistant genomeRelevance of Viroporin Ion Channel Activity on Viral Replication and PathogenesisCoronavirus virulence genes with main focus on SARS-CoV envelope geneThe M, E, and N structural proteins of the severe acute respiratory syndrome coronavirus are required for efficient assembly, trafficking, and release of virus-like particles.Immune responses against severe acute respiratory syndrome coronavirus induced by virus-like particles in mice.PLP2 of mouse hepatitis virus A59 (MHV-A59) targets TBK1 to negatively regulate cellular type I interferon signaling pathway.Severe acute respiratory syndrome coronavirus envelope protein ion channel activity promotes virus fitness and pathogenesisPathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.A single tyrosine in the severe acute respiratory syndrome coronavirus membrane protein cytoplasmic tail is important for efficient interaction with spike proteinCoronaviruses post-SARS: update on replication and pathogenesis.Immunization with an attenuated severe acute respiratory syndrome coronavirus deleted in E protein protects against lethal respiratory disease.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.Palmitoylation of SARS-CoV S protein is necessary for partitioning into detergent-resistant membranes and cell-cell fusion but not interaction with M protein.Pathogenesis of murine coronavirus in the central nervous system.Severe acute respiratory syndrome coronavirus envelope protein regulates cell stress response and apoptosis.A major determinant for membrane protein interaction localizes to the carboxy-terminal domain of the mouse coronavirus nucleocapsid protein.Contribution of trafficking signals in the cytoplasmic tail of the infectious bronchitis virus spike protein to virus infection.An interaction between the nucleocapsid protein and a component of the replicase-transcriptase complex is crucial for the infectivity of coronavirus genomic RNA.Dendritic cell-specific antigen delivery by coronavirus vaccine vectors induces long-lasting protective antiviral and antitumor immunity.Severe acute respiratory syndrome coronavirus group-specific open reading frames encode nonessential functions for replication in cell cultures and mice.Intracellular targeting signals contribute to localization of coronavirus spike proteins near the virus assembly site.Coronavirus pathogenesis and the emerging pathogen severe acute respiratory syndrome coronavirus.A single polar residue and distinct membrane topologies impact the function of the infectious bronchitis coronavirus E protein.Evolved variants of the membrane protein can partially replace the envelope protein in murine coronavirus assembly.The hydrophobic domain of infectious bronchitis virus E protein alters the host secretory pathway and is important for release of infectious virus.Deficient incorporation of spike protein into virions contributes to the lack of infectivity following establishment of a persistent, non-productive infection in oligodendroglial cell culture by murine coronavirus.Identification of functionally important negatively charged residues in the carboxy end of mouse hepatitis coronavirus A59 nucleocapsid protein.Supramolecular architecture of severe acute respiratory syndrome coronavirus revealed by electron cryomicroscopy.Identification of a Golgi complex-targeting signal in the cytoplasmic tail of the severe acute respiratory syndrome coronavirus envelope protein.The 3' cis-acting genomic replication element of the severe acute respiratory syndrome coronavirus can function in the murine coronavirus genome.Incorporation of spike and membrane glycoproteins into coronavirus virions.A hypervariable region within the 3' cis-acting element of the murine coronavirus genome is nonessential for RNA synthesis but affects pathogenesisA severe acute respiratory syndrome coronavirus that lacks the E gene is attenuated in vitro and in vivo.The structure and functions of coronavirus genomic 3' and 5' ends.The cytoplasmic tail of the severe acute respiratory syndrome coronavirus spike protein contains a novel endoplasmic reticulum retrieval signal that binds COPI and promotes interaction with membrane protein.Exceptional flexibility in the sequence requirements for coronavirus small envelope protein functionRole of the coronavirus E viroporin protein transmembrane domain in virus assembly.The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus InfectionSubcellular localization of SARS-CoV structural proteins.A Coronavirus E Protein Is Present in Two Distinct Pools with Different Effects on Assembly and the Secretory Pathway.
P2860
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P2860
The small envelope protein E is not essential for murine coronavirus replication.
description
2003 nî lūn-bûn
@nan
2003 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2003年の論文
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2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
name
The small envelope protein E is not essential for murine coronavirus replication.
@ast
The small envelope protein E is not essential for murine coronavirus replication.
@en
The small envelope protein E is not essential for murine coronavirus replication.
@nl
type
label
The small envelope protein E is not essential for murine coronavirus replication.
@ast
The small envelope protein E is not essential for murine coronavirus replication.
@en
The small envelope protein E is not essential for murine coronavirus replication.
@nl
prefLabel
The small envelope protein E is not essential for murine coronavirus replication.
@ast
The small envelope protein E is not essential for murine coronavirus replication.
@en
The small envelope protein E is not essential for murine coronavirus replication.
@nl
P2860
P1433
P1476
The small envelope protein E is not essential for murine coronavirus replication.
@en
P2093
Paul S Masters
P2860
P304
P356
10.1128/JVI.77.8.4597-4608.2003
P407
P577
2003-04-01T00:00:00Z