MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystals. - Wikidata - wikimedia - TriplyDB

MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystals.

MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystals.

http://www.wikidata.org/entity/Q34872946

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Developments in the scientific and clinical understanding of gout Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Function of Nod-like receptors in microbial recognition and host defense Neutrophil microvesicles resolve gout by inhibiting C5a-mediated priming of the inflammasome Trauma equals danger--damage control by the immune system Activation of NLRP3 inflammasome by crystalline structures via cell surface contact.Intracellular innate immunity in gouty arthritis: role of NALP3 inflammasome Inflammasome-associated nucleotide-binding domain, leucine-rich repeat proteins and inflammatory diseases Interleukin-1β Processing Is Dependent on a Calcium-mediated Interaction with Calmodulin Engagement of fatty acids with Toll-like receptor 2 drives interleukin-1β production via the ASC/caspase 1 pathway in monosodium urate monohydrate crystal-induced gouty arthritis How dying cells alert the immune system to danger The inflammasome: a caspase-1-activation platform that regulates immune responses and disease pathogenesis Synaptotagmin-mediated vesicle fusion regulates cell migration.Plasmodium-induced inflammation by uric acid Airway uric acid is a sensor of inhaled protease allergens and initiates type 2 immune responses in respiratory mucosa Gout increases risk of fracture: A nationwide population-based cohort study.Risk of infections in patients with gout: a population-based cohort study.Caspase 1-independent activation of interleukin-1beta in neutrophil-predominant inflammation Chlamydia muridarum Infection of Macrophages Stimulates IL-1β Secretion and Cell Death via Activation of Caspase-1 in an RIP3-Independent Manner MARK4 regulates NLRP3 positioning and inflammasome activation through a microtubule-dependent mechanism Uric acid promotes an acute inflammatory response to sterile cell death in mice The inflammatory response to double stranded DNA in endothelial cells is mediated by NFκB and TNFα Uric acid: the oxidant-antioxidant paradox Bacterial pathogen-associated molecular patterns stimulate biological activity of orthopaedic wear particles by activating cognate Toll-like receptors Mechanistic aspects of inflammation and clinical management of inflammation in acute gouty arthritis.Uric acid induces renal inflammation via activating tubular NF-κB signaling pathway.Expression and secretion of TNF-α in mouse taste buds: a novel function of a specific subset of type II taste cells.Increased responsiveness of peripheral blood mononuclear cells to in vitro TLR 2, 4 and 7 ligand stimulation in chronic pain patients Vaccine-induced protection against 3 systemic mycoses endemic to North America requires Th17 cells in mice.An old disease with new insights: Update on diagnosis and treatment of gout.Gout: new insights into an old disease.The inflammatory response to cell death.Dendritic cell IL-1α and IL-1β are polyubiquitinated and degraded by the proteasome.Alum adjuvant boosts adaptive immunity by inducing uric acid and activating inflammatory dendritic cells.Impact of cagPAI and T4SS on the inflammatory response of human neutrophils to Helicobacter pylori infection.The role of uric acid as an endogenous danger signal in immunity and inflammation.Chemokine-mediated inflammation in the degenerating retina is coordinated by Müller cells, activated microglia, and retinal pigment epithelium Whole tumor antigen vaccines.Altered adherent leukocyte profile on biomaterials in Toll-like receptor 4 deficient mice.Interleukin-1 as a common denominator from autoinflammatory to autoimmune disorders: premises, perils, and perspectives

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MyD88-dependent IL-1 receptor signaling is essential for gouty inflammation stimulated by monosodium urate crystals.

http://www.wikidata.org/entity/Q34872946

description

2006 nî lūn-bûn

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2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

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Journal of Clinical Investigation

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MyD88-dependent IL-1 receptor ...... by monosodium urate crystals.

@en

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Arron Hearn

http://www.w3.org/2001/XMLSchema#string

Chun-Jen Chen

http://www.w3.org/2001/XMLSchema#string

Douglas Golenbock

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George Reed

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Kate Fitzgerald

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Kenneth L Rock

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Yan Shi

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P2860

A Toll-like receptor recognizes bacterial DNA

Discrimination of bacterial lipoproteins by Toll-like receptor 6

Cutting edge: role of Toll-like receptor 1 in mediating immune response to microbial lipoproteins

Species-specific recognition of single-stranded RNA via toll-like receptor 7 and 8

Toll-like receptor signalling

LPS-TLR4 signaling to IRF-3/7 and NF-kappaB involves the toll adapters TRAM and TRIF

Gout-associated uric acid crystals activate the NALP3 inflammasome

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Toll-like receptors as adjuvant receptors

Small anti-viral compounds activate immune cells via the TLR7 MyD88-dependent signaling pathway

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2262-2271

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10.1172/JCI28075

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8

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116

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